Pedia Eruption

7/29/2019 Pedia Eruption

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Chronologic development and eruption of teeth:

A variety of developmental defects that are evident after eruption of

primary and permanent teeth can be related to systemic and localfactors that influence matrix formation and the calcification process.

The sequence of calcification of primary teeth is central incisor,lateral

incisor,canine and second molar.the factors that have been related to

the eruption of teeth include elongation of root,forces exerted by

vascular tissue around and beneath the root ,growth of the alveolar

bone,growth if dentin,growth of pull of periodontal

memberane,hormonal influnece,present of viable dental

follcile,pressure from muscular action and resorption of alveolar crest.


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Tooth eruption is a series of metabolic events inalveolar bone characterized by bone resorptionand formation on opposite sides of dental

follicle and the tooth does not contribute to thisprocess.teeth eruption is influenced bypituitary growth hormone and thyroidhormone and parathyroid hormone related

protein for tooth eruption


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Eruption of premolars teeth are delay inchildren who loss primary molar at 4 or 5 and

before , if extraction of primary molar occursafter the age of 5 there is a decrease in delay ofpremolar eruption , at 8-10 premolar eruptionresulting from premature loss of primary teeth

is greatly accelerated.


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The mandibular canine erupt before maxillary andmandibular first premolar in girls ,in boys eruption isreversed,..

Most common sequence eruption of permanent teeth in

mandibular is 1st molar,central incisor,lateralincisor,canine,first premolar,second premolar and 2ndmolarbut in maxilla is 1st molar,central incisor,lateralincisor,first premolar,second premolar,canine and

second molar


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There may be insufficient room in the arc for

the newly erupted permanent tooth , itsposition will improve over several months , insome case there is justification for removal ofthe corresponding primary tooth , extraction of

other primary teeth in the area is notrecommended , because it will onlytemporarily relieve the crowding and mayeven contribute to the development of moresevere arch length in adequancy.


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Because the eruption of teeth is a normalphysiologic process , the association with feverand systemic disturbances is not justified ,A

fever or respiratory tract infection during thistime should be considered coincidental to theeruption process rather that related to it .inflemation of gingival tissue before complete

emergence of crown may cause a temporarypainful condition that subside within a fewdays.


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A bluish purple,elevated area of tissue,commonlycalled an hematoma,occasionally develops a few weeksbefore the eruption of primary or permanentteeth,usually within in a few days the tooth breaks

trough the tissue and because of condition is almostself limited treatment of eruption hematoma is rarelynecessary.


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THE PREVALANCE OF NATAL TEETH(PRESENT ATBRITH)AND NOENATAL TEETH(TEETH THAT ERUPTDURING FIRST 30 DAYS) IS LOW.THE TERM NATALAND NEONATAL CONSTITUTE A RELATIVELYARTIFICIAL DISTINCTION AND SHOULD BEFURTHER QUALIFIED TO PROVIDE A MORE

PRATICAL CLINICAL MEANINGTHEY SUGHESTEDTHE TERM MATURE AND IMMATURE ARE MOREKEEPING WITH THE VARYING PROGNOSE

ASSOCIATED WITH SUCH TEETH.


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Small , white or grayish white lesions on the alveolarmucosa of the newborn may on rare occasions beincorrectly diagnosed as natal teeth . the lesion areusually multiple but do not increase in size , notreatment is indicated , since the lesions are

spontaneously shed a few weeks after birth. Three types of inclusion cysts:

1.Epstein pearls

2.Bohn modules

3..Dental lamina cysts


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1)Ankylosed teeth: The mandibular primary molars are the teeth most often observed to be

ankylosed,in unusual case all primary molars may become firmly attached to thealveolar bone before their normal exfoliation time.

Anlylosis of primary molar to alveolar bone dose not usually occur until after its

root resorption begins.

2)Ankylosis of primary molars with absence of permanentsuccessors:

the importance of the presence of a permanent successor for normalexfoliation of a primary molar,no ankylosed primary molars without

permanent successors were found to exfoliate spontaneously,howeververy small root absorption was observed for most of ankylosed tooth.

3)Ankylosed permanent teeth: The incomplete eruption of a permanent molar may be related to a small

area of root ankylosis


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4)Trismoy 21 syndrome(down sundrome)

Trismoy 21 syndrome (down syndrome) is one of thecongenital anomalies in which delayed eruption of theteeth frequently occurs.the orbits are small,the eyes

slope upward and the bridge of the nose is moredepressed than normal.


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5)HYPOTHYROIDISM

Hypothyroidism is another possible cause of delayed eruption .patients in whom the function of thyroid gland is extremely

deficient have characteristic dental finding .

6)CONGENITAL HYPOTHYROIDISM(CRETINISM)

Hypothyroidisim occuring at birth and during the period of mostrapid growth,if undetected and treated,cause mental deficiencyand dwarfism


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7)Juvenile hypothyroidism(acquired hypothyroidism) Juvenile hypothyroidism result from a malfunction of thyroid

gland,usually between 6 and 12 yrs.because the deficiency occursafter period of rapid growth,the unusual facial and body patternchararcteristic of a person with congenital hypothyroidism is not

peresent however obsity is evident to a lesser degree.

8)Hypopituitarism:

A pronounced deceleration of a growth of bones and soft tissue ofthe body will result from a deficiency in secretion of the growthhormone


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9)Achondroplastic DWARFISM:

It diagnosed at birth, demonstrate a few characteristicdental findings , growth of extremities is limitedbecause of a lack of calcification in the cartilage of longbone ,it occur when the ages of parents differ

significantly.


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SABETRASEKH SEPEHR

KARAMI SHARAM

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