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SYSTEMATIC APPROACH TO THE SERIOUSLY ILL OR INJURED CHILD
PEDIATRIC ADVANCED LIFE SUPPORT -DR.HARDIK SHAH
Initial impression
• First quick “from the doorway” observation.• This initial visual and auditory observation of
the child’s consciousness, breathing and color is accomplished within seconds of encountering the child.
Initial impression
Consciousness Level of Consciousness(eg. Unresponsive, irritable, alert)
Breathing Increased work of breathing, absent or decreased respiratory effort, or abnormal sounds heard without auscultation
color Abnormal skin color, such as cyanosis, pallor or mottling
Evaluate-Identify-Intervene
• Use the evaluate-identify – intervene sequence.
• Always be alert to a life-threatening problem.• If any point , identify a life-threatening problem,
immediately activate emergency response.
Evaluate Clinical
AssessmentBrief Description
Primaryassessment
A rapid, hands-on ABCDE approach to evaluate respiratory , cardiac and neurologic function; this step includes assessment of vital signs and pulse oxymetry
secondaryassessment
A focused medical history and a focused physical examination
Diagnostic test
laboratory, radiological and other advanced tests that help to identify the child’s physiologic condition and diagnosis
Identify Type severity
Respiratory •Upper airway obstruction•Lower airway obstruction•Ling tissue disease•Disordered control of breathing
•Respiratory distress•Respiratory failure
Circulatory •Hypovolemic shock•Distributive shock•Cardiogenic shock•Obstructive shock
•Compensated shock•Hypotensive shock
Cardiopulmonary failure
Cardiac arrest
Intervene
On the basis of identification of child’s problem, intervene with appropriate action.
• Positioning the child to maintain a patent airway• Activating emergency respone• Starting CPR
obtaining the code cart and monitor• Placing the child on a cardiac monitor and pulse oximeter• Support ventilation• Starting medications and fluids
Primary assessment
Primary assessement uses an ABCDE model:• Airway• Breathing• Circulation• Disabilyty• Exposure
A B
C
D
E
Airway
To assess upper airway patency:• Look for movement of chest or abdomen• Listen for air movement and breath soundsDecide if UA is clear , maintainable or not maintainableSigns suggest UA obstruction:• Increase inspiratory effort with retraction• Abnormal inspiratory sounds• Episodes where no airway or breath sounds are present
despite respiratory effort
Status Description
Clear Airway is open and unobstructed for normal breathing
Maintainable Airway is obstructed but can be maintainable by simple measures (eg head tilt-chin lift)
NotMaintainable
Airway is obstructed but cannot be maintainable without advanced intervention (eg intubation)
Simple measures:• Allow the child to assume a position of comfort or position the child
to improve airway patency• Use head tilt-chin lift or jaw thrust to open the airway:- If cervical spine injury suspect, open airway by using a jaw thrust
without neck extension. If this maneuver does not open the airway, use head tilt-chin lift without neck extension.
• Avoid overextending the head/neck in infants because this may occlude the airway.
• Suction the nose and oropharynx.• Perform foreign- body airway obstruction releif tech if suspect that
child has aspirated foreign body:- <1 yr old, a combination of 5 back blows and 5 chest thrusts - >1 yr old, providers should give a series of 5 abdominal thrusts
(Heimlich maneuver)• Use airway adjuncts (NPA or OPA) to keep the tongue from falling
back and obstructing the airway.
Head tilt-chin lift
Jaw thrust
Advanced interventions• Endotracheal intubation or placement of a laryngeal
mask airway• Application of continuous positive airway pressure
(CPAP) or noninvasive ventilation• Removal of a FB; whis intervention may require
direct laryngoscopy• Cricothyrotomy
Breathing
Assessment of breathing includes:• Respiratory rate• Respiratory effort• Chest expasion and air movement• Lung and airway sound• O2 saturation by pulse oxymetry
Normal respiratory rateAge Breaths / min
Infants (< 1 year) 30 – 60
Toddler (1-3 yrs) 24-40
Preschooler (4-5 yrs) 22-34
School age (6-12 yrs) 18-30
Adolescent (13-18 yrs) 12-16
Abnormal respiratory rate
Tachypnea : • First sign of respiratory distress in infants.• Quite tachypnea- tachypnea without signs of increased
respiratory effort.Bradypnea:• Possible caused are resp muscle fatigue, central nervous
system injury or infection, hypothermia or medication that depress resp drive.
Apnea:• Cessation of breathing for 20 secs or cessation for less than
20 secs if accompanied by bradycardia, cynosis or pallor.
Respiratory effort
• Increase respiratory effort results from conditions that increase resistance to airflow or that cause lungs to be stiffer and difficult to inflate.
Signs of increase respiratory effort include.• Nasal flaring• Retractions• Head bobbing or seesaw raspirations
Nasal flaring:• Dilatation of nostrils with each inhalation.• Most common in infant and younger childrenRetractions:• Inward movement of the chest wall or tissues, neck or
sternum during inspiration.Head bobbing or seesaw respiration:Indicate increased risk of deterioration- Head bobbing- caused by use of neck muscles to assist
breathing.• Most frequently seen in infants and sign of respiratory
failure- Seesaw respiration- chest retract and abdomen expand
during inspiration.
Retractions:Breathing difficulty Location of retraction Description
Mild to moderate subcostal Retraction of abdomen just below ribcage
Substernal Retraction of abdomen at the bottom of breast bone
intercostal Retraction between ribs
Severe Supraclavicular Retraction in the neck just above the collar bone
Suprasternal Retraction in the chest just above breast bone
sternal Retraction of sternum toward the spine
Chest expansion and Air movement
• Evaluate magnitude of chest wall expansion and air movement to assess adequecy of the child’s tidal volume.
• Normal tidal volume- 5-7 ml/kg• Tidal volume is difficult to measure unless a
child is mech ventilated, so clinical assessment imp.
Chest wall expansion:• Chest expansion during inspiration should be symmetric.• Decreased or asymmetric chest expansion may result
from in adequate effort, airway obstruction, atelectasis, pneumothorax, hemothorax, PE, mucosal plug or FB aspiration.
Air movement:• Auscultation for air movement is critical.• Listen for the intensity of breath sounds and quality of
air movement, particularly in the distal lung fields.• Decreased chest excrusion or air movement
accompanies poor resp effort.• Diminished distal air entry suggests air flow obstruction
or lung tissue disease.
Lung and airway sounds
• Stridor: - coarse, usually higher pitched breathing sound
typically heard on inspiration.- Sign of upper airway obstruction- Indicate – obstruction is critical and requires
immediate intervention.Causes: FBAO, Croup , laryngomalacia, tumor or
cyst, upper airway edema
• Grunting- Typically a short, low pitched sound heard during
expiration.- Misinterpreted as soft cry- Sign of lung tissue disease resulting from small
airway collapse or alvelolar collapse.- Indicate progression of RD to RF.- Causes: pneumonia, ARDS, Pulmonary contusion.• Gurgling:- Bubbling sound heard during inspiration or
expiration.- Results from upper airway obstruction d/t airway
secretions, vomting or blood.
• Wheezing- High pitched or low pitched whistling sound heard
most often during expiration.- Indicate lower airway obstruction.- Causes: Bronchiolitis and Asthma• Crackles/ Rales: - Sharp creckling inspiratory sounds.- Dry crackles: atelectasis and interstitial lung
disease..- Moist crackles: indicate accumulation of alveolar
fluid.
Oxygen saturation by pulse oxymetry
• Monitor the % of HB that is saturated with O2.(SPo2)
• Interpret pulse oxymetry readings in conjuction with clinical assessment and other signs.
• Pulse oxymeter does not accurately recognize methemoglobin or carboxyHB.
Circulation
Circulation assessed by evaluation of • Heart rate and rhythm• Pulse• Capillary refill time• Skin color and temp• Blood pressure
Heart rate and rhythmAge Awake rate mean
New bon to 3 months
85-205 140
3 month to 2 yrs 100-190 130
2 yrs to 10 yrs 60-140 80
> 10 yrs 60-100 75
Bradycardia: heart rate slower than normal for child’s age.
- Most common cause- hypoxia- If bradycardia associated with poor perfusion
immediately support ventilation wth B&M and administer supplementry O2..
Tachycardia: heart rate faster than normal for child’s age.
Pulses:
• Evaluation of pulses is critical to assessment of systemic perfusion in an ill or injured child.
• Palpate both central and peripheral pulses.Central pulses: Brachial (In infants) , Carotid (older
children) , femoral , axillaryPeripheral: radial, dorsalis pedis , post. tibial.• Weak central pulses are worrisome and indicate need
for very rapid intervention to prevent cardiac arrest.• Beat to beat fluctuation in pulse volume may occur in
children with arrythemias.
Capillary refill time
• Time takes for blood to return to tissue blanched by pressure.
• Increase as skin perfusion decrease.• Prolonged CFT indicate low cardiac out put.• Normal CFT <= 2• To evaluate CFT lift extremity slightly above
the level of the heart, press on the skin and rapidly release the pressure.
Skin color and Temperature
• Mucous membrane, nail beds, palms and soles should be pink.
• When perfusion deteriorates and O2 delivery to tissue becomes inadequate the hands and feet are typically affected 1st.
• They may become cool , pale, dusky or mottled.• If perfusion become worst skin over the trunk
and extremities may under go similar changes.
• Pallor: - Decreased blood supply to the skin (cold, stress,
shock )- Anemia- Decreased skin pigmentation• Mottling:- Irregular or patchy discoloration of the skin.- Serious condition such as hypoxemia, hypovolemia
or shock, may cause intense vasoconstriction from an irregular supply of oxygenated blood to the skin, leading to mottling.
• Cyanosis:- Peripheral cyanosis: bluish discoloration of hands
and feet. Seen in shock , CCF , PVD- Central cyanosis: bluish discoloration of lips and
other mucous membranes. - Causes :- low ambient O2 tension -alveolar hypoventilation -diffusion defect -ventilator/ persion imbalance -intracardiac shunt
Blood pressure
• Cuff bladder should cover about 40% of the mid upper arm circumference.
• BP cuff should extend at least 50-75% of the length of the upper arm.
Hypotension
Age Systolic blood pressure (mmhg)
Term neonate(0- 28 days)
< 60
Infants (1-12 months)
<70
Children (1-10 yrs)
< 70 + (age in yrs x 2 )
Children > 10 yrs < 90
Disability
• Disability assessment is a quick evaluation of neurologic function.
• Clinical signs of brain perfusion are imp indicators of circulatory function in the ill or injured patient.
• Signs include level of consciousness, muscle tone and pupil response.
• Signs of inadequate O2 delivery to the brain correlate with the severity and duration of cerebral hypoxia.
• Standard evaluations include- AVPU pediatric response scale- GCS- Pupil response to light
• Mild head injury- GCS score 13-15• moderate head injury- GCS score 9-12• Mild head injury- GCS score 3-8
Pupils response to light
• Indicator of brainstem function.• If the pupils fail to constrict in response to
direct light, suspect brain stem injury.• Irregularities in pupil size or response to light
may occur as result of ocular trauma or ICP.Assess and record size of pupils , equality of
pupil size , constriction pupil to light.
Exposure
• Undress the seriously ill and injured child as necessary to perform a focused physical examination.
• Maintain cervical spine precaution when turning any child with suspected neck or spine injury.
• Assess core temperature and maintain temp.• Look any trauma such as bleeding , burns and
unusual marking that suggest nonaccidental trauma.• Look for petechiae and purpura s/o septic shock
Secondary assessement
• Focused history• Focused physical examination
• Focused history: to identify imp aspects of the child’s presenting complaint.
- Signs and symptoms: breathing difficulty, decrease level of consciousness, agitation, anxiety, fever, decrease oral intake, diarrhea, vomiting , bleeding , fatigue, time course of symptoms
- Allergies: medication, foods , latex- Medications: name of drug, duration, last dose- Past medical history: Health history (premature birth),
Significant underlying medical problem, Past surgeries , Immunization
- Last meal: time and nature of last intake of lipid or food- Events: event leading to current illness or injury, hazards at
scene , treatment during interval from onset of disease or injury until evaluation, estimated time of arrival
Diagnostic test
• ABG• VBG• HB• Central venous O2 saturation• Arterial lactate• Central venous pressure monitoring• Invasive arterial pressure monitoring• Chest X-ray• ECG• Echocardiography• Peak expiratory flow rate
ABG/ VBG:- Measures Pao2 and paco2 dissolved in blood
plasma. Measurement indicates
Pao2 Adequacy of O2 tension in arterial blood
Paco2 Adequacy of ventilation
Diagnosis ABG result
Hypoxemia Low pao2
Hypercarbia High paco2
Acidosis PH < 7.35
Alkalosis PH >7.45
• Hemoglobin concentration:- Determine O2 carrying capacity of blood.• Central Venous Oxygen Saturation:- Venous blood gases may provide a useful indicator of changes in
balance between O2 delivery to the tissues and tissue O2 consumption.
- Normal SvO2 is about 70 – 75%, assuming arterial O2 saturation is 100%.
• Arterial lactate:- concentration of lactate reflect the balance between lactate
production and use.- Good prognostic indicator.- With the treatment of shock lactate concentration should
decrease.- Lack of response to therapy is more predictive of poor outcome
than the initial elevated lactate concentration.
• Central Venous Pressure monitoring:- Central venous pressure can be monitor through a
central venous catheter.- Measurement of CVP may provide helpful
information to guide fluid and vasoactive therapy.• Invasive arterial Pressure monitoring:- Require arterial catheter , monitoring line ,
transducer and monitoring system.- Enable cont evaluation and display of the SBP and
DBP.- Arterial waveform pattern may provide information
about SVR and visual indication of compromised cardiac out put.
• CXR: - Useful in respiratory illness- airway obstruction,
lung tissue disease, barotraumas, pleural disease.- Evaluation of circulatory abnormality to assess
heart size and presence or absence of CCF.• ECG: - To assess for cardiac arrhythmias.• Echocardiogram:- Noninvasive cardiac imaging- Cardiac chamber size, ventricular wall thickness,
ventricular wall motion, valve configuration, pericardial space, estimated ventricular pressure etc
• Peak expiratory flow rate:- Represents the maximum flow rate generated
during forced expiration.- Decreases in the presence of airway obstruction.