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8/3/2019 Child With Respiratory Disorder
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PREPARED BY: MARLON JAPITANA, RN
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Key TERMS atelectasis
atropy
clubbing
coryza
cyanosis
expiration
hypoxemia
hypoxia
infiltrate
inspiration
laryngitis
oxygenation
pharyngitis
Pulmonary
pulse oximetry
rales
retractions
rhinitis
rhinorrhea
stridor
subglottic stenosis
suctioning
tachypnea
tracheostomy
ventilation
wheeze
work of breathing
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Physical ExaminationPhysical examination of the respiratory systemIncludes: inspection and observation, auscultation,
percussion, and palpation.
Inspection and Observation Color. Observe the childs color, noting pallor
or cyanosis(circumoral or central). Pallor (pale appearance) occurs as a result of
peripheral vasoconstriction in an effort toconserve oxygen for vital functions.
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Cyanosis (a bluish tinge to the skin) occurs as aresult ofhypoxia.
It might first present circumorally (justaround the mouth) and progress to centralcyanosis.
Newborns might have blue hands and feet(acrocyanosis), a normal finding.
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Grunting occurs on expiration and isproduced by premature glottic closure. It isan attempt to preserve or increase functionalresidual capacity.
Grunting might occur with alveolar collapseor loss of lung volume, such as in atelectasis,pneumonia, and pulmonary edema.
Stridor, a high-pitched, readily audibleinspiratory noise, is a sign of upper airwayobstruction.
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Retractions (the inward pulling of soft tissues withrespiration) can occur in the intercostal, subcostal,substernal, supraclavicular, or suprasternalregions.
Document the severity of the retractions: mild,moderate, or severe. Seesaw (or paradoxical) respirations are veryineffective for ventilation and oxygenation. Thechest falls on inspiration and rises on expiration.
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Suprasternal notch(Suprasternal retractions)
Xiphoid area(Suprasternal retractions)
Ribs(Intercostalretractions)
Location of retractions
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Synchronizedrespirations
Lag on respirations Seesaw respirations
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Clubbing. Inspect the fingertips for the presence ofclubbing, an enlargement of the terminal phalanx
of the finger, resulting in a change in the angle ofthe nail to theFingertip.
A. Normal B .Early clubbing C. Advanced clubbing
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The common cold is also referred to as a viralupper respiratory infection (URI) ornasopharyngitis.
Colds can be caused by a number of differentviruses, including rhinoviruses, parainfluenza, RSV,enteroviruses, and adenoviruses.S/Sx:
stuffy or runny nose.( Nasal discharge is usuallythin and watery at first but may become thickerand discolored.)
hoarse and complain of a sore throat. Cough usually produces very little sputum. Fever, fatigue, watery eyes, and appetite loss
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DIAGNOSIS: Laboratory (CBC), Chest X-ray
MGT.
Nasal congestion may be relieved with theuse of normal saline nose drops, followed bybulb syringe suctioning in infants andtoddlers.
A cool mist humidifier also helps with nasalcongestion.
Promotion of adequate oral fluid intake isimportant to liquefy secretions.
Antibiotics are not indicated unless the childalso has a bacterial infection.
Rest and immuno booster for viral causes.
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Counsel parents about symptoms of complicationsOf the common cold. These include: Prolonged fever
Increased throat pain or enlarged, painful lymph
nodes Increased or worsening cough, cough lasting
longer than 10 days, chest pain, difficultybreathing
Earache, headache, tooth or sinus pain
Unusual irritability or lethargy
Skin rash
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Sinusitis (also called rhinosinusitis) generallyrefers to a bacterial infection of the paranasalsinuses. The disease may be either acute orchronic in nature, with the treatment
approach varying with chronicity.
Symptoms lasting less than 30 days generallyindicate acute sinusitis, whereas symptomspersisting longer than 4 to 6 weeks usuallyindicate chronic sinusitis.
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The most common presentation of sinusitis ispersistent signs and symptoms of a cold. Ratherthan improving after 7 to 10 days, nasal
discharge persists. Explore the history for: Cough
Fever
In preschoolers or older children, halitosis (bad
breath) Facial pain may or may not be present, so is not
a reliable indicator of disease.
Eyelid edema (in the case ofethmoid sinusinvolvement)
Irritability
Poor appetite
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Mgt.
Normal saline nose drops or spray, cool misthumidifiers, and adequate oral fluid intake.
full course of antibiotics to eradicate thecause of infection.
decongestants, antihistamines, and intranasalsteroids as adjuncts in the treatment of
Sinusitis.
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Influenza viral infection occurs primarily duringthe winter.
The flu is spread through inhalation of dropletsor contact with fine-particle aerosols. Infected
children shed the virus for 1 to 2 days beforesymptoms begin. Influenza viruses primarily affect the upper
respiratory epithelium but can cause systemiceffects as well.
Influenza viruses primarily affect the upperrespiratory epithelium but can cause systemiceffects as well.
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Less common complications includeReyessyndrome and acute myositis. Reyes syndrome is an acute encephalopathy
that has been associated with aspirin use inthe influenza-infected child.
Acute myositis s particular to children. Asudden onset of severe pain and tendernessin both calves causes the child to refuse to
walk.
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Abrupt onset of fever (39.5 C), Facial flushing,
chills,
headache, myalgia, and malaise are accompanied by
cough and coryza. dry or sore throat,
Ocular symptoms such as photophobia,tearing, burning, and eye pain are common.
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Nursing management of influenza is mainly
supportive.
Symptomatic treatment of cough and feverand maintenance of hydration are the focus
of care. Amantadine hydrochloride (Symmetrel) and
other newer antiviral drugs can be effective inreducing symptoms associated with influenza
if started within the first 24 to 48 hoursof theillness.
vaccination
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Infectious mononucleosis is a self-limitedillness caused by the Epstein-Barr virus.
It is characterized by fever, malaise, sorethroat, Petechiae may be present on the
palate and lymphadenopathy. Mononucleosis is commonly called thekissing disease since it is transmitted by
oropharyngeal secretions.
Complications nclude splenic rupture,Guillain-Barr syndrome, and asepticmeningitis.
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analgesics and salt-water gargles arerecommended.
Bed rest should be encouraged while the childis febrile.
Concomitant strep throat in the presence of
infectious mononucleosis should be treated
with an antibiotic other than ampicillin.
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Croup is also referred to as Laryngotracheobronchitis because inflammation and
edema of the larynx, trachea, and bronchi occur
as a result of viral infection.
Children between 3 months and 3 years of age arethe most frequently affected with croup.
Parainfluenza is responsible for the majority ofcases of croup.
Other causes include adenovirus, influenza virusA and B, RSV, and rarely measles virus or
Mycoplasma pneumoniae.
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inflammation and edema obstruct the airway, Narrowing of the subglottic area of the
trachea results in audible inspiratory stridor.
Edema of the larynx causes hoarseness. Inflammation in the larynx and trachea
causes the characteristic barking cough Symptoms occur most often at night, and
croup is usually self-limited, lasting onlyabout 3 to 5 days.
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Humidified air (via a cool mist humidifier orsteamy bathroom).
Administer dexamethasone if ordered orepinephrine.
Home Care of Croup Keep the child quiet and discourage crying.
Allow the child to sit up (in your arms).
Encourage rest and fluid intake.
Ifstridor occurs, take the child into a steamybathroom
for 10 minutes.
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Administer medication (corticosteroid) asdirected.
Watch the child closely. Call the physician if: The child breathes faster, has retractions, or
has any other difficulty breathing.
The nostrils flare or the lips or nails have abluish tint.
The cough or stridor does not improve withexposure to moist air.
Restlessness increases or the child isconfused
The child begins to drool or cannot swallow.
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Antecedent viral URI Fever
Cough (note type and whether productive ornot)
Increased respiratory rate
History of lethargy, poor feeding, vomiting,or diarrhea in infants
Chills, headache, dyspnea, chest pain,abdominal pain, and nausea or vomiting inolder children
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Ensure adequate hydration and assist in thinning
of secretions. In children with increased work of breathing,
intravenous fluids may be necessary to maintainhydration. assume a position of comfort, usually with the
head of the bed elevated to promote aeration ofthe lungs.
If pain due to coughing or pneumonia itself is
severe, administer analgesics as prescribed. Provide supplemental oxygen to the child with
respiratory distress or hypoxia as needed.
Antibiotic therapy
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Tuberculosis is a highly contagious diseasecaused by inhalation of droplets ofMycobacterium tuberculosis orMycobacterium bovis.
The inhaled tubercle bacilli multiply in thealveoli and alveolar ducts, forming aninflammatory exudate.
After exposure to an infected individual, theincubation period is 2 to 10 weeks.
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Though pulmonary tuberculosis is the most
common, children may also have infection
in other parts of the body, such as theGastrointestinal tract or central nervous system.
S/Sx: Dullness to percussion might be present, as well
as diminished breath sounds and crackles.
Fever persists and pallor, anemia, weakness, andweight loss are present.
Diagnosis is confirmed with a positive Mantouxtest, positive gastric washings for acid-fastbacillus, and/or
a chest x-ray consistent with tuberculosis.
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Mgt: In the case of drug-sensitive tuberculosis, the
American Academy of Pediatrics recommends a6-month course of oral therapy. The first two months consist ofisoniazid,rifampin, and pyrazinamide given daily. This is followed by twice-weekly isoniazid andrifampin; administration must be observed
directly (usually by a public health nurse).
In the case of multidrug-resistant tuberculosis:
ethambutol or streptomycin is given viaIntramuscular injection.
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Respiratory distress syndrome (RDS) is arespiratory disorder that is specific toneonates.
It results from lung immaturity and a
deficiency in surfactant, so it is seen mostoften in premature infant.
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The onset of RDS is usually within several hoursof birth.
The newborn exhibits signs of respiratorydistress, including tachypnea, retractions, nasalflaring, grunting, and varying degrees ofcyanosis.
Auscultation reveals fine rales and diminishedbreath sounds.
If untreated, RDS progresses to seesawrespirations, respiratory failure, and shock.
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surfactant administration. Suctioning as indicated.
Maintenance of normothermia,
prevention of infection, maintenance of fluid and electrolyte balance,
and promotion of adequate nutrition(parenterally or via gavage feeding).
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Asthma is a chronic inflammatory airwaydisorder characterized by airway hyperresponsiveness, airway edema, and mucusproduction.
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Common signs and symptoms reported duringthe
health history might include:
Cough, particularly at night: hacking type ofcough that is initially nonproductive,becoming productive offrothy sputum
Difficulty breathing: shortness of breath,
chest tightness or pain, dyspnea withexercise
Wheezing
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Normal airway Airway with inflammation Airway withinflammation,
bronchospasm,and mucusproduction
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Pulse oximetry: oxygen saturation may besignificantly decreased or normal during amild exacerbation.
Chest x-ray: usually reveals hyperinflation. Blood gases: might show carbon dioxide
retention and hypoxemia. Peak expiratory flow rate (PEFR): is decreased
during an exacerbation.
Allergy testing: skin test or RAST candetermine allergic triggers for the asthmaticchild.
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Administration of Brochodilators:Nebulization and Puff inhalation
Corticosteriods: Montelocast
Oxygen administration
Allergens identification
Administration of Epinephrine
High Fowlers Position
Avoid warm environment. Adequate rest.
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Cystic fibrosis is an autosomal recessivedisorder that caused by a deletion occurringon the long arm of chromosome 7 at thecystic fibrosis transmembrane regulator
(CFTR) is the responsible gene mutation. occurs about once in every 3,300 live white
births and about once in every 16,000 liveblack births (Boat, 2004).
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A salty taste to the childs skin (resulting fromexcess chloride loss via perspiration) Meconium ileus or late, difficult passage of
meconium stool in the newborn period Abdominal pain or difficulty passing stool
(infants or toddlers might present with intestinalobstruction or intussusception at the time ofdiagnosis).
Bulky, greasy stools Poor weight gain and growth despite good
appetite. Chronic or recurrent cough and/or upper or
lower respiratory infections.
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Defect intheCFTR GeneAffects Pathophysiology Clinical Manifestation
Gastrointes
tinaltract
Decreased chloride and
water secretioninto the intestine (causingdehydration ofthe intestinal material) andinto the bileducts (causing increased
bile viscosity) Reduced pancreaticbicarbonatesecretion Hypersecretion of gastricacid
Insufficiency of pancreaticenzymesnecessary for digestion andabsorption Pancreas secretes thickmucus.
Meconium ileus Retention of fecal matter in distalintestine,resulting in vomiting, abdominaldistentionand cramping, anorexia, right lowerquadrant pain Sludging of intestinal contentsmay lead tofecal impaction, rectal prolapse,bowelobstruction, intussusception. Obstructive cirrhosis withesophagealvarices, and splenomegaly Gastroesophageal reflux disease Altered absorption of iron andvitamins A,D, E, and K Failure to thrive Hyperglycemia and developmentofdiabetes later in life
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hemoptysis, pneumothorax,
bacterial colonization, cor pulmonale, volvulus, intussusception,
intestinal obstruction, rectal prolapse, gastroesophageal reflux disease, diabetes, portal hypertension,
liver failure, gallstones, and decreased fertility.
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Maintain patent airways Prevent infection
Antibiotic therapy
Oxygenation
Chest physiotherapy.
Prevent possible occurrence of statedcomplications.
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Acid- BaseCondition
Cause Findings Mgt.RespiratoryAlkalosis
Hyperventilation Rapid, deepbreathingConfusion,unconsciousnessElevated plasmapH (7.45)bicarbonate=initially normalDecreased PCO2(below 40 mmhg)
Give brown bagfor breathing.Adequate restand relaxationtechnique.
RespiratoryAcidosis
Hypoventilationtrapping carbon
dioxide in alveoli
Shallow breathingInability to expire
freelyDecreased plasmapH (7.35)Bicarbonate=initially normalElevated PCO2
(over 40 mmhg)
Oxygenationtherapy.SodiumBicarbonatetherapy.
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