Adenotonsillitis New

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    Dr.Ravikumar MS(ENT)

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    Etiology : Viral followed bysecondarily invaded by

    Hemolytic streptococcus

    Staph aureas

    H.influenza Dipl. Pneumoniae

    Age : Commonest

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    Spread : Droplet infection

    Types:

    Ac cattarhal

    Ac parenchymatous

    Ac follicular

    Ac membranous

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    Viral Infections

    Bacterial Infections

    Inflammatory exudates of the crypts

    Epithelial keratinisation

    Deep-seated multiple abscess formation

    with increasing germ centers

    Parenchyma destruction

    Immunologic Factors

    Pathophysiology

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    When tonsilsare inflamed as

    part of thegeneralisedinfection of theoropharyngeal

    mucosa it iscalled catarrhaltonsillitis.

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    Some timesexudation fromcrypts may

    coalesce to forma membraneover the surfaceof tonsil, giving

    rise to clinicalpicture ofmembranoustonsillitis.

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    When thewhole tonsil is

    uniformlycongested andswollen it is

    called acuteparenchymatous tonsillitis

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    SYMPTOMS Sore throat Fever Malaise Odynophagia

    Thick speech Earache

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    Acute Tonsillitis - Signs

    Enlarged

    Erythematous

    Exudative forming at

    timespseudomembrane

    Enlarged neck nodes

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    Grading the Size of Tonsils

    Grading system:

    A. 0

    tonsils in fossaB. +1 tonsils less than 25%C. +2 tonsils less than 50%D. +3 tonsils less than 75%E. +4 tonsils greater than 75%

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    Rapid strep tests:latex agglutination or ELISAmethods extract antigen from swab

    Throat swab:

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    Bed rest : Isolation

    Mouth gurgles :

    Analgesics :

    Antibiotics :

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    First Line Penicillin/Cephalosporin for 10 days

    Injectable forms for noncompliance

    Macrolides Penicillin allergy Erythromycin/Clarithromycin 10 days

    Azithromycin (12mg/kg/day) 5 days

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    Peritonsillar abscess

    Parapharyngeal abscess

    Retropharyngeal abscess

    Oedema of larynx

    Cervical supp. Lymphadenitis

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    Acute middle ear cleft infection

    Chronic tonsillitis

    Rheumatic heart disease

    Chorea

    S.B.E.

    Acute nephritis

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    Infectious Mononucleosis Faucial diphtheria Agranulocytosis Scarlet fever Oral thrush ALL Vincents angina Tertiary syphilis

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    Differential

    Diagnosis ofpseudomembranous

    tonsillitis

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    Infectious Mononucleosis

    Cheesy exudatescovering tonsil

    Lymphadenopathy ofneck, axilla & groin

    Hepato/Spleenomegaly

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    Oral Thrush

    Painful throat White candidiasis

    patches whenremoved leaveserythematous ulcer

    Immunosuppressivestate

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    Keratosis tonsils

    Incidental finding

    May cause slightdiscomfort

    Yellow hornyoutgrowths in thecrypts

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    Agranulocytosis

    Halistosis, fever,headache &

    dysphagia Single , multiple or

    coalesce necroticslough covered

    ulcers Leucopenia H/O causative drugs

    intake

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    Vincents angina Fetor oris, pyrexia Tonsillar deep ulcers with

    grey slough in its base

    Necrotising gingivitis Enlarged tender cervical

    adenitis

    Smear:

    Spirochaetes & Fusiform

    bacilli

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    Acute lymphatic leukemia

    Fever, anaemia &bleeding disorders

    Slough coveredmembrane formingulcerations

    Cervical

    lymphadenopathy Exaggeratedleucocytosis

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    Diphtheria

    Malaise, fever &headache

    Greyish greenmembrane acrosstonsils to larynx

    Tender bilateralcervicallymphadenopathy

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    Diphtheria is an acute, toxin-mediated disease

    caused by toxigenic Corynebacterium diphtheriae

    Its a very contagious and potentially life-

    threatening bacterial disease.

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    Causative organism of diphtheria

    Gram- positive bacillus

    Produces exotoxin at site of infection

    Travels to heart and nervous system

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    Spread by close contact via droplets or

    contaminated articles

    Humans are the sole carriers of theorganism

    More common in children < 10 years

    Rare occurrence today because of routine

    vaccination

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    Clinical manifestations Systemic symptoms from exotoxin

    Fatigued

    Lethargic

    Tachycardic

    toxic

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    Clinical characteristics Pharynx grayish membrane (composed of

    fibrin, leukocytes, and cellular debris) extends from pharynx to larynx Extensive cervical lymphadenopathy

    (bull neck)

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    Diagnosis Isolation of the organism

    Culture from local lesion

    Grows on selective media containingpotassium tellurite

    Notify microbiology lab if diphtheriasuspected

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    Treatment Started before culture confirmation

    Airway

    Resuscitation

    Skin test for allergy to horse serum

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    Administer diphtheria antitoxin

    Have epinephrine available

    Antibiotics : penicillin G is the drug

    of choice Erythomycin

    Pt allergic to both drugs- rifampin,

    and clindamycin

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    Prevention Vaccine

    Trivalent vaccine diphtheria toxoid,tetanus toxoid and pertussis (DTP)

    6 weeks of age, 2 more 4-8 weeksintervals, and 4th 6-12 months later.

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    Myocarditis.

    Cardiac arrhythmias.

    Acute circulatory failure.

    Paralysis of soft palate,diaphragm & ocular

    muscles.

    Laryngeal-airway obstruction.

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    Ac tonsillitis Faucialdiphtheria

    Onset Acute Insidious

    Membrane Yellowish,easily

    separable

    Ashy gray,bleeds onseparation

    Fever High Low

    Pulse Proportionate Disproportionate

    Toxaemia +/- + +

    Throat swab Heam.strepto CBD / KLB

    Urine:

    Albuminuria

    +/- + +

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    Local Complications

    Respiratory obstruction Quinsy

    Acute retropharyngeal abscess Parapharyngeal abscess Neck space infectionsAcute otitis media

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    . Comp of acute tonsillitis ordenovo with no precedingtonsillitis

    . Collection pus b/w supr

    constrictor & fibrous capsule

    at upper pole

    .

    Peritonsillar Abscess or Quinsy

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    The bacteriology of acute tonsillitisand peritonsillar abscess is differentalthough one is a complication of the

    other.

    The bacteriology of the quinsy is

    characterized by mixed flora withmultiple organisms both aerobic andanaerobic.

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    repeated attacks of acute tonsillitis Fever

    severe throat pain referred otalgia swelling in the neck

    patients voice develops acharacteristicplummy quality

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    Ill looking patient Pyrexia Often with severe

    trismus

    Striking asymmetrywith oedema andhyperaemia of the softpalate.

    Enlarged hyperaemicand displaced tonsil Usually enlarged lymph

    nodes in JD region.

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    Preferably admitted to hospital and treatedwith analgesics and antibiotics.

    In a patient with an early peritonsillar abscess

    which is really a peritonsillar cellulitis incisionand drainage are not recommended.

    http://www.mdconsult.com/das/book/body/0/0/1492/f4-u1.0-B978-1-4160-2805-5..50460-2..gr4.jpg
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    Indications for I/Dinclude markedbulging of soft palate

    This is undertaken atthe point ofmaximum bulge.

    Interval tonsillectomy

    after 6 weeks. Abscess tonsillectomy.

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    Quinsy is a

    potentially lethalcondition

    Pharyngeal &Laryngealoedema

    Parapharyngealspace abscess

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    Recurrent Acute Tonsillitis

    Same signs andsymptoms asacute

    Occurring in 4-7separate episodesper year

    5 episodes peryear for 2 years 3 episodes per

    year for 3 years

    Ant pillar peritonsillar erythema

    Smooth glisteningtonsil with dilatedblood vessels on thesurface

    Debris in cryptswhich are few dueto loss of tonsilarchitecture

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    Chronic Tonsillitis

    Chronic sore throat

    Malodorous breath

    Presence of tonsilliths

    Peritonsillar erythema

    Persistent cervical lymphadenopathy Lasting at least 3 months

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    RECURRENT PAINHALITOSIS

    COUGH

    SNORING

    SLEEP APNOEA

    ASYMPTOMATIC

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    LARGE TONSILS

    SMALL FIBROTIC INADULTS

    ANTERIOR PILLARCONGESTED

    SQUEEZE IRWIN

    MOORES SIGN

    LYMPHADENOPATHY

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    Apparent enlargement vs true enlargement

    Non-neoplastic: Acute infective

    Chronic infective Hypertrophy Congenital

    Neoplastic

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    Pleomorphic Adenoma ICAAneurysm

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    Normally regress by 10 yrs

    Etiology:

    Age ; 3-4 years

    Physiological hypertrophy

    Infection

    Rarely tuberculosis

    Predisposing factors

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    Associated with nasal obstruction :

    Adenoid facies (develop gradually)

    Nose Pinched ,narrow Mouth - Remains open,dribbling of

    saliva,mouth breathing

    Teeth Protruded,irregular,crowded Lower jaw Undershot

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    Palate High arched.

    Feeding difficulties.

    Face - Loss of nasolabial furrow,dulllook.

    Chest Pigeon shaped.

    Pot belly.

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    Hyponasality

    Snoring Openmouthbreathing

    Triad

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    NASAL DISCHARGE.

    SINUSITIS.

    EPISTAXIS.

    VOICE CHANGE.

    THE DEVELOPMENT OF EXUDATIVE OTITIS

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    The closing of Eustachian tube

    The accumulation of exudate in

    the middle ear

    The lack of mobility of

    eardrum

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    Diagnosis :

    Clinical features: clinch diagnosis

    Posterior rhinoscopy:

    Digital palpation :--bag of worms

    X-ray nasopharynx soft tissue lateralview

    Nasal endoscopy , Nasopharyngoscopy

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    Lateral neck films areuseful only when

    history and physical

    exam are not in

    agreement.

    Accuracy of lateral neck

    films is dependent onproper positioning

    and patientcooperation.

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    Conservative : in acute & mild cases

    Antibiotics,Decongestants,Breathing exercises

    Surgical : Adeoidectomy-for persistent & rec. infection

    Precautions:

    Grommet insertion : in case of SOM

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    A. Absolute1. Recurrent infections of throat

    2. Peritonsillar abscess

    3. Tonsillitis causing febrile seizures

    4. Hypertrophy of tonsils causing obstruction

    5. Suspicion of malignancy

    B. Relative1. Diphtheria carriers,

    2. Streptococcal carriers

    3. Chronic tonsillitis with bad taste or halitosis

    4. Recurrent streptococcal tonsillitis in a patient with valvular heart disease

    C. As a Part of Another Operation1. Palatopharyngoplasty

    2. Glossopharyngeal neurectomy.

    3. Removal of styloid process.

    d d d

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    Adenoidectomy-Indications

    Recurrent or chronic sinusitis or adenoiditis Poorly understood - possibly caused by obstructive

    adenoid tissue causing stasis of secretions predisposingthe nasal cavity to infection.

    Otitis media Proximity of adenoid tissue to eustachian tube

    Adenoidectomy can be recommended on 1st set of tubes

    if nasal obstruction and recurrent rhinorrhea is presentor on 2nd set of tubes if needed.

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    Epidemic of polio Age below 3 years Acute infections Blood dyscrasiasis: hemophilia, purpura

    Uncontrolled systemic diseases like diabetes and heartdiseases

    Velopharyngeal insufficiency Overt cleft palate, submucous (covert) cleft

    Neurologic or neuromuscular abnormality leading to impaired palate

    function Anemia

    ABSOULTECONTRAINDICATION-PULSATILE TONSILE

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    Most common lab test is a CBC

    Coagulation studies when the history orphysical examination suggests a bleedingdisorder.

    Lateral Neck/Adenoid films

    R ' iti

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    Rose's position for tonsillectomy. Neck is extended by a sand bagunder the shoulders and the head is supported on a ring.

    Downloaded from: StudentConsult (on 6 December 2012 06:54 PM)

    2005 Elsevier

    Rose's position

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    DAVIS MOUTH GAG BOYLES TONGUE BLADE

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    Set of instruments for tonsillectomy.(1) Knife in kidney tray, (2) & (3) Toothed and non-toothedWaugh's forceps, (4) Tonsil holding forceps, (5) Tonsil dissector and anterior pillar retractor, (6) Luc's

    forceps, (7) Scissor, (8) Curved artery forceps, (9) Negus artery forceps, (10) Tonsillar snare, (11) BoyleDavis mouth gag with three sizes of tongue blades, (12) Doyen's mouth gag, (13) Adenoid curette, (14)

    Tonsil swabs, (15) Nasopharyngeal pack, (16) Towel clips.Downloaded from: StudentConsult (on 6 December 2012 06:54 PM) 2005 Elsevier

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    DISSECTION GUILLOTINE ELECTROCAUTERY

    CRYOSURGERY

    LASER HARMONIC

    SCALPEL

    COBLATION MICRODEBRIDER

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    TONSILLAR DISSECTOR

    EVES TONSILLAR SNARE

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    (A) Tonsil being dissected from its bed. (B) The pedicle at the lower pole oftonsil being cut with a snare.

    Downloaded from: StudentConsult (on 6 December 2012 06:54 PM)

    2005 Elsevier

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    Cold HotDissection and snare Electrocautery

    Guillotine method Laser tonsillectomy (CO2

    or KTP)Intracapsular (capsule

    preserving) tonsillectomy

    Coblation tonsillectomy

    Harmonic scalpel Radio frequency

    Plasma-mediated ablation

    technique

    Cryosurgical technique

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    TONSIL GUILLOTINE GUILLOTINE TONSILLECTOMY

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    HARMONIC SCALPEL

    KNIFE HARMONIC SCALPEL TONSILLECTOMY

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    Primary haemorrhage O t th

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    Primary haemorrhage.Occurs at thetime of operation. It can be controlled by

    pressure, ligation or electrocoagulation of thebleeding vessels

    . Reactionary haemorrhage.Occurswithin a period of 24 hours and can be

    controlled by simple measures such asremoval of the clot, application of pressure

    or vasoconstrictor.

    3 Injury to teeth

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    3. Injury to teeth.

    4. Aspiration of blood.

    5. Facial oedema. Some patients get oedema of

    the face particularly of the eyelids.

    6. Surgical emphysema. Rarely occurs due to

    injury to superior constrictor muscle.

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    profuse bleeding, general anaesthesia is given

    and bleeding vessel is electrocoagulated or

    ligated

    approximation of pillars

    , external carotid ligation

    Transfusion of blood or plasma

    Systemic antibiotics are given for control of infection

    topical application of dilute adrenaline or

    hydrogen peroxide with pressure usually

    suffice.

    Delayed Complications cont..

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    Delayed Complications cont..

    1. Infection

    2. Lung complications

    3. Scarring in soft palate and pillars.

    4. Tonsillar remnants.

    5. Hypertrophy of lingual tonsil

    Thank u

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    Thank u