Five Level of Prevention

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    LEVELS OF PREVENTION

    (LEAVELL & CLARK)

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    THE NATURAL HISTORY OF ANY DISEASE OF MANInterrelation of Agent, Host, and Environmental

    factors

    Production of STIMULUS

    Reaction of the HOST to the STIMULUS

    Early

    pathogenesis

    Discernible

    earlyl esions

    Advanced

    disease Convalescence

    LEVELS OF APPLICATION of PREVENTIVE MEASURES

    HEALTH PROMOTION

    Health education

    Good standard

    of nutrition

    Provision of adequate

    Housing, recreation,

    and agreeable

    work conditions

    Marriage counseling

    and sex education

    Genetics

    SPECIFIC PROTECTION

    Use of specific

    Immunizations

    Attention to

    personal hygiene

    Use of environmental

    sanitation

    Protection against

    occupational hazards

    Protection from accidents

    Use of specific nutrients

    Protection from carsinogens

    Avoidance of allergens

    EARLY DIAGNOSIS and

    PROMPT TREATMENT

    Case-finding measures,

    individual and mass

    Screening survey

    Selective examinations

    Objectives :

    To cure and prevent

    disease processes

    To prevent the spread ofcommunicable diseases

    To prevent complications

    and sequele

    To shorten period of disability

    DISABILITY LIMITATION

    Adequate treatment

    to arrest

    the disease process

    and to prevent

    further complication

    and sequelae

    Provision of

    facilities to limit

    disability

    and to prevent death

    REHABILITATION

    Provision of hospital

    and community facilities

    for retraining and

    education for

    maximum use

    of remaining capacities

    Education of the public

    and industry to utilized

    the rehabilitated

    as full employment

    Selective placement

    Work therapy in hospitals

    Use of sheltered colony

    PRIMARY PREVENTION SECONDARY PREVENTION TERTIARY PREVENTION

    PREPATHOGENESIS PERIOD PERIOD OF PATHOGENESIS

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    THENATURAL HISTORY OF OMSK

    Interrelations of the various

    AGENT: PYOGENIC BACTERIA

    HOST: HUMAN

    (IMMUNOLOGIC, AGE,

    NUTRITION

    ENVIRONMENT:

    (HYGIENE,SANIATION

    Bring AGENT and

    HOST together

    STIMULUS or

    AGENT becomes

    Or Produce a

    disease-provoking

    STIMULUS

    PREPATHOGENESISPERIOD

    The course of the disease in man

    CLINICAL HORIZON

    P E R I O D O F P A T H O G E N E S I S

    Early

    pathogenesis

    Discernible

    early lesions

    Advanced

    diseaseConvalescence

    STIMULUS and AGENT

    BECOMES established and

    increases by multiplication or

    increment

    Signs and

    symptom :

    URTI / OMA

    illness

    Disability

    Defect

    Chronic

    state

    DEATH

    Interactions of HOST

    and SIMULUS

    Tissue and

    physiologic

    changesImmunity and

    resistance

    HOST REACTION

    RECOVERY

    in

    hu

    man

    HOST

    ss

    Eustachian tube

    resistance

    URTI

    OMSK

    DEAFNESS

    MASTOIDITIS

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    THE NATURAL HISTORY OF OMSKInterrelation of Agent, Host, and Environmental

    factors

    Production of STIMULUS

    Reaction of the HOST to the STIMULUS

    Early

    pathogenesis

    URTI/OMA OMSK/MAS

    TOIDITIS Convalescence

    LEVELS OF APPLICATION of PREVENTIVE MEASURES

    HEALTH PROMOTION

    Health education

    Good standard

    of nutrition

    Provision of adequate

    Housing

    Hygiene, sanitation

    SPECIFIC PROTECTION

    Avoid recurrent URTI

    EARLY DIAGNOSIS and

    PROMPT TREATMENT

    Early/ adequate

    treatment of URTI

    Adequate Treatment of

    OMA

    Objectives :

    To cure and prevent

    disease processes

    To prevent complications

    and sequele

    To shorten period of disability

    DISABILITY LIMITATION

    Adequate treatment

    to arrest

    the disease process

    and to prevent

    further complication

    and sequelae

    Provision of

    facilities to limit

    disability

    and to prevent death

    REHABILITATION

    Provision of hospital

    and community facilities

    for retraining and

    education for

    maximum use

    of remaining capacities

    Education of the public

    and industry to utilized

    the rehabilitated

    as full employment

    Selective placement

    Work therapy in hospitals

    Use of sheltered colony

    PRIMARY PREVENTION SECONDARY PREVENTION TERTIARY PREVENTION

    PREPATHOGENESIS PERIOD PERIOD OF PATHOGENESIS

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    Levels of prevention

    (diabetic retinopathy)

    Primary prevention:

    Health promotion

    Balance diet, exercise (healthy life)

    Specific protection:

    For high risk : low GI diet, exercise

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    SecondaryPrevention

    Early diagnosis & prompt treatment:

    - DM screening

    - Adequate and continuing treatment- Avoid complication:

    (microvascular, macrovascular) throughmaintenance normal blood sugar

    to avoid hyperglycemia or hypoglycemia

    DM diet

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    Tertiary prevention

    - Disability limitation

    Prevent from blindness caused bydiabetic retinopathy through DM control

    - Rehabilitation: tosave and rehabilitatedvision as much as possible.

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    Health promotion

    Stroke

    Heart/CV

    disease

    Smoking

    Communityenvironment

    Quit

    Clinical

    care

    Time

    Tobaccocontrol

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