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Cerebellar Hemorrhage
Intern 胡朝凱2002/12/09
Pathophysiology Long-standing hypertension ----most common
cause Tumor Trauma Anticoagulant use Blood dyscrasias Arteriovenous malformation rupture Amyloid angiopathy
Key point Location ( midline vs. hemisphere ) Brain stem compression Size
Symptoms Headache of abrupt onset Nausea and vomiting Inability to walk ( truncal ataxia) Dizziness, vertigo Dysarthria Nuchal pain Loss or alteration of consciousness
Physical examination Alert or unconscious Irregular respiration Impaired or absent pupillary responses Abnormal eye movement
Impaired oculo-cephalic responses ( Doll eye ) Decreased or absent corneal responses Facial weakness Nuchal rigidity Possible presence of extensor plantar responses
(unilateral or bilateral)
Neural examination (0) Ipsilateral half body No definite functional location
Neural examination (1) Nystagmus -- up and down Dysarthria -- irregular, explosive, slurring
of words
Neural examination (2) Ataxia ( past-pointing ; dysmetria ) finger to nose test heel-to-shin test
Intention tremor Adiadochokinesis
pronation and supination test finger to thumb test
Asynergy decomposition of movements
Neural examination (3) Hypotonia Vermis syndrome
paraxial musculature, truncal ataxia Gait
wide-based, irregular, staggering, like an intoxicated person
Romberg’s sign Tandem walking
Lab study Coagulation studies and a platelet count
Imaging studies CT scan -- acute CH should be visible as
increased density in the posterior fossa Location Brainstem compression Absolute size of the clot in maximum diameter Ablation of the fourth ventricle Obstructive hydrocephalus
MRI -- vascular anatomy, extent of damage, and other abnormalities (eg, tumor, AVM)
Risk for deterioration Admission systolic blood pressure > 200 mmHg Pinpoint pupils and abnormal corneal and
oculocephalic reflexes Hemorrhage extending into the cerebellar vermis Hematoma > 30 mm Brainstem distortion Intraventricular hemorrhage Acute hydrocephalus
Criteria for medical conservative treatment Best --
Glasgow coma scale score of 14 or greater Small hemorrhage ( < 30 mm ) Without hydrocephalus
Worst -- Comatose Flaccid Without brainstem reflexes Large midline hemorrhage
Medical treatment O2
Fluid -- isotonic saline. Mannitol (1 g/kg) -- preoperatively in patients
with tight posterior fossa. Antihypertensive agent -- persistent hypertension
( mean arterial pressure >130 mm hg ) Atropine (0.5-1 mg) -- cushing response,
symptomatic bradycardia
Surgical intervention Controversial. Ventriculostomy -- hemorrhage and
hydrocephalus. Suboccipital craniotomy with clot
evacuation -- altered level of consciousness and a large clot (30 ~ 40 mm).
Further patient care: Careful monitoring
Consciousness Vital signs ICP
If immediate surgical intervention is deferred, a deteriorating clinical course may necessitate surgery at a later time.
Physical and Occupational therapy.
Medical/legal pitfalls: Failure to diagnose and delayed diagnosis
following clinical deterioration. Small, lateral hemorrhage. Patients with diminished level of consciousness
or intractable vomiting unable to cooperate with examination.
Testing the gait complaint of dizziness accompanied by headache, nausea, vomiting, or obtundation.
Take-Home Message Headache
Worst First Days or weeks Fever Vomiting precedes
headache Disturbs sleep Abrupt Onset after age of 55
Neural examination Pupil Ocular movement Optic fundi Facial movement Muscle power DTR Pain and vibration
sense in hands and feet Gait
Thank You For Your Attention !! May God Bless Our Friends !!