21
Changes in Cardiac Arrest Management

Changes in Cardiac Arrest Management. Pathophysiology of V- Fib Arrest

Embed Size (px)

Citation preview

Page 1: Changes in Cardiac Arrest Management. Pathophysiology of V- Fib Arrest

Changes in Cardiac Arrest Management

Page 2: Changes in Cardiac Arrest Management. Pathophysiology of V- Fib Arrest

Pathophysiology of V-Fib Arrest

Page 3: Changes in Cardiac Arrest Management. Pathophysiology of V- Fib Arrest
Page 4: Changes in Cardiac Arrest Management. Pathophysiology of V- Fib Arrest
Page 5: Changes in Cardiac Arrest Management. Pathophysiology of V- Fib Arrest
Page 6: Changes in Cardiac Arrest Management. Pathophysiology of V- Fib Arrest
Page 7: Changes in Cardiac Arrest Management. Pathophysiology of V- Fib Arrest
Page 8: Changes in Cardiac Arrest Management. Pathophysiology of V- Fib Arrest
Page 9: Changes in Cardiac Arrest Management. Pathophysiology of V- Fib Arrest
Page 10: Changes in Cardiac Arrest Management. Pathophysiology of V- Fib Arrest
Page 11: Changes in Cardiac Arrest Management. Pathophysiology of V- Fib Arrest

Defibrillation

No more stacked shocksTakes too longAll shocks maximum energy.EMS probably should not use AED’sBiphasic increases efficacy

Page 12: Changes in Cardiac Arrest Management. Pathophysiology of V- Fib Arrest

Defibrillation

• Primary treatment for V-fib at 3 minutes and under

• Should be delayed until good CPR for 2 minutes if down time over 3 minutes

• Biphasic should be used• AED’s good in 3 minutes, bad after• One shock only with no pulse checks

after

Page 13: Changes in Cardiac Arrest Management. Pathophysiology of V- Fib Arrest

Pulse Checks

Deadly!!Only check pulses when rhythm appears to have converted thru CPR on ECG or signs of lifeECC says check before shock delivered after 5 cycles of 30:2 CPR

Page 14: Changes in Cardiac Arrest Management. Pathophysiology of V- Fib Arrest

Vascular Access

• Avoid ET drugs whenever possible• Peripheral IV’s OK• Central IV’s slightly better, but

compression interruption frequent with placement

• Interosseous recommended when peripheral IV’s not obtainable

Page 15: Changes in Cardiac Arrest Management. Pathophysiology of V- Fib Arrest

Pharmacology

• No improvements evident based on science with drugs to improve outcome

• Epinephrine every 5 minutes• No added benefit to Vasopressin• Amiodarone and Lidocaine equal

effectiveness

Page 16: Changes in Cardiac Arrest Management. Pathophysiology of V- Fib Arrest

What about intubation?

In first 6 minutes, not a priority (V-fib) ASAP in PEA and Asystole.Understand that positive pressure breaths decrease cardiac output.Some air exchange from CPR plus gasping.Once intubated, 1 second breaths,six per minute. NO MORE.

Page 17: Changes in Cardiac Arrest Management. Pathophysiology of V- Fib Arrest

AirwayCombitube or ET equivalentRSA Mentality-view and see cords place ET, otherwise

immediate Combitube first try.

Page 18: Changes in Cardiac Arrest Management. Pathophysiology of V- Fib Arrest

Recommendations

Bystander CRR program911 CRR phone instructionDefib in first 2-3 minutes

CRR before shocks otherwise

Page 19: Changes in Cardiac Arrest Management. Pathophysiology of V- Fib Arrest

Recommendations

• AED’s in community, not on ambulance

• 200 uninterrupted compression• No airway first 3 rounds of CRR• Immediate vascular access- IO if

needed• Epinephrine 1mg as soon as possible

Page 20: Changes in Cardiac Arrest Management. Pathophysiology of V- Fib Arrest

Recommendations

• When airway is placed, use non-visualized airway or RSA technique if intubating

• No pause to put in airway• Never a pause after defib to check

pulse or rhythm.

Page 21: Changes in Cardiac Arrest Management. Pathophysiology of V- Fib Arrest

Testimony and Example

• A great example