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I
Just
Can’t
Smile
Without
You….
Interhospital Case PresentationApril 2, 2014
PERPETUAL SUCCOUR HOSPITAL
DEPARTMENT OF PEDIATRICS
Ma. Claire R. Granada, M.D.
Pediatric Resident
Objectives
• To present cases of children with dental caries
• To review dental anatomy and development
• To discuss early childhood caries as to its definition, epidemiology, etiology, diagnosis, prevention and its update
• To emphasize the role of the Pediatrician in the prevention, early diagnosis and management of early childhood caries
CASE 1 CASE 2
PROFILE L.T. 13 y.o. male
Cebu City
dental abscess
Ht: 140cm wt: 34.6kg
BMI: 17.6 kg/m2
Z score: -2
M.M. 5 y.o. Female
Cebu City
PSH-OPD palpable purpura
+ abdominal pain
Ht: 117 cm wt: 24 kg
BMI: 17.5 kg/m2
Z score: 1
PRENATAL Unremarkable Unremarkbale
NATAL Delivered Full term via
NSD
Delivered Full term via NSD
POSTNATAL Breastfed until 1 month oldBottle fed until 5 years old(+) carious teeth started at 3 years old
Bottle fed up to presentDaytime 3 bottlesBedtime 5 bottles2 y.o. (+) dental trauma sec to fall4y.o. (+) carious teeth; (+) recurrent dental abscess over central incisors
HEREDOFAMILIAL DISEASE
None HypertensionDiabetes MellitusBronchial Asthma
MANIFESTATIONS 6 months PTA (+) recurrent
toothache tolerated
1 day PTA (+) toothache PS 7/10
(Right premolar)
Afternoon (+) toothache; (+)
fever; (+) swelling of Right
submandibular area
PE:
(+) swelling on right submandibular
area, (+) carious teeth at the right
and left premolar teeth, (+) abscess
on right premolar area
1 day PTC (+) palpable
purpura on both lower
extrimities
AM PTC (+) abdominal pain
PSH OPD:
(+) palpable purpura on lower
ext
(+) epigastric tenderness
(+) carious teeth on upper
incisors, 1st & 2nd molar
DIAGNOSTICS CBC
Hgb - 16.4 Hct - 50.2 Platelet – 244
WBC 10.27 (Neutro - 81, Lympho - 10,
Mono - 8, Eos – 1)
CBC & UA requested
CASE 1 CASE 2
MANAGEMENT Mefenamic acid 500mg/tab;
1 tab q6H PO
Co-amoxiclav 1 gram/ tab;
1 tab BID PO
Discharged on the 3rd hospital
day
Opted to visit her Pediatrician
Advised for dental evaluation
FINAL DIAGNOSIS Dental Abscess, Right
Submandibular area
T/C Henoch-schonlein Purpura
Early Childhood Caries
Anatomy of a Tooth
The tooth consists of a crown and a root.
The crown is visible above the gums.
The root is covered with cementum, which anchors it to the periodontal membrane.
Used with permission from Miller Medical Illustration & Design
•The enamel protects the dentin, a hard, thick substance containing thousands of tubules that surround the nerve.
•These tubules contain tiny projections of the nerve and are sensitive to exposure to air, acid, and touch.
•The pulp is the soft core of the tooth that contains blood vessels, connective tissue, and the nerve itself.
Normal Development • Oral structures begin to form during the third and fourth weeks of embryonic development.
• The teeth begin to develop around the sixthweek of fetal life
• Development continues throughout fetal life and beyond.
Tooth Eruption: Primary Teeth
Primary teeth begin to erupt around 6 months of age.
Eruption is completed by 24 to 36 months.
Delays of more than 12 months merit further dental evaluation.
http://www.aap.org/oralhealth/pact
Delayed Eruption
Delayed eruption of more than 12 months can be caused by:
• Endocrine disorders
• Genetic disorders
• Oral space issues
• Dense gingival tissue
• Dental infection
• Radiation therapy
It is reasonable to refer a child who has not erupted a tooth by 18 months of age
to a dentist if they are not seeing one already.
Tooth Eruption: Permanent Teeth
Eruption for the permanent teeth is similar to that of the primary teeth.
Eruption of the permanent teeth begins between 5 and 7 years of age and usually finishes by 13 to 14 years.
http://www.aap.org/oralhealth/pact
Tooth Eruption: Permanent Teeth
•The typical pattern for the eruption of permanent teeth is:
Central incisors
Lateral incisors
First molars
Premolars
Canines
Second molars
Third molars (wisdom teeth)
http://www.aap.org/oralhealth/pact
Natal and Neonatal Teeth
• Extraction of these incisors may be
considered if they are mobile,
interfere with breastfeeding, or lead
to Riga-Fede ulceration.
Objective # 3
• To discuss early childhood caries as to its definition, epidemiology, etiology, diagnosis, prevention and its update
Early Childhood Caries
ECC is a transmissible infectious process that affects children younger than 5 years and results in tooth destruction.
ECC is a virulent form of caries that rapidly spreads within the mouth.
S-ECC (Severe Early Childhood Caries)
< 3 y.o. any sign of smooth caries
3 – 5 y.o. 1 or more cavitated, missing, or filled smooth surfaces in primary maxillary anterior teeth, or a decayed, missing or filled score of ≥ 4 (age 3), ≥ 5 (age 4), ≥ 6 (age 5) surfaces
PPS Policy Statements Series 2004 Vol2 No.2
Early Childhood Caries
United States 24.7%
58.6 % of all 5 to 17 years old
poor and minority children 70%
Philippines
ECC is at least 1 in 4 (25%) of 5 to 6 years old children
2nd in the whole Western Pacific Region
PPS Policy Statements Series 2004 Vol2 No.2
PHILIPPINE DENTAL ASSOCIATION
< 12 y.o = 78%< 6 y.o = 97%
Affected Teeth
•ECC tends to affect the upper (maxillary) incisors first
•The primary molars are affected next
because of their grooved surfaces.
•Food easily becomes lodged in the
molar’s pits and fissures, which are
difficult areas to clean with
a toothbrush.
• The canines tend to be spared
because they are smooth
teeth that erupt later.
• The lower teeth are better
protected by saliva and the tongue.
http://www.aap.org/oralhealth/pact
Diagnosis and Stages
1. Plaque
2. Incipient lesions or white spots
3. Enamel caries
4. Dentine caries
5. Pulpitis
Factors in Development of ECC
1. Microbial factors
2. Salivary factors
3. Dietary factors
AAP Textbook of Pediatric CareChapter 33: Prevention of Dental Caries
Factors in Development of ECC
1. Microbial factors
2. Salivary factors
3. Dietary factors• S. mutans (30%)
• S. sobrinus
• Actinomyces sp.
• Lactobacillus sp.AAP Textbook of Pediatric CareChapter 33: Prevention of Dental Caries
http://www.aap.org/oralhealth/pact
S Mutans
• S mutans is transmitted from the primary
caregiver to infant by saliva.
• Transmission rates increase when parents:
Share utensils or toothbrushes.
Taste food or drink before serving it.
“Clean” a dropped pacifier with saliva.
Allow a child to place fingers into
an adult's mouth.
Factors in Development of ECC
1. Microbial factors
2. Salivary factors
3. Dietary factors
saliva buffers acid
saliva is bacteriostatic
saliva aids in remineralizing the
teeth by supplying calcium,
phosphate, and fluoride to help
construct enamel.AAP Textbook of Pediatric CareChapter 33: Prevention of Dental Caries
Factors in Development of ECC
1. Microbial factors
2. Salivary factors
3. Dietary factors
AAP Textbook of Pediatric CareChapter 33: Prevention of Dental Caries
1. FLUORIDE
• Water Fluoridation
• Fluoride Supplements
• Fluoride Dentifrice (Toothpaste)
• Fluoride Rinses
• Fluoride Varnish
AAP Textbook of Pediatric CareChapter 33: Prevention of Dental Caries
1. FLUORIDE
• Water Fluoridation
• Fluoride Supplements
• Fluoride Dentifrice
• Fluoride Rinses
• Fluoride Varnish
Recommended level
0.7 to 1.2 ppm fluoride
AAP Textbook of Pediatric CareChapter 33: Prevention of Dental Caries
MCWD
< 1ppm fluoride
Ocean water 1.2 ppm fluoride
Well water 0-7.22 ppm fluoride
1. FLUORIDE
• Water Fluoridation
• Fluoride Supplements
• Fluoride Dentifrice (Toothpaste)
• Fluoride Rinses
• Fluoride Varnish
AAP Textbook of Pediatric CareChapter 33: Prevention of Dental Caries
1. FLUORIDE
• Water Fluoridation
• Fluoride Supplements
• Fluoride Dentifrice (Toothpaste)
• Fluoride Rinses
• Fluoride Varnish
AAP Textbook of Pediatric CareChapter 33: Prevention of Dental Caries
1. FLUORIDE
• Water Fluoridation
• Fluoride Supplements
• Fluoride Dentifrice (Toothpaste)
• Fluoride Rinses
• Fluoride Varnish
AAP Textbook of Pediatric CareChapter 33: Prevention of Dental Caries
1. FLUORIDE
• Water Fluoridation
• Fluoride Supplements
• Fluoride Dentifrice (Toothpaste)
• Fluoride Rinses
• Fluoride Varnish
AAP Textbook of Pediatric CareChapter 33: Prevention of Dental Caries
WHO recommends varnish
applications for all children
2 – 4 x a year
http://www.aap.org/oralhealth/pact
Fluoride Varnish
Application frequency for fluoride varnish
ranges from 2 to 6 times per year.
The use of fluoride varnish leads to a 33%
reduction in decayed, missing, and filled tooth surfaces in the primary
teeth and a 46% reduction in the permanent teeth.
2. ORAL HYGIENE
daily brushing and flossing of teeth
Parents should verify that the child’s teeth are clean before bedtime
because the buffering capacity and antimicrobial action of saliva
decreases during sleep with diminished secretion of saliva.
AAP Textbook of Pediatric CareChapter 33: Prevention of Dental Caries
3. SEALANTS
effective in preventing pit and fissures caries
AAP Textbook of Pediatric CareChapter 33: Prevention of Dental Caries
Summary of evidence-based clinical recommendations regarding pit-and-fissure sealants.
JADA, Vol. 139 http://jada.ada.org March 2008
4. Diet
↓ frequency of cariogenic substrate ingestion
use of gum, candy, and soft drinks containing sugar substitutes
(mannitol, sorbitol, xylitol and aspartame)
infants should be weaned from the bottle by 1 year of age to
eliminate their risk for ECC
bedtime and naptime nursing bottles should contain only water
AAP Textbook of Pediatric CareChapter 33: Prevention of Dental Caries
Jeff Burges et al., Diet and Oral Health; Medscape article, updated: May 4, 2012
Dietary Factors Causing Dental Caries
Food with Anticaries Activity
• green tea
• Apple
• Red grape seeds
• Red wine
• Nutmeg
• Ajowan craway
• Coffee
• barley coffee
• chicory
• mushroom
• cranberry
• Glycyrrhiza root
• ethanolic ectract of Myrtuscommunis
• garlic aqueous extract
• cocoa extracts
• propolis
Jeff Burges et al., Diet and Oral Health; Medscape article, updated: May 4, 2012
Caries Prevention: Three Materials Found Equivalent
1. Silver diamine fluoride
2. Resin-modified glass ionomer
3. Resin-based sealant
Laird Harrison, Caries Prevention: Three Materials Found Equivalent; Medscape article, March 22, 2013
Risk assessment tools
aid in the identification of reliable predictors and allow
dental practitioners, physicians, and other nondental health
care providers to become more actively involved in in
identifying and referring high-risk children.
Clinical management protocols
• assist in clinical decision-making
• provide criteria regarding diagnosis and treatment
• recommended courses of action
Treatment
low risk may not need any restorative therapy
moderate risk may require restoration of progressing and cavitated lesions, while white spot and enamel proximal lesions should be treated by preventive techniques and monitored for progression
high risk require earlier restorative intervention of enamel proximal lesions, as well as intervention of progressing and cavitatedlesions
Hakan et al; J Nat Sci Biol Med. 2013 Jan-Jun; 4(1): 29–38.
Treatment
Silver amalgam
Plastic composite
Stainless steel crowns
Pulpotomy
Pulpectomy
Tooth extraction
Penicillin
Clindamycin
Erythromycin
Ibuprofen
Nelson textbook of PEDIATRICS 19th ed; ch 304 p 1255
Objective # 4
•To emphasize the role of the Pediatrician in the prevention, early diagnosis and management of early childhood caries
Role of the Pediatrician
Pediatricians should routinely question parents about feeding behaviors
and make recommendations that promote dental health
Examination of the dentition should be performed on every child as a
part of a routine physical examination
Any child with evidence of cavitation, stained fissures, or areas of
enamel decalcification should be referred to a dentist immediately
Every child should be evaluated by a dentist by 12 months of age
AAPD Guideline on Fluoride Therapy; revised 2013
Interhospital Case PresentationApril 2, 2014
Discussant: Ma. Claire R. Granada, M.D.
Pediatric Resident
PERPETUAL SUCCOUR HOSPITAL
DEPARTMENT OF PEDIATRICS