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8/11/2019 Decompression Sickness - Dr Guritno
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Decompression sickness (DCS) refers to the clinicalsyndrome of neurological deficits, pain, or other clinical
disorders resulting from the body tissues being
supersaturated with inert gas after a reduction in the
ambient pressure.
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Arterial Gas Embolism (AGE) refers to gas bubbles inthe systemic arterial system resulting from pulmonarybarotrauma, iatrogenic entry of gas into the arterialsystem, or arterialized venous gas emboli.
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Classification of DI
1. The traditional or
Golding Classification2. The descriptive or
Francis & Smith Classification
3. The ICD-10 Classification
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Modified Golding Classification for DI
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Table The Francis & Smith Classificationfor Dysbaric Illness
Evolutiono Spontaneously Recovery (Clinical improvement is
evident)
o Static (No change in clinical condition)
o
Relapsing (Relapsing symptoms after initial recovery)
Progressive (Increasing number or severityof signs)
Organ System:
o Neurological
o Cardiopulmonary
o Limb pain exclusively
o Skin
o Lymphatic
o
Vestibular
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Table The Francis & Smith Classificationfor Dysbaric Illness
Time of onset:o
Time before surfacingo
Time after surfacing (or estimate)
Gas Burdeno Low (e.g., within NDL)
o Medium (e.g., Decompression Dive)o
High (e.g., Violation of Dive Table)
Evidence of Barotraumao Pulmonary (Yes / No)
o Ears
o
Sinuses
Other Comments
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The ICD-10 Classification
The ICD-10 codes most frequently used are:o T70 (Effects of air pressure and water
pressure)
o T70.0 (Otitic barotrauma)
o
T70.1 (Sinus barotrauma)o T70.3 (Caissons disease)o T70.4 (Effects of high-pressure fluids)
o T70.8 (Other effects of air pressure and water
pressure)o T79.0 (Traumatic air embolism)
o T79.7 (Traumatic subcutaneous emphysema)o M90.3 (Osteonecrosis in caisson disease
T70.3+)
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Clinical Setting
1. Diving2. Flying
3. HBOT
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PATHOGENESIS OF DCS
Denaturation of Plasma Proteins
Endothelial Damage
Interaction of Bubbles with the Blood
Coagulation System
General Aspect :Most of the clinical manifestations of DCS arethought to result from tissue distortion of vascularobstruction produced by bubbles
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Presenting Symptoms Type Cases (%)
Local Pain
ArmLeg
Vertigo (staggers)
ParalysisShortness of breath (chokes)
Extreme fatigue with pain
Collapse + unconsciousness
Type I
Type 2
89
3070
5.3
2.31.6
1.3
0.5
Frequency of Various Symptoms of DCS
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The time of onset ofsymptoms after surfacing
30 % occurred < 30 minutes
85 % occurred < 1 hour
95 % occurred < 3 hours
1 % Delayed more than 6hours
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Predisposing Factors
Exercise
Injury
Cold
Obesity
Increased FractionalConcentration of CO2 toinspired Gas
Age
Ingestion of Alcohol
Dehydration
Fatigue
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Treatment of DCS
Type I Treatment Table 5 (TT5) Musculoskeletal pain
Skin bends
Lymphatic bends
Type II Treatment Table 6 (TT6) Includes all other manifestations of DCS
Recompress to 60 FSW on 100% O2 and begin TT6
Diving Medical Officer (DMO) has option to go to 165 early if
patient has unsatisfactory response at 60 FSW
*Note: Severe Type II signs/symptoms warrant full extensions of 60 FSWoxygen breathing periods even if S/S resolve during the first oxygenbreathing period
Deep Uncontrolled Ascents Treatment Table 8 (TT8) 225 FSWtable for treating deep, uncontrolled ascents when more than 60minutes of decompression have been missed.
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Treatment of DCS
Persistent Symptoms at 60 FSW
Extend TT6 for two 25-minute periods at 60 FSW
Extend TT6 for two 75-minute periods at 30 FSW
DMO may recommend customized treatment
Stay at 60 FSW for 12 hours or longer come out on TT7
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If shallower than 60 FSW go to 60 FSW
If deeper than 60 FSW go to 165 FSW
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Treatment of DCS Persistent Symptoms at 60 FSW
Extend TT6 for two 25-minute periods at 60 FSW
Extend TT6 for two 75-minute periods at 30 FSW
DMO may recommend customized treatment
Stay at 60 FSW for 12 hours or longer come out on TT7
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If shallower than 60 FSW go to 60 FSW
If deeper than 60 FSW go to 165 FSW
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Treatment of DCS
In-Water RecompressionOnly when:
No recompression facility on site
Significant signs/symptoms
No prospect of reaching chamber in 12-24 hrs
No improvement after 30 min of 100% oxygen onsurface
Thermal conditions are favorable
Not for unconsciousness, paralysis, respiratorydistress, or shock
Keep these individuals on the surface with 100% O2
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Treatment of DCS
In-Water RecompressionOnly when:
In-Water Recompression with oxygen preferred
Purge rebreather 3 times with oxygen
30 FSW with stand-by diver
60 min at rest for Type 1
90 min at rest for Type II
20 FSW for 60 min
10 FSW for 60 min
100% O2 for additional 3 hours on the surface
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Treatment of DCS
In-Water Recompression with air (if no oxygenavailable) Follow TT1A
Full face mask or surface-supplied helmet preferred
SCUBA used only as last resort Stand-by diver required
* Note: In divers with severe Type II symptoms or symptoms ofarterial gas embolism (e.g. unconsciousness, paralysis, vertigo,respiratory distress (chokes), shock, etc), the risk of increasedharm to the diver from in-water recompression probablyoutweighs any anticipated benefit.
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Screening for Patent Foramen Ovale (PFO)
History of DCS Disqualifying for diving duty Deselection of divers for repeated episodes of DCS Not
recommended
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:1&O8.3& &1I.2&P Q4 recommendation
Table modifications based on water temp No recommendation
Hydration (in warm water diving)
Dive depth limits: For SCUBA dives maximum depth of 130 ft(on-site chamber recommended for military diving if dive depth isgreater than100 ft)
Clean times: Surface interval required for the diver to beconsidered clean for the next dive: 2 hours 20 minutes forrepetitive group Alpha 15 hours 50 minutes for repetitive groupZulu
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DCS PREVENTION
3. DCS Prevention (During the Dive)
Ascent Rate 30 feet per minute
4. DCS Prevention (Post-Dive)
Exercise restrictions Both aerobic (e.g. running) andanaerobic (e.g. weight lifting) exercise performed
within 4 hours after a compressed gas dive with
significant decompression stress may be associated
with an increased risk of DCS
Ascent to altitude restrictions (Up to 10,000 ft) Time/
ascent Table - up to 29:15 for Repet Group Zulu 48
hours for Exceptional Exposure Dives
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Manifestations of AGE
Loss of consciousness
Confusion Focal neurological deficits
Cardiac arrhythmias or ischemia
Cardiac arrest and death 4%
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Causes of AGE
Pulmonary barotrauma Iatrogenic events (radiologic
procedures and cardiac bypass
surgery) Right-to-left shunt
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Treatment of AGE
The primary goal of treatment is theprotection and maintenance of vitalfunctions
Pre-hospital 100% oxygen by rebreathing face mask
Supine position
Maintain hydration
HBO is the treatment of choice Adjunctive therapy: lidocaine,
anticoagulant, corticosteroid
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Benefits of HBOT
1.Compression of existing gas bubbles
2. Establishment of a high diffusion gradient tospeed dissolution of existing bubbles
3. Improved oxygenation of ischemic tissues
and lowered intracranial pressure
4. Reduction of ischemic-reperfusion injury
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Treatment table selection
Initial treatment USNTT6 extend Table 6 orUNSTT6A
Follow-up treatments
Daily or twice daily
Until complete relief of symptoms or until there isno further clinical improvement after 2 consecutivetreatments
Until complete relief of symptoms or until there isno further clinical improvement after 2 consecutivetreatments
No consensus: table 5, 6 and 9
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