Oligohydramnion on Post Term Pregnancy

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    Oligohydramnion in post

    term pregnancyKheluwis.s

    406148098

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    ,hysiology

    uring the 7rst hal o pregnancy!transer o water and other smallmolecules ta*es place across the

    amnion intramem/ranous fow! andacross etal s*in

    'etal urine 8 and 11 wee*s (/ecamemaor component ater 18 wee*s+

     %ransport across the etal s*incontinues until *eratiniation occursat )) to ): wee*s.

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    Amniotic 'luid 2olume ;egulationin ate ,regnancy

    Pathway Efect on Volume Approximate DailyVolume (mL)

    'etal urination ,roduction 1000

    'etal swallowing ;esorption

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    Amniotic fuid "olume assessment

    O/ecti"e assessment depends on =>? tomeasure

    eepest "ertical poc*et (2,+

    Amniotic fuid inde (A'#+ #t is a total o the

    2,s in each o the our @uadrants o theuterus. it is a more sensiti"e indicator o A'2throughout pregnancy

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    Amniotic fuid a/normalities

    Oligohydramnios: 

    e7ned as reduced amniotic fuid

    i.e. amniotic fuid inde o : cm or

    less or the deepest "ertical pool ) cm

    Polyhydramnios: e7ned as ecessi"e amount o amnioticfuid o )000 ml or more (A'# o B )4 cmor the deepest "ertical pool o B 8 cm+.

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    ETIOLOGY

    FETAL PROM

    CHROMOSOMAL ANOMALIES

    CONGENITAL ANOMALIES

    IUGR

    IUFD POSTTERM PREGNANCY

    MATERNAL PREECLAMPSIA

    CHRONIC HT 

    PLACENTAL CHRONIC ABRUPTION TTTS CVS

    DRUGS NSAIDs

    ACE INHIBITORS

    IDIOPATHIC

    9

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    DIAGNOSIS

     SYMPTOMS

    NO SPECIFICSYMPTOMS

    Postterm

    preeclampsaLess !etal mo"eme#ts

      SIGNS

    Uter$s % small!or &ate

    Feels !$ll o!!et$sMalprese#tato#sIUGR

    10

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    =?$

    METHODS 

    DVP '( cms

      )'* se"ere+

    AFI ', cms

      ),-./or&erl#e+

    11

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     %echni@ue o A'#

    Uter$s &"&e& #to 0 1$a&ra#tsTra#s&$cer # "ertcal pla#eS$m o! 0 1$a&ra#ts ma2 poc3et

    &ept4 e2cl$&#5 cor& 6 lm/s7Pror to (8 93s ( 4al"esT9#s: composte AFI or #&"&$al

    "ertcal poc3ets

    1)

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    O-,#A%#O?

      FETAL

    A/orto#Premat$rt;

    IUFD

    De!ormtes %CTEV

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    MANAGEMENT 

    C,C? =,O

    ETIOLOGYGESTATIONAL AGESEVERITYFETAL STATUS 6 =ELL BEING

    14

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    DETERMINE ETIOLOGY

    R>O PROM< 4>o me&cal ll#essTARGETED USG FOR ANOMALIESR>O IUGR

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    TREATMENT ADEQUATE REST – decreases dehydration

    HYDRATION – Oral/IV Hypotonic l!ids"# $it/d%

      help!l d!rin& la'o!r(prior

    to E)V( US*

    SERIA$ US* – +onitor &ro,th(A-I

    INDU)TION O- $A.OUR

     

    16

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    TREATMENT ACC7 TO CAUSE

    Dr$5 #&$ce&PROM % INDUCTIONPPROM % A#t/otcs

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