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STI College of Nursing
Sta. Cruz, Laguna
College of nursing
Submitted to:
Mrs. Aurea Celino, RN
Clinical Instructor
Submitted by:
GROUP III
Members:
Avanzado, leonel
Dawinan, Paullette Edilyn
Del Rosario, Jason
Mallari, Kim
Miras, Roxanne
Napiza, Charlene Diane
Salvador, Leomar
Valenzuela, Abrelaine
December 6, 2010
1
TABLE OF CONTENTS
I. Introduction --------------------------------------------------------------------------------3
A. Objectives -------------------------------------------------------------------------------5
B. Background of the Study ------------------------------------------------------------5
C. Rationale for Choosing the Case -------------------------------------------------6
D. Significance of the Study -----------------------------------------------------------7
E. Scope and Limitations ---------------------------------------------------------------8
II. Clinical Study ------------------------------------------------------------------------------9
A. Demographic Data --------------------------------------------------------------------9
B. Physical Assessment -----------------------------------------------------------------10
C. System Affected -----------------------------------------------------------------------13
D. Laboratory and Diagnostic Exam -------------------------------------------------13
III. Clinical Discussion -----------------------------------------------------------------------16
A. Anatomy and Physiology -------------------------------------------------------------16
B. Pathophysiology ------------------------------------------------------------------------19
C. Nursing Care Plan ----------------------------------------------------------------------20
D. Drug Study -------------------------------------------------------------------------------26
E. Course in the Ward --------------------------------------------------------------------35
F. Discharge Plan --------------------------------------------------------------------------37
G. Evaluation --------------------------------------------------------------------------------38
H. Summary ---------------------------------------------------------------------------------38
I. Recommendation -----------------------------------------------------------------------39
2
I. INTRODUCTION
CEREBROVASCULAR ACCIDENTA stroke, previously known medically as a cerebrovascular accident (CVA), is the
rapidly developing loss of brain function(s) due to disturbance in the blood supply to the
brain. This can be due to ischemia (lack of blood flow) caused by blockage
(thrombosis, arterial embolism), or a hemorrhage (leakage of blood). As a result, the affected
area of the brain is unable to function, leading to inability to move one or more limbs on one
side of the body, inability to understand or formulate speech, or an inability to see one side of
the visual field.
A stroke is a medical emergency and can cause permanent neurological damage,
complications, and even death. It is the leading cause of adult disability in the United States
and Europe and it is the number two cause of death worldwide.
The severity associated with cerebrovascular accident can best be demonstrated by the
following facts:
CVA is the leading cause of adult disability in the world.
Worldwide, one-quarter of all strokes are fatal.
Stroke is the third leading cause of death in the United States and the leading cause
of disability.
It is estimated that four of every five families in the United States will be affected by
stroke in their lifetime,
More than half a million people in the United States experience a new or recurrent
stroke each year.
Stroke kills about 150,000 Americans each year, or almost one out of three stroke
victims.
Three million Americans are currently permanently disabled from stroke.
In the United States, stroke costs about $43 billion per year in direct costs and loss of
productivity.
Two-thirds of strokes occur in people over the age of 65.
Strokes affect men more often than women, although women are more likely to die
from a stroke.
Strokes affect African Americans more often than Caucasians, and are more likely to
be fatal among African Americans.
3
The incidence of strokes among people ages 30 to 60 is less than 1%. This figure
triples by the age of 80.
The rate of occurrence for strokes in the United States fell by 15.52% between 1988
and 1998. But the number of deaths from stroke actually rose by 5%.
According to the World Health Organization, one in ten of the 55 million deaths that
occur every year worldwide are due to stroke and two-thirds of which occur among
people living in developing countries). In the Philippines, stroke remains to be a
leading cause of disability, afflicting 400,000 Filipinos yearly
(Manila Bulletin, 13 September 2004).
What Are the Risk Factors for Developing Stroke?
1) smoking
2) excessive alcohol intake
3) uncontrolled high blood pressure
4) high cholesterol
5) overweight/unhealthy diet
6) illegal drugs/abuse of Rx drugs
7) known or unknown heart problems
8) diabetes
9) known or unknown vascular brain defects - aneurysm, etc.
10)family history of stroke
4
A. OBJECTIVES
GENERAL OBJECTIVE
This study aims to:
Have a further study about cerebrovascular accident (CVA) or stroke.
Perform physical assessment to the patient with this kind of ailment.
Understand the pathophysiology of the disease, and how it affects our body
system.
Formulate nursing care plan for this patient with CVA.
Have further knowledge about the different medicines and treatment modalities
about this disease.
Give proper information through health teaching, rendering appropriate nursing
interventions to the patient about his present conditions are the main objectives of
this study.
B. BACK GROUND OF THE STUDY
MR. CVA is a 68 years old tricycle driver from Mabitac, Laguna. The patient was
rushed to the Emergency Room last November 25, 2010 at 12:30am with complaints of
chilling, chest pain, nausea and vomiting. The patient has been diagnosed with
cerebrovascular accident (CVA) or stroke and was admitted at the hospital.
We decided to present this case for the reason that we want to learn and explore
new knowledge and information about this type of condition. Our group wants to improve
our skill in rendering care with this kind of client.
5
C. RATIONALE FOR CHOOSING THE CASE
Nurses in all settings perform a cruical role in assessing the patient, helping prevent
the occurence of possible complications and rendering supportive nursing care. Thus, it is
vital that we, as future nurses be enlighten with significant information that we may use, as
we render health encouraging services to our client.
This presentation has been chosen by the group under the following reasons:
1. Assess the patient’s family history and identify risk factors for developing the disease.
2. Apply a nursing process to the patient under the students care and resolve problem,
implement the plan and evaluate the progress of the plan.
3. Identify the appropriate nursing care interventions in managing the clinical signs and
symptoms that manifest in patient.
4. Utilize effective communication techniques for educating the client and the family.
5. Determine factors and conditions that may influence to the development of the disease.
Further enhance knowledge and understanding regarding the nature of the disease.
6
D. SIGNIFICANCE OF THE STUDY
This would serve as an important source to the following sectors:
To the students:
This study discusses Cerebrovascular Accident, its definition, causes,
manifestation and proper treatment which will broaden our understading of the
disease.
This would also improve our knowledge in the anatomy and physiology of the
Nervous system and how its directly affected by the disease.
To the Family and Patient:
This study can also benefit the patient through better understanding of the
underlying causes of the diseases and to have knowledge on proper managament of
the illness.
This can also provide the patient on how to deal with the possible signs and
symptoms and other noticeable changes might observed.
7
D. SCOPE AND LIMITATION
This research study primarily focused on the different nursing intervention and health
teaching that we students nurses can give/advice our patients to help alleviate pain and
discomfort. This study also focused on the nature and occurances of the diagnosis, as
well as the primary factors present in the environment that may worsen the situation.
We, as student nurses are limited in formulation of the best nursing care plan
for our patient. We also focused on the the different types of medication prescribed by the
physician,for us to know and understand its effect to our patient. Therapeutic nurse
patient interaction was also established to be able to render the best possible care.
8
II. CLINICAL STUDY
DEMOGRAPHIC DATA
Patient Name : MR. CVA
Age : 68 years old
Sex : Male
Birth Date : April 15, 1942
Address : Mabitac, Laguna
Date of Admission : November 25, 2010
Time of Admission : 12:30am
Admitting Diagnosis: Cerebrovascular Accident
Source of Information
Secondary Source : Patient’s Relative
Chief Complaint: “nanginig, nagsuka at sumakit ang dibdib ng pinsan ko kaya namin sya
dinala sa hospital.” As verbalized by the patient’s relative.
History of Present Illness
Few days prior to admission according to his cousin MR. CVA was complaining of
experiencing dizziness, headache and mild chest pain. When MR. CVA was rushed to the
hospital he was having chills, nausea, vomiting, and mild chest pain. The Physician on duty
who have seen and examined MR. CVA decided to admit him in Medicine Ward.
9
History of Past Illness
MR. CVA has suffered from stroke previously; he had his first stroke last 2000. His
cousin said that MR. CVA had recovered from the first accident without any permanent
damage or complication and then eight years after the first accident he had his second attack
on the year 2008. Fortunately MR. CVA recovered fully after hospitalization and treatment.
His third attack of stroke happened last November 25, 2010 and he was then admitted at the
Medicine ward.
Dietary History
Prior to the episode of his first stroke, MR. CVA was fond of eating fatty foods like pork
chop, meat viands and chicken skin. He loves to eat rice and fried foods. After suffering from
stroke MR. CVA began to regulate his diet. His relatives also prepared for him healthy foods
such as vegetables and fish and set a limit with their intake of meat and fried foods.
History of Elimination pattern
Output: 900 cc level of urine at urine bag for 24 hours. (Nov. 30, 2010)
400 cc level of urine at urine bag @ 12 noon.
Color: Amber Red.
B. PHYSICAL ASSESSMENT
Levels of consciousness: Glasgow coma scale
Faculty measured
Eye opening: 3
Motor response: 3
Verbal responses: 2
Total: 8
10
General Appearance
- With flushes, dry skin
- Medium body built
- With IVF of @ right metacarpal vein
- With oxygen therapy regulated @ 10 LPM delivered by
face mask
- With NGT ( Nasogastric tube)
Head
- Normocephalic
- symmetrical in shape
- no masses, no lesions, no tenderness
Hair - evenly distributed over the scalp
- no dandruff
- Hair with few gray hairs.
Eyelids - with water eyelids
- no edema, and no discharges
Sclera - yellowish
Iris - symmetrical in size
- round and black
Pupils
- Symmetrical in size
- round and dark brown in color
- PERRLA (Pupils Equally Round And React To
Light and Accommodation)
Lower conjunctiva - Moist
Ears
- equal in size
- auricles are smooth and symmetrical
- pinna recoils after it is folded
Nose
- the external nose is symmetrical and straight
- color is the same with the entire face
- lesions and tenderness were both absent
- nasal septum is intact and in midline without
deviations
11
- cilia present in internal nares
- absence of nasal discharge
- with oxygen mask
Mouth
- with cyanotic lips
- dry oral mucous membrane
- dentures are removed
Neck
- free from lumps and no tenderness
- at the center and midline
Thorax
- skin color was not evenly distributed
- no masses and tenderness upon palpation
- with wheezing sounds
- With irregular lub-dub sound * cannot be distinguished.
Breast
- round in shape, no lumps, no masses
- areola dark brown in color
- nipples round, equal in size
Abdomen
- skin color evenly distributed
- no lumps , masses and tenderness
Upper extremities
- skin uniform in color
- with IVF @ right metacarpal vein
- Left arm paralyzed ( limb ataxia)
- Capillary refill: 2-3 seconds
Lower extremities
- Skin uniform in color
- Left leg paralyzed
- Sole of the foot is pale
- No Deep tendon reflex
Genitalia
- With indwelling catheter
12
C. SYSTEM AFFECTED
Cardiovascular system Increased BP
Nervous system Hemiplagia Limb ataxia Dysarthia Numbness
Respiratory system Increased RR
D. LABORATORY AND DIAGNOSTIC EXAM
HEMATOLOGY
COMPONENT RESULT NORMAL VALUES
SIGNIFICANCE INTERPRETATION
WBC (x109/L) 23.2 4.5 – 10.5 x 109 /L High Increased due to infection.
SEGMENTED NEUTROPHILS
87 45 – 65% High
MONOCYTES 18 2 – 10% High Increased in viral infections.
RBC (x1012/L) 4.16 5.5 – 6.5 x 1012/L Low Decreased in anemia.
MCH (pg) 22 27 – 32 pg Low Decreased in iron deficiency anemia.
MCHC (g/L) 24.4 32.0 – 36.0 g/L Low Decreased in iron deficiency anemia
Evaluation of Blood Smear: TROPONIN E – NEGATIVE
BLOOD ANALYSIS REPORT
COMPONENT RESULT NORMAL VALUES INTERPRETATION SIGNIFICANCE
13
PH 7.51 7.35 – 7.45 High Indicates alkalosis.
PCO2 25 mm Hg 35 – 45 Low Indicates respiratory alkalosis (hyper or over ventilation).
PO2 90 mm Hg 80 – 100 Normal Normal
HCO3 20 mmol/L 22 – 26 Low Indicates metabolic acidosis.
TCO2 21 mmol/L 23 – 27 Low Indicates respiratory alkalosis
BE ecf -3 mmol/L (-2) – (+2) Low A negative base excess indicates metabolic acidosis (primary or secondary to respiratory alkalosis).
SO2 92% 95 – 100% Low Indicates that the hemoglobin is deoxygenated.
Readings: Uncompensated Respiratory alkalosis with unconnected oxygenation.
COMPONENT RESULT NORMAL VALUES INTERPRETATION SIGNIFICANCE
BUN kinetic UV (Blue)
9.8 mmo/L 2.5 – 6.4 High The BUN levelis significantlyabove thenormal limitswhich denotesan impairment in renal functions.
Creatinine Kinetic (2 *
133.7 umo/L 35.4 – 123.8 High The creatininelevel issignificantlyabove thenormal limitswhich is aresult of renalimpairmentrelated to theclient’s activerenal disease.
Direct HDL 0.214 mmo/L 0.700 – 2.200 Low Indicates heightened risk for heart disease.
ECG ANALYSIS RESULT
14
819 Undefined Tachycardia
81 A Undefined Arrhythmia
303 High Voltage (Right Ventricular)
701 Poor R Progression
401 Short P-R Internal
618 Unspecified ST – T Abnormalities
III. CLINICAL DISCUSSION
A. ANATOMY AND PHYSIOLOGY
Circulatory
system
15
NORMAL BLOOD CIRCULATION
BLOOD from the superior and inferior vena cava enters to the right atrium and
passed the tricuspid valve.
After the blood passed the tricuspid valve it flows to the right ventricle and
enters the pulmonary valve then flows to the pulmonary artery into the lungs.
The blood that enters the lungs will be oxygenated and by passing the
pulmonary vein it goes to left atrium.
And then it will passed the bicuspid valve to the left ventricle and pass the
aortic valve through aorta then the blood is ready for distributing and collecting
carbon dioxide to the tissues and organs.
Nervous system
FUNCTIONS OF THE NERVOUS SYSTEM
1. Sensory input- sensory receptors monitor numerous external and internal
stimuli that may be
interpreted as touch,
temperature, taste, smell,
sound, blood pressure and
body position. Action potential
from the sensory receptors
travel along nerves to the
spinal cord and brain, where
they are interpreted.
16
STIMULUS
Sensory division conducts action potentials from the periphery to the
CNS
CNS processes and integrates information, initiates response, and
carries out mental activity
Motor division conducts action potentials to the periphery
2. Integration- the brain and the spinal cord are the major organs processing
sensory input and initiating responses. The input may produce an immediate
response, may be stored as memory, or may be ignored.
3. Homeostasis- the nervous system plays an important role in maintenance of
homeostasis. This function depends on the ability of the nervous system to
detect, interprets, and responds to the changes in the internal and external
conditions. In response, the nervous system can stimulate or inhibit the
activities of other system to help maintain a constant internal environment.
4. Mental activity- the brain is the center of the mental activity, including
consciousness, memory and thinking.
5. Control of muscles and glands- skeletal muscle normally contract only when
stimulated by the nervous system. Thus, through the control of skeletal muscle,
the nervous system controls the major movement of the body. The nervous
system participates also in controlling cardiac muscle, smooth muscle, and
many glands.
Organization of the nervous system
17
PREDISPOSING FACTORAge: 68 yrs.old,
Sex: maleFamily history of hypertension 2 times Hx of stroke accident.
PRECIPITATING FACTORHypertension
Lifestyle: Lack of ExerciseDiet: Eating Foods Rich in Cholesterol
Deposition of fats in the artery of the brain or neck
Narrowing of the vein occurs
Occlusion of the artery in the brain or neck
Disruption of the blood flow
Ischemic cascade occurs * cellular metabolic events
Oxygen on the neurons (aerobic respiration of the neurons fails)
Switching of the mitochondria into anaerobic respirations
Production of the lactic acid
pH is changed
Acidosis occurs
Insufficiency in the production of ATP
Electrolyte ion imbalance occurs
Depolarization of the cells
Intracellular calcium increased Depolarization of the cell
Increased of glutamate
Cell membrane and protein breakdown, formation of free radicals, protein production
Cell injury and death occurs
Dizziness
Body temperature
B. PATHOPHYSIOLOGY OF CVA
18
19
C. NURSING CARE PLAN
HYPERTHERMIA
ASSESSMENT NURSING DIAGNOSIS
PLANNING INTERVENTION RATIONALE EVALUATION
Subjective: “Nilalagnat ang pinsan ko.” As verbalized by the patient’s relative.
Objective:
- Febrile- Warm to
touch- Flushed
skin- Temp: 37.7
0 C
Hyperthermia related to increased intracellular calcium as evidenced by elevated body temperature.
After 8 hours of nursing interventions the client body temperature will be decreased from 37.7 0C to the normal range.
1. Monitor vital signs.
2. Noted body temperature
3. Provide tepid sponge bat (if not contraindicated).
4. Promote ventilation ofskin by means of undressing or unclothing
1. Regular temperaturemonitoring will identifyadequatethermoregulation.
2. Noting the body temperature indicates and show the progress of the client’s condition
3. To promote cooling of body surface.
4. To maintain stablebody temperature of
After 8 hours of nursing interventions the clients body temperature remain 37.7 0 C. GOAL UNMET.
20
(heat loss by radiation andconduction).
5. Administered medication (antipyretics) e.g paracetamol, as prescribed.
the clients
5. Medication such as antipyretics can decrease and normalized body temperature.
21
IMPAIRED PHYSICAL MOBILITY
ASSESSMENT NURSING DIAGNOSIS
PLANNING INTERVENTION RATIONALE EVALUATION
Subjective: “Hindi makagalaw ang lolo ko simula noong ma-stroke siya” as verbalized by the patient’s relative.
Objective:
(+) general body weakness
(+) Paralysis of left side of the body (hemiplagia)
(+) lethargy Functional
level scale: 0 (completely independent)
Inability to perform gross/fine motor skills
Impaired physical mobility related to disturbance of upper motor neuron in one part of the brain (neurovascular impairment) as evidenced by hemiplagia.
After 8 hours of nursing interventions the patient’s:
S/O will demonstrate techniques that will enable safe repositioning.
1. Determine diagnosis that contributes to immobility.
2. Assist or have S/O reposition client on a regular schedule by turning to side every 3 hours (as ordered by the physician).
3. Provide safety measure such as putting side rails up, or using pillows to support body part.
1. To identify causative/contributing factors.
2. To prevent complications like bedsore.
3. To provide safety.
After series of nursing intervention the patient relatives:
Relatives can demonstrated techniques that will enable safe repositioning.
GOAL MET.
22
4. Involve S/O in care, assisting them to learn ways to managing problems of immobility.
5. Teach relatives to perform passive Range of Motion Exercises
4. To impart health teaching to the patient’s family.
5. To enhance blood circulation and prevent contractures
23
IMPAIRED VERBAL COMMUNICATION
ASSESSMENT NURSING DIAGNOSIS
PLANNING INTERVENTION RATIONALE EVALUATION
Subjective: “Hindi makapagsalita ng maayos ang lolo ko” as verbalized by the patient’s relative.
Objective:
Difficulty producing speech
Dysarthria Presence of
face mask
Impaired verbalcommunicationrelated to loss offacial or oral muscletone control as evidenced by difficulty speaking.
After 1 hr. of nursingintervention, thepatient will establishmethod ofcommunication inwhich needs can beexpressed
1. Provide alternativemethods ofcommunication, likepictures or visual cues,gestures ordemonstration and Hand gestures
2. Talk directly to patient.Speaking slowly anddirectly. Use yes or noquestion to begin with.
3. Speak in normal tones
1. Providecommunication needsor desires based onindividual situation orunderlying deficit.
2. It reduces confusionor anxiety and havingto process andrespond to largeamount ofinformation at onetime.
3. Patient is notnecessary hearing
After 1 hr. ofnursingintervention, thepatient was able toestablish method ofcommunication inwhich needs can beexpressed.
24
and avoid talking too fast.Give patient ample timeto respond.
4. Encourage familymembers and visitors topersist efforts tocommunicate with thepatient.
impaired and raisingvoice may irritate oranger the patient
4. It is important forfamily members tocontinue talking tothe patient to reducepatients isolation,promoteestablishment ofeffectivecommunication andmaintain sense ofconnectedness orbonding with thefamily
25
D. DRUG STUDY
GENERIC NAME
BRAND NAME
CLASSIFICATION
MECHANISM OF ACTION
INDICATION CONTRAINDICATION
ADVERSE REACTION
NURSING RESPONSIBLITY
Acetyl Salicylic Acid
Dosage: 80mg tabOnce a day
ASA, Aspergum, Aspercin
Pharmacologic Class: NSAIDs
Therapeutic Class:Non opioid Analgesic, Antipyretic, Anti platelet drug
Reduces pain and inflammation by inhibiting prostaglandin production. Fever reduction mechanism unknown; may be linked to decrease in endogenous pyrogens in hypothalamus resulting from prostaglandin inhibition. Exerts anti platelet effect by inhibiting synthesis of prostacyclin and thromboxane A2.
Mild pain or fever
Mild to moderate pain caused by inflammation
Acute rheumatic fever
To reduce the risk of transient ischemic attacks or cerebrovascular accident in men with a transient ischemic attack caused by emboli
To reduce the risk of myocardial infarction in patients with history of MI or unstable angina
Thromboembolic disorders.
Hypersensitivity to salicylates, other NSAIDs
Renal impairment
Severe hepatic impairment
Vitamin K deficiency caused by dehydration
Pregnancy or breastfeeding
Concurrent anticoagulant use
EENT: hearing loss
GI: nausea, vomiting
Hematologic:thrombocytopenia, leukopenia
Hepatic: hepatotoxicity
Respiratory: wheezing
Skin: rash, urticaria
Tell to relatives of the patient to report ototxicity symptoms, unusual bleeding and bruising
Instruct the relative of the patient to tell all prescribers he’s taking the drug because it may cause serious interactions with many common medicines
Tell patien’s relative not to take over the counter preparations containing aspirin
GENERIC NAME
BRAND NAME
CLASSIFICATION MECHANISM OF ACTION
INDICATION CONTRAINDICATION
ADVERSE REACTION
NURSING RESPONSIBLITY
26
Isordil
Dosage: 5 tabletssublingual
Isosorbide Dinitrate
Pharmacologic Class:Nitrate
Therapeutic Class:Anti anginal
Promotes peripheral vasodilation and reduces preload and afterload, decreasing myocardial oxygen consumption and cardiac output. Also dilates coronary arteries, increasing blood flow and improving collateral circulation
Treatment and prophylaxis in situations likely to provoke acute angina pectoris.
Prophylaxis of angina pectoris.
Hypersensitivity to drug
Severe anemia
Acute myocardial infarction
CNS: dizziness,headache
CV: tachycardia, orthostatic hypotension
GI: nausea , vomiting
SKIN: flushing
Teach patient relative to give oral drug 30 mins. before or 1 to 2 hours after a meal
Inform patient relative that drug may cause headache.
Advise his relative to treat headache as usual and not to alter drug schedule.
GENERIC NAME
BRAND NAME
CLASSIFICATION MECHANISM OF ACTION
INDICATION CONTRAINDICATION ADVERSE REACTION
NURSING RESPONSIBLITY
Acetaminophen
DOSAGE:Ampule IV q4h
PARACETAMOL
Pharmacologic Class:Synthetic non opioid p- aminophenol derivative
Therapeutic class:analgesic, antipyretic
Unclear. Pain relief may result from inhibition of prostaglandin synthesis in cns, with subsequent blockage of pain impulses. Fever reduction may result from vasodilation
Mild to moderate caused byheadache, muscle ache, backache, mild arthritis, common cold, toothache, and fever.
Hypersensitivity to drug.
HEMATOLOGIC: thrombocytopenia, neutropenia, hemolytic anemia
HEPATIC: Jaundice, hepatotoxicity
SKIN: rash. urticaria
Tell patient, parents, or other caregivers not to use drug concurrently with other acetaminophen-containing products.
Advise patient , parents or other caregivers to contact prescriber
27
and increased peripheral blood flow in hypothalamus, which dissipates heat and lowers body temperature.
METABOLIC: hypoglycemic coma
OTHERS: hypersensitivity reaction
if fever or other symptoms persist despite taking recommended amount of drug.
Inform patient with chronic alcoholism that drug may increase severe liver damage.
As appropriate, review all other significant and life threatening adverse effect and interactions and behaviors mentioned above.
28
GENERIC NAME
BRAND NAME
CLASSIFICATION MECHANISM OF ACTION
INDICATION CONTRAINDICATION ADVERSE REACTION
NURSING RESPONSIBILITY
Berodual
Dosage:Nebule q6h
Ipratropium Bromide
Anti cholinergic drug, Anti asthmatic
It blocks the muscarinic cholinergic receptors in the smooth muscles of the bronchi in the lungs. This opens the bronchi, and provides relief in chronic obstructive pulmonary disease and acute asthma.
Prevention and treatment of symptoms in chronic obstructive airway disorders with reversible bronchospasm (eg bronchial asthma and chronic bronchitis.
Hypertrophic obstructive cardiomyopathy, tachyarrythmias. hypersensitivity to atropine like substance.
Nervousness. Restlessness, tachycardia, palpitations, dizziness, headache. dry mouth and cough.
Instructed the clients relative on how to use of inhaler, nebulizer.
Advised the patients relative to use dry cotton and tap the lips to avoid dryness.
Instructed the clients relative to do back tapping or vibration after the medication.
GENERIC NAME
BRAND NAME
CLASSIFICATION
MECHANISM OF ACTION
INDICATION CONTRAINDICATION ADVERSE EFFECTS
NURSING RESPOSIBILITY
29
Digoxin
Dosage:20 g OD.
Lanoxin Antiarrythmic, inotropic
Increase force of and velocity of mayocardial contraction and prolongs refractory period of AV node by increasing calcium entry into myocardial cells. Slows conduction through sinoatrial and AV nodes and produces antiarrythmic effect.
Heart failure; tachyarrythmias; atrial fibrillation and flutter; paroxysmal atrial tachycardia.
Tab.20mg OD
Hypersensitivity to drugs
Uncontrolled ventricular arrhythmias
AV block
Idiopathic hypertropicsubaortic stenosis
Constrictive pericarditis
CNS: fatigue, headache, astheniaCV: bradycardia, ECG changes, arrhythmiasEENT: blurred or yellow visionGU: gynecomastiaGI: n/v, diarrhea
Check PR regularly, if it’s below 60 or above 110bpm/min, tell him to withhold dose and notify the prescriber.
Instruct patient relative not to take OTC drugs without prescribers approval
Teach patient relative on how to recognize and report signs and symptoms of digoxin toxicity
GENERIC NAME
BRAND NAME CLASSIFICATION
MECHANISM OF ACTION
INDICATION CONTRAINDICATION
ADVERSE EFFECTS
NURSING RESPOSIBILITY
30
Mannitol
Dosage: 100 cc every q6 hours.
Osmitrol, resectisol
Diuretics Enhance water flow from various tissues and ultimately decrease intracranial and intraocular pressure. Also protects kidneys by preventing toxins from forming and blocking tubules.
To prevent acute renal failure during cardiovascular and other surgeries 50-100g I.V. infusion as 5%-25% solution.
To reduce intracranial pressure and brain mass. 0.5-2g/kg I.V infusion as 15-25% given over 30-60 mins.
To promote dieresis in drug toxicity 25g I.V. infusion as loading dose, followed by infusion of 5-25% solution given continuously to maintain high urine output.
Active intracranial bleeding
Anuria secondary to severe renal disease
Progressive heart failure, pulmonary congestion, renal damage or renal dysfunction after mannitol therapy begins
Severe dehydration
CNS: dizziness, headache, seizureCV: chest pain, hypotension, hypertension, tachycardia, thrombophlebitis, heart failure, vascular overloadEENT: blurred vision, rhinitisGI: n/v, diarrhea, dry mouthGU: polyuria, urinary retentionRespi: pulmonary congestionSkin: rash, urticaria
Teach patient relative about importance of monitoring exact urinary output.
Advise patient relative to report adverse reaction, such as increase shorthness of breath or back
Tell patient relative that drug may cause thirst or dry mouth.
GENERIC NAME
BRAND NAME CLASSIFICATION
MECHANISM OF ACTION
INDICATION CONTRAINDICATION
ADVERSE EFFECTS NURSING RESPOSIBILITY
Furosemide
Dosage: 20 mg IV
Furoside, lasix, novosemide
Diuretics, antihypertensive
Unclear. Thought to inhibit sodium and chloride reabsorption from ascending loop of Henle and distal renal tubules, promoting renal excretion of water, sodium, chloride,
Edema caused by heart failure, hepatic cirrhosis, or renal diseaseAdults: initially, 20-80 mg/day PO as a single dose; may increase 20-
Hypersensitivity to drug or other sulfonamides
Anuria
CNS: dizziness, headache, vertigo weakness, lethargy, restlessness, light headednessCV: hypotension, tachycardiaEENT: blurred vision, xanthopsia, hearing loss, tinnitus
Take the drug in the morning with foods to prevent nocturia.
Instruct S/O to report s/sx of ototoxicity.
31
magnesium, hydrogen and calcium
40mg increments PO q 6-8 hrs until desired response occur.
GI: n/v, constipation, dyspepsia, cramping, anorexia, dry mouthGU: excessive and frequent urination, nocturia.Musculoskeletal: muscle pain and muscle cramps
Caution patient relative not to give any alcohol content beverages and herbs while taking this drug.
GENERIC NAME
BRAND NAME
CLASSIFICATION
MECHANISM OF ACTION
INDICATION CONTRAINDICATION
ADVERSE EFFECTS
NURSING RESPOSIBILITY
32
Ceftazidime
Dosage:IV q8 hours
Ceptaz, portaz, tazidime
Anti-inffective
Interferes with bacterial cell wall synthesis and division by binding to cell wall, causing cell to die. Active against gram negative and gram positive bacteria, with expanded activity against gram negative bacteria. Exhibits immunosuppressant activity.
Skin infection, bone and joint infection urinary tract and gynaecologic infection; adults and children ages 12 and older: 500mg to 2 g. IV or IM q 8-12hrs. UTIs 500mg q 8-12hrs. IV or IM
Hypersensitivity to cephalosporins and penicillin
CNS: headache, lethargy, confusion, syncope seizuresCV: hypotension, palpitation, chest pain vasodilationEENT: hearing lossGI: n/v, abdominal crampingGU: vaginal candidiasisRespi: dyspneaSkin: urticaria, maculopapular rash
Instruct patient relative to report reduced urine output, persistent diarrhea, bruising and bleeding
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E. COURSE IN THE WARD
November 30, 2010
Received client on a bed lying down asleep with IVF of 200cc of 0.9
NaCl in SS level of 70cc of mannitol and with oxygen regulated at 10
LPM. IVF infusing well.
DATE TIME VITAL SIGNS NURSES OBSERVATION
November 30, 2010 8:00 am
12:00 noon
BP: 120/ 90 mmHg
PR: 62 bpmRR: 30 cpmTemp: 37.7 O C
BP: 120/ 90 mmHg
PR: 62 bpmRR: 30 cpmTemp: 37.7 O C
- Asleep
- Febrile
- With flushed skin
- Warm to touch
- With cyanotic lips
- DOB
- Lethargic
- Hemiplagia
- Limb ataxia
- Pale sole of the foot
- With indwelling
catheter
- Dysathria
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December 1, 2010
Received client on a bed lying down asleep with IVF of 275cc of 0.9
NaCl in SS level of 81cc of mannitol and with oxygen regulated at 10
LPM. IVF infusing well.
DATE TIME VITAL SIGNS NURSES OBSERVATION
December 1, 2010 8:00 am
12:00 noon
BP: 140/ 100 mmHg
PR: 71 bpmRR: 36 cpmTemp: 38.0 O C
BP: 140/ 100 mmHg
PR: 98 bpmRR: 41 cpmTemp: 38.8 O C
- Asleep
- Febrile
- With flushes skin
- Warm to touch
- With cyanotic lips
- DOB
- Lethargic
- Hemiplagia
- Limb ataxia
- Pale sole of the foot
- With indwelling
catheter
- Dysathria
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DISCHARGED PLAN
M-EDICATION
Patient has to take his medication on time.
E-XERCISEThe patient was advised to have complete bed rest until strength is regained. Have
turn side to side every 2 hours to prevent bed sores. Have ROM exercise on to enhance client's body function.
T-REATMENTInsist physical therapy for improving strength and walking. Undergo Occupational
therapy for regaining dexterity of the arms and hands. The client should go through speech therapy to learn talking and swallowing. Oxygen inhalation should be available if necessary.
H-EALTH TEACHINGTeach the client how to have a healthy lifestyle. Teach patient the foods to eat and
the foods to avoid. Teach the family members how to prepare low sodium and low fat diet.Encourage environmental modification to enhance safety and prevent injury.
O-UT PATIENT
The client was advised to have a follow-up check-up, as indicated by the physician.
D-IET
Patient was instructed to maintain the low salt and low fat diet. The low salt diet is
designed to induce a loss of sodium and water from the body or avoid sodium retention. A
2000 mg low sodium diet is sufficient to control blood pressure. A low fat diet help lose
weight to decrease risk of having CVA again.
S-SPIRITUAL ADVICE
The patient needs a spiritual advice by their relative to have faith in GOD that his
illness will lessen, and only GOD can cure his pain with the help of medicine.
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F. EVALUATION
The nursing process and procedure rendered to the patient is accordingly.
Health teaching and nursing interventions were done properly and effective. In order to
provide care our group applies the knowledge, skills and right attitude for caring for our
patient. In terms of rendering and giving independent intervention, we as student nurses
believe that we have done our best to be able to provide our outmost care. The client was
admitted in the hospital because he suffered from chills, dizziness, nausea and vomiting. The
Physicians diagnosis was Cerebral Vascular Accident (CVA) or stroke. He is confined in the
hospital for more than seven days. We assisted this patient and made some independent
nursing interventions to help alleviate his conditions and to be of assistance to the client’s
relatives.
G. SUMMARY
This study focused on Cerebral Vascular Accident (CVA). The condition is caused by
the interruption of the blood supply to the brain, usually because a blood vessel bursts or is
blocked by a clot. This cuts off the supply of oxygen and nutrients, causing damage to the
brain tissue.
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The most common symptom of a stroke is sudden weakness or numbness of the face, arm
or leg, most often on one side of the body. Other symptoms include: confusion, difficulty
speaking or understanding speech; difficulty seeing with one or both eyes; difficulty walking,
dizziness, loss of balance or coordination; severe headache with no known cause; fainting or
unconsciousness.
The effects of a stroke depend on which part of the brain is injured and how severely it
is affected. A very severe stroke can cause sudden death.
After choosing this case, we researched about CVA to know the cause, clinical
manifestation and nursing intervention that we are going to perform. After doing procedures
such as physical assessment and assessing the vital signs, along with our research, this
helped us to gain more knowledge and understanding about the disease.
H. RECOMMENDATION
As a student nurse we recommended the following:
To the patient:
1. Continue his medication with the help of the relatives
2. Eat healthy and nutritious foods avoid cholesterol that trigger his condition
3. Initiate physical therapy after discharged in the hospital
4. Teach the relatives how to monitors patients vital signs, frequently specially blood
pressure because patient is unstable in her condition.
5. The relatives should always pray for the fast recovery of their love ones.
6. Be patient in caring for his, who suffering from this kind of disease.
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