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ACLS Arrhythmias and Treatment Modalities
Presented by CMR CPR (a division of CMR Medical Supply, LLC)
An American Heart Association Affiliated Training Site
DISCLAIMER
• The following information is provided by the American Heart Association.
• Monies collected do not represent income for the American Heart
Association. • Please review and study your American
Heart Association ACLS Manual before attempting to complete the AHA ACLS
Course.
QRS Complex
P wave indicates Atrial depolarization. QRS complex indicates Ventricular depolarization
T wave indicates re-polarization
Normal Sinus Rhythm
1st Degree Heart Block
• Usually benign, very common
• Not treatable in ACLS
• Progressively gets worse over time
• Not actually a block- just a delay in conduction
• PRI- >20 (4 small boxes)
• Normally regular
2nd Degree Heart Block, type 1 aka “Wenckebach”
• Usually Irregular
• Not treatable in ACLS
• PRI- Long, long, longer, DROP- must be a Wenckebach!
2nd Degree, Type 2 Heart Block
• Blocked P waves/dropped QRS Complexes
• Usually 1 or 2 additional P waves
• This is getting worse…
3rd Degree Heart Block (Complete Heart Block)
• Malignant
• Usually Bradycardic, Irregular
• Won’t take long…NEED TO TREAT!
• P waves regular, not associated w/ QRS complexes
Supra Ventricular Tachycardia (SVT)
• Firing somewhere above the Ventricles
• Treatable over 150 BPM per ACLS
• Regular and FAST!
Ventricular Tachycardia (V- tach with or w/o Pulses)
• Extremely dangerous arrhythmia
• Patient wont last long
• Wide and Bizarre pattern, but regular
• Shock-able rhythm (if no pulse present)
Torsades de Pointes
• Translates to: “Twisted of the Spikes”
• Described as Polymorphic Tachycardia
• May degenerate to V-Fib if persistent
• Caused by HYPOkalemia/HYPOmagnasemia
Ventricular Fibrillation (V-Fib)
• Won’t have a pulse
• Fine or coarse
• Shock-able rhythm
• Patient is in CARDIAC ARREST at this point
Pulseless Electrical Activity (PEA)
• ANY rhythm can be PEA as long as it doesn’t have a PULSE!
• YOU MUST CHECK A PULSE WITH EVERY RHYTHM- In a Cardiac Arrest!
Asystole (Flat line)
• Heart not producing ANY electrical activity
• NON-Shock-able Rhythm
• Patient is DEAD
Acute M.I. (Leads 2,3 & aVF)
ACLS DRUGS
Atropine
• Derived from the Nightshade Plant (deadly)
• Dilates pupils, increases heart rate
• Used to treat symptomatic bradycardia
• No longer used for PEA
DOSE: 0.5 mg with a maximum of 3.0 mg
ACLS DRUGS
Adenosine
• Inhibits neurotransmitters
• “Resets” heart
• Asystole for 3-5 seconds
• Causes a transient heart block in the AV node
• Used to treat Asymptomatic SVT over 150bpm
• NOT for wide complex IRREGULAR V -Tach
DOSE: 6.0 mg then 12.0 mg
ACLS DRUGS
Amiodarone Bolus
• Anti-arrythmic
• Works on the Atria and the Ventricles
DOSE: 300 mg then 150 mg. MAX: 450 mg
ACLS DRUGS Epinephrine (Bolus)
• Hormone naturally occurring in the body
• Affects the Sympathetic Nervous System
• Constricts blood vessels, increases peripheral resistance
• Increases Heart Rate (Inotropic effects and Chronotropic effects) (electricity and rate)
DOSE: 1.0 mg NO MAX DOSE!
ACLS DRUGS Vasopressin
• Anti-diuretic hormone
• Retains water in the body and constricts blood vessels
• May be used in cardiac arrest in place of 1st or second dose of epinephrine
• Half life is 10-20 minutes
DOSE: 40 units
ACLS DRUGS
Dopamine
• Second-line drug for symptomatic bradycardia when atropine is not effective
• Used for cardiogenic shock in the absence of hypovolemia
Dose: 2-10 micrograms/kg/min infusion
ACLS DRUGS
Epinephrine (Infusion)
• Second-line drug for symptomatic bradycardia when atropine is not effective. Choose EPI or Dopamine
Dose: 2-10 micrograms/kg/min infusion
ACLS DRUGS
Magnesium Sulfate
• For the treatment of Torsades with pulses present
Dose: 1-2 Grams
H’s and T’s
• Hypovolemia
• Hypoxia
• Hydrogen Ions (acidosis)
• Hyper/Hypo kalemia
• Hypothermia
• Toxins
• Tamponade (cardiac)
• Tension Pneumothorax
• Thrombosis (coronary)
Circular Algorithm
Treatment Modalities per ACLS
• V-FIB or (V-TACH w/o pulses) are the only shock-able rhythms.
• Start at 360J, and continue at 360J* (With a MONO-phasic Defibrillator)
• High Quality CPR
• EPI or (Vasopressin, 1st or 2nd dose)
• Amiodarone- 300mg, then 150mg (450mg MAX)
SVT
• Treatable at 150 BPM
• Use Valsalva Maneuver First (Think BLS)
• Stable= Drugs. Adenosine 6mg, 12mg, done
• Unstable= Electricity. Synchronized Cardioversion. (sedate first) 100 J,200J, 300J 360J* (discussed in class)
• MAKE SURE YOU PUSH SYNCH BUTTON!
• Be careful w/ rapid A fib- throw a clot…BAD
V-TACH w/PULSES (Regular)
• Dangerous arrhythmia- PT. wont last long
• Valsalva maneuver first (Think BLS)
• Stable= Use Adenosine
• Unstable= Electricity (Monophasic)
• 100J,200J, 300J 360J
• Try to sedate first. Don’t delay treatment.
• PT. will usually be UNSTABLE!
V-Tach (Irregular, Torsades)
• Dangerous arrhythmia- PT. wont last long
• Valsalva maneuver first (Think BLS)
• Stable= Adenosine- not indicated.
• Magnesium Sulfate
• Electricity. 100J,200J, 300J 360J (Monophasic)
Bradycardias
• Less than 60 BPM
• Stable= DO NOT TREAT- watch patient
• Unstable= (1) Atropine (2) Dopamine or EPI Drip (3)Pacing…
Pacing
• Apply D-Fib Pads on Pt
• Set Pacer at 80 bpm
• Increase mA until 1 pacer spike precedes QRS
• Check Femoral Artery for pulse that matches monitor
• If is doesn’t, increase mA until it does
PEA (Pulsless Electrical Activity)
• A rhythm that appears on a monitor to have a pulse, but does not when the pulse is checked
• High quality CPR
• Epinephrine, 1.0 mg
• NON- SHOCKABLE!
Complete Heart Block (3rd degree)
• The use of Atropine is not indicated
• Transcutaneous pacing
• Fluids
• Oxygen
• Cardiology consult