Decompression Sickness - Dr Guritno

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    Decompression sickness (DCS) refers to the clinicalsyndrome of neurological deficits, pain, or other clinical

    disorders resulting from the body tissues being

    supersaturated with inert gas after a reduction in the

    ambient pressure.

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    Arterial Gas Embolism (AGE) refers to gas bubbles inthe systemic arterial system resulting from pulmonarybarotrauma, iatrogenic entry of gas into the arterialsystem, or arterialized venous gas emboli.

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    Classification of DI

    1. The traditional or

    Golding Classification2. The descriptive or

    Francis & Smith Classification

    3. The ICD-10 Classification

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    Modified Golding Classification for DI

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    Table The Francis & Smith Classificationfor Dysbaric Illness

    Evolutiono Spontaneously Recovery (Clinical improvement is

    evident)

    o Static (No change in clinical condition)

    o

    Relapsing (Relapsing symptoms after initial recovery)

    Progressive (Increasing number or severityof signs)

    Organ System:

    o Neurological

    o Cardiopulmonary

    o Limb pain exclusively

    o Skin

    o Lymphatic

    o

    Vestibular

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    Table The Francis & Smith Classificationfor Dysbaric Illness

    Time of onset:o

    Time before surfacingo

    Time after surfacing (or estimate)

    Gas Burdeno Low (e.g., within NDL)

    o Medium (e.g., Decompression Dive)o

    High (e.g., Violation of Dive Table)

    Evidence of Barotraumao Pulmonary (Yes / No)

    o Ears

    o

    Sinuses

    Other Comments

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    The ICD-10 Classification

    The ICD-10 codes most frequently used are:o T70 (Effects of air pressure and water

    pressure)

    o T70.0 (Otitic barotrauma)

    o

    T70.1 (Sinus barotrauma)o T70.3 (Caissons disease)o T70.4 (Effects of high-pressure fluids)

    o T70.8 (Other effects of air pressure and water

    pressure)o T79.0 (Traumatic air embolism)

    o T79.7 (Traumatic subcutaneous emphysema)o M90.3 (Osteonecrosis in caisson disease

    T70.3+)

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    Clinical Setting

    1. Diving2. Flying

    3. HBOT

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    PATHOGENESIS OF DCS

    Denaturation of Plasma Proteins

    Endothelial Damage

    Interaction of Bubbles with the Blood

    Coagulation System

    General Aspect :Most of the clinical manifestations of DCS arethought to result from tissue distortion of vascularobstruction produced by bubbles

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    Presenting Symptoms Type Cases (%)

    Local Pain

    ArmLeg

    Vertigo (staggers)

    ParalysisShortness of breath (chokes)

    Extreme fatigue with pain

    Collapse + unconsciousness

    Type I

    Type 2

    89

    3070

    5.3

    2.31.6

    1.3

    0.5

    Frequency of Various Symptoms of DCS

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    The time of onset ofsymptoms after surfacing

    30 % occurred < 30 minutes

    85 % occurred < 1 hour

    95 % occurred < 3 hours

    1 % Delayed more than 6hours

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    Predisposing Factors

    Exercise

    Injury

    Cold

    Obesity

    Increased FractionalConcentration of CO2 toinspired Gas

    Age

    Ingestion of Alcohol

    Dehydration

    Fatigue

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    Treatment of DCS

    Type I Treatment Table 5 (TT5) Musculoskeletal pain

    Skin bends

    Lymphatic bends

    Type II Treatment Table 6 (TT6) Includes all other manifestations of DCS

    Recompress to 60 FSW on 100% O2 and begin TT6

    Diving Medical Officer (DMO) has option to go to 165 early if

    patient has unsatisfactory response at 60 FSW

    *Note: Severe Type II signs/symptoms warrant full extensions of 60 FSWoxygen breathing periods even if S/S resolve during the first oxygenbreathing period

    Deep Uncontrolled Ascents Treatment Table 8 (TT8) 225 FSWtable for treating deep, uncontrolled ascents when more than 60minutes of decompression have been missed.

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    Treatment of DCS

    Persistent Symptoms at 60 FSW

    Extend TT6 for two 25-minute periods at 60 FSW

    Extend TT6 for two 75-minute periods at 30 FSW

    DMO may recommend customized treatment

    Stay at 60 FSW for 12 hours or longer come out on TT7

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    If shallower than 60 FSW go to 60 FSW

    If deeper than 60 FSW go to 165 FSW

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    Treatment of DCS Persistent Symptoms at 60 FSW

    Extend TT6 for two 25-minute periods at 60 FSW

    Extend TT6 for two 75-minute periods at 30 FSW

    DMO may recommend customized treatment

    Stay at 60 FSW for 12 hours or longer come out on TT7

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    If shallower than 60 FSW go to 60 FSW

    If deeper than 60 FSW go to 165 FSW

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    Treatment of DCS

    In-Water RecompressionOnly when:

    No recompression facility on site

    Significant signs/symptoms

    No prospect of reaching chamber in 12-24 hrs

    No improvement after 30 min of 100% oxygen onsurface

    Thermal conditions are favorable

    Not for unconsciousness, paralysis, respiratorydistress, or shock

    Keep these individuals on the surface with 100% O2

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    Treatment of DCS

    In-Water RecompressionOnly when:

    In-Water Recompression with oxygen preferred

    Purge rebreather 3 times with oxygen

    30 FSW with stand-by diver

    60 min at rest for Type 1

    90 min at rest for Type II

    20 FSW for 60 min

    10 FSW for 60 min

    100% O2 for additional 3 hours on the surface

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    Treatment of DCS

    In-Water Recompression with air (if no oxygenavailable) Follow TT1A

    Full face mask or surface-supplied helmet preferred

    SCUBA used only as last resort Stand-by diver required

    * Note: In divers with severe Type II symptoms or symptoms ofarterial gas embolism (e.g. unconsciousness, paralysis, vertigo,respiratory distress (chokes), shock, etc), the risk of increasedharm to the diver from in-water recompression probablyoutweighs any anticipated benefit.

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    Screening for Patent Foramen Ovale (PFO)

    History of DCS Disqualifying for diving duty Deselection of divers for repeated episodes of DCS Not

    recommended

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    Table modifications based on water temp No recommendation

    Hydration (in warm water diving)

    Dive depth limits: For SCUBA dives maximum depth of 130 ft(on-site chamber recommended for military diving if dive depth isgreater than100 ft)

    Clean times: Surface interval required for the diver to beconsidered clean for the next dive: 2 hours 20 minutes forrepetitive group Alpha 15 hours 50 minutes for repetitive groupZulu

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    DCS PREVENTION

    3. DCS Prevention (During the Dive)

    Ascent Rate 30 feet per minute

    4. DCS Prevention (Post-Dive)

    Exercise restrictions Both aerobic (e.g. running) andanaerobic (e.g. weight lifting) exercise performed

    within 4 hours after a compressed gas dive with

    significant decompression stress may be associated

    with an increased risk of DCS

    Ascent to altitude restrictions (Up to 10,000 ft) Time/

    ascent Table - up to 29:15 for Repet Group Zulu 48

    hours for Exceptional Exposure Dives

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    Manifestations of AGE

    Loss of consciousness

    Confusion Focal neurological deficits

    Cardiac arrhythmias or ischemia

    Cardiac arrest and death 4%

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    Causes of AGE

    Pulmonary barotrauma Iatrogenic events (radiologic

    procedures and cardiac bypass

    surgery) Right-to-left shunt

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    Treatment of AGE

    The primary goal of treatment is theprotection and maintenance of vitalfunctions

    Pre-hospital 100% oxygen by rebreathing face mask

    Supine position

    Maintain hydration

    HBO is the treatment of choice Adjunctive therapy: lidocaine,

    anticoagulant, corticosteroid

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    Benefits of HBOT

    1.Compression of existing gas bubbles

    2. Establishment of a high diffusion gradient tospeed dissolution of existing bubbles

    3. Improved oxygenation of ischemic tissues

    and lowered intracranial pressure

    4. Reduction of ischemic-reperfusion injury

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    Treatment table selection

    Initial treatment USNTT6 extend Table 6 orUNSTT6A

    Follow-up treatments

    Daily or twice daily

    Until complete relief of symptoms or until there isno further clinical improvement after 2 consecutivetreatments

    Until complete relief of symptoms or until there isno further clinical improvement after 2 consecutivetreatments

    No consensus: table 5, 6 and 9

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