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7/29/2019 Bahin Case Study
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Nursing Management of Patient with Pregnancy Induced
Hypertension
a 24 year old Army servant named CAA Christopher Bahin,
was diagnosed of Left Pleural Effusion, probably secondary to
pulmonary tuberculosis and an anterior chest wall mass on his
admission. An anticipated pain on his chest was present because
of
the wound from thoracentesis.
Prior to his admission, he experienced difficulty of breathing,
shortness of breath, easy fatigability, weight loss, and a non-
productive cough. Due to the persistence of symptoms, he
consulted
a station hospital where he was managed as a case of Pleural
Effusion secondary to Pulmonary Tuberculosis. He underwent five
times of thoracentesis and drained a 1200-1500cc occasion of
serosanguinous pleural fluid.
Pathophysiology
A tuberculous pleural effusion that occurs in the absence of
radiologically apparent TB may be the sequel to a primary infection
or it may represent reactivation TB. The pathogenesis of a
tuberculous pleural effusion is thought to be related to the rupture
of a subpleural caseous focus in the lung into the pleural space.
The basis for this is the observation by Stead et al. that they could
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demonstrate a caseous tuberculous focus in the lung contiguous
to
the diseased pleura in 12 of 15 patients with tuberculous pleuritis.
The three other patients in this study had parenchymal disease
although they did not have caseous foci adjacent to the pleura. It is
believed that delayed hypersensitivity plays a large role in the
pathogenesis of tuberculous pleural effusion. The hypersensitivity
reaction is initiated when tuberculous protein gains access to the
pleural space. Evidence for the role of hypersensitivity includes the
following: When tuberculous protein is injected into the pleural
spaces of guinea pigs sensitized to purified protein derivative, an
exudative pleural effusion rapidly develops. When the sensitized
guinea pigs are given antilymphocyte serum, the development of
the
pleural effusion is suppressed.The mycobacterial cultures of the
pleural fluid from most patients with tuberculous pleural effusions
are negative. The tuberculous pleural effusion develops when the
delayed hypersensitivity reaction increases the permeability of the
pleural capillaries to protein and then the increased protein levels
in the pleural fluid result in a much higher rate of pleural fluid
formation. In addition, the lymphocytic pleuritis obstructs the
lymphatics in the parietal pleura, which leads to decreased pleural
fluid clearance from the pleural space. The pleural effusion results
from the combination of the increased pleural fluid formation and
the decreased pleural fluid removal.
History
P.E., a 24 year old Army servant named CAA Christopher Bahin,
was diagnosed of Left Pleural Effusion, probably secondary to
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pulmonary tuberculosis and an anterior chest wall mass on his
admission. An anticipated pain on his chest was present because
of
the wound from thoracentesis.
2 months prior to admission, patient started to experience
difficulty
of breathing described as shortness of breath and easy fatigability,
weight loss, anorexia and non productive cough. Persistence of
symptoms prompted consult case of pleural effusion secondary to
pulmonary tuberculosis. He was started on anti-Kochs medication
and also given IV antibiotics for pneumonia. He underwent five (5)
times thoracentesis and drained 1200-1500cc/ occasion of
serosanguinous pleural fluidPersistence prompted transfer in
AFPMC.
Nursing Physical Assessment
We have noted for the patients vital signs and all as such are as
follows;
Blood pressure: 100/70normal
Temperature: 36.2 deg. Celciusnormal
Pulse: 83 BPMnormal
Respiratory rate: 30 CPMabnormal
Related Treatments:
The doctor ordered the following for the patient:
Regular diet Multivitamins + EAA cap at 1cap OD
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1NH + RIF + EMB + PZA tab at 4tabs ODBB Celecoxib 200g/cap at 1cap BID PRN Paracetamol 500mg tab at 1tab Co-amoxiclav 1.2g TIV then 600mg IV q 8hrs. Tramadol 25mg TIV q 8hrs. PRN for pain
Nursing Care Plan:
*Short Term Goals
> to eliminate pain which the patient feels periodically from the
thoracentesis wound.
*Long Term Goals
> to prevent discomfort of the patient until he is able to function
normally.
Nursing Interventions:
>Observe changes in the clients cough, sputum, respiratory depth,
and breath sounds, and note complaints of chest pain.
>Position the client appropriately.
>Some agency protocols recommend that the client lie on the
unaffected side with the head of the bed elevated 30 degrees for at
least 30 minutes because this position facilitates expansion of the
affected lung and eases respirations.
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>Position the patient in a side-lying position with the unaffected
side
down for an hour or longer.
>Include date and time performed; the primary care providers
name; the amount, color, and clarity of fluid drained; and nursing
assessments and interventions provided.
>Transport the specimens to the laboratory.
>The dressing over the puncture site will be monitored for bleeding
or other drainage. Monitor patients blood pressure, pulse, and
breathing until are stable.
>Document all relevant information.y
>Administer Tramadol 25mg TIV q 8hrs. PRN for pain
Evaluation:
After an hour of proper nursing interventions, following the
managements listed above, the Sir Christopher Bahin was
responding well to the medication given for pain and to thepositioning implemented to for his own comfort. Vital signs were
normal, especially, the patients respiratory rate decreased near to
normal. Sir Bahin reports of comfort after doing proper nursing
interventions.
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Recommendations:
I highly recommend to the future researchers that a case like this
of
CAA Christopher Bahin should have proper nursing interventions
which focus on the promotion patients comfort especially that
which is concerned of the patients difficulty of breathing(shortness
of breath) and pain from the patients thoracentesis wound,
moreover, not putting the patients other concerns about the
disease
at stake.
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Manalo Maiko Dan T.
BSN 3A2-8/ Group 30
Dr. Ferriol
AFPMC