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™ Revised 2009 by Dorothy W. Bird, MD Suresh Agarwal, MD, FACS Department of Surgery Boston University Medical Center Based on the original presentation

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Page 1: ™ Revised 2009 by Dorothy W. Bird, MD Suresh Agarwal, MD, FACS Department of Surgery Boston University Medical Center Based on the original presentation

PROPERTIESAllow user to leave interaction: AnytimeShow ‘Next Slide’ Button: Show alwaysCompletion Button Label: View Presentation

Page 2: ™ Revised 2009 by Dorothy W. Bird, MD Suresh Agarwal, MD, FACS Department of Surgery Boston University Medical Center Based on the original presentation

Revised 2009 by

Dorothy W. Bird, MD Suresh Agarwal, MD, FACS

Department of SurgeryBoston University Medical Center

Based on the original presentation created by: N.K. Durrani, MD; M. McCann, DO; M.M. Brandt, MD, FACS, FCCM; P. Patton, MD, FACS; H.M. Horst, MD, FACS, FCCM; I. Rubinfeld, MD

Dept. of Trauma SurgeryHenry Ford Hospital, Detroit

Surgical Issues in Critical Care Medicine

Page 3: ™ Revised 2009 by Dorothy W. Bird, MD Suresh Agarwal, MD, FACS Department of Surgery Boston University Medical Center Based on the original presentation

™ Slide 3

Surgical Complications

• Airway: airway loss and emergent management

• Pulmonary: simple and tension pneumothorax

• Cardiac: tamponade

• GI: abdominal pain, ileus, ischemia, abdominal compartment syndrome, GI bleeding

• Extremities: vascular occlusion syndromes, compartment syndrome

Page 4: ™ Revised 2009 by Dorothy W. Bird, MD Suresh Agarwal, MD, FACS Department of Surgery Boston University Medical Center Based on the original presentation

™ Slide 4

Surgical Airways

• Only reason not to intubate is inability to do so, nonsurgical always preferred: i.e., orotracheal, nasotracheal

• Relative contraindications to intubation

– C-spine instability

– Midface fractures

– Laryngeal disruption

– Obstruction of lumen

Page 5: ™ Revised 2009 by Dorothy W. Bird, MD Suresh Agarwal, MD, FACS Department of Surgery Boston University Medical Center Based on the original presentation

™ Slide 5

Emergent Surgical Airway

• Needle cricothyroidotomy:

– 12-14G Angiocath +syringe

– Hyperextend neck

– Palpate cricothyroid membrone

– Apply Betadyne, Lidocaine

– Advance needle at 45o angle until air is aspirated

– Advance catheter, remove needle, attach hub to 3-mm ET adapter and oxygen

• Only useful for 45min due to poor CO2 exchange!

Page 6: ™ Revised 2009 by Dorothy W. Bird, MD Suresh Agarwal, MD, FACS Department of Surgery Boston University Medical Center Based on the original presentation

™ Slide 6

Emergent Surgical Airway

• Cricothyroidotomy

– Hyperextend neck

– Palpate cricothyoid membrane

– Apply Betadyne, Lidocaine

– 3-4cm midline vertical incision through cervical fascia and strap muscles

– Incise cricothyroid membrane horizontally; use hemostat to hold open

– Insert 5-7mm tracheostomy tube (or ET tube), attach to oxygen supply

• Convert to formal tracheostomy in 24h!

Page 7: ™ Revised 2009 by Dorothy W. Bird, MD Suresh Agarwal, MD, FACS Department of Surgery Boston University Medical Center Based on the original presentation

™ Slide 7

Surgical Airway

• Tracheostomy: Rarely for emergencies

– Usually for ventilator weaning

• Many techniques (percutaneous, surgical)

• Emergency Indications:

– Laryngeal crush injury

– Fracture of thyroid or cricoid membranes

– Very small children

Page 8: ™ Revised 2009 by Dorothy W. Bird, MD Suresh Agarwal, MD, FACS Department of Surgery Boston University Medical Center Based on the original presentation

™ Slide 8

Airway Emergency: Massive Hemoptysis

• Due to pulmonary, bronchial, or innominate artery injury/disease

• Results from erosion (slow, with herald bleed) or iatrogenic (tracheostomy, trauma)

• Bronchoscopy to determine source

• Bronchial blocker for isolation

• Angiography: embolize bleeding source

• Emergent lobectomy or sternotomy if uncontrolled

Innominate a.

trachea

Page 9: ™ Revised 2009 by Dorothy W. Bird, MD Suresh Agarwal, MD, FACS Department of Surgery Boston University Medical Center Based on the original presentation

™ Slide 9

Tracheoinnominate Artery Fistula

• Dreaded complication of tracheostomy (1%)

• Due to:

– Erosion of the artery by tracheostomy tube or

– High pressure cuff directly injurs artery

• Temporize by:

– Insert endotracheal tube into tracheostomy stoma, inflate cuff

– Apply downward, outward tamponade to fistula with finger in tracheostomy stoma

Page 10: ™ Revised 2009 by Dorothy W. Bird, MD Suresh Agarwal, MD, FACS Department of Surgery Boston University Medical Center Based on the original presentation

™ Slide 10

Surgical Pulmonary Emergencies

• Pneumothorax (simple): partial or complete collapse —increases pulmonary shunt

– Chest tube in emergency

– Attempt catheters as well

– Treat “conservatively” in stable asymptomatic patients

– Aggressive therapy if on positive pressure

– Can progress to tension pneumothorax

Page 11: ™ Revised 2009 by Dorothy W. Bird, MD Suresh Agarwal, MD, FACS Department of Surgery Boston University Medical Center Based on the original presentation

™ Slide 11

Tension Pneumothorax

• True Surgical Emergency!

• Signs:– Decreased breath sounds– Ipsilateral tympany– Tracheal shift– Distended neck veins– Asymmetric chest expansion

• Hypotension

• CXR: mediastinal shift

• Emergent decompression– Chest tube– Temporary needle decompression

Tracheal shift

pneumothorax

Page 12: ™ Revised 2009 by Dorothy W. Bird, MD Suresh Agarwal, MD, FACS Department of Surgery Boston University Medical Center Based on the original presentation

™ Slide 12

Chest Tube Insertion

• Sterile prep and drape

• +/- Local anesthesia- 1% lidocaine to pleura

• 2-3cm incision at midaxillary line, 5th intercostal space

• Blunt dissection with finger/clamp to pleura

• Listen/feel for gush of air exiting pleural space

• Insert 36F chest tube apically, posteriorly; secure with suture, occlusive dressing

• Attach distal end of tube to suction (-20cm water) with water seal

Page 13: ™ Revised 2009 by Dorothy W. Bird, MD Suresh Agarwal, MD, FACS Department of Surgery Boston University Medical Center Based on the original presentation

™ Slide 13

Hemothorax

• Surgical Indications:

• Massive hemothorax = >1500mL immediate return of blood on tube thoracostomy

• Persistent hemothorax = 300mL/h x 3hours

• >1500mL blood/24h

• Chest tube with massive air drainage, or GI contents

Page 14: ™ Revised 2009 by Dorothy W. Bird, MD Suresh Agarwal, MD, FACS Department of Surgery Boston University Medical Center Based on the original presentation

™ Slide 14

Cardiac Tamponade

• Blood in pericardial space, compresses heart

• Beck’s triad: hypotension, jugular venous distension, distant heart sounds

• Echocardiogram: impaired diastolic filling

• Treatment: needle decompression or pericardial window

• Image from: http://upennanesthesiology.typepad.com/photos/uncategorized/2007/07/26/tamponade2_b_milas.jpg

Fluid in pericardial space

Page 15: ™ Revised 2009 by Dorothy W. Bird, MD Suresh Agarwal, MD, FACS Department of Surgery Boston University Medical Center Based on the original presentation

™ Slide 15

Abdominal Pain

• Abdominal pain syndromes in the ICU:

– Pancreatitis

– Acalculous cholecystitis

– Bowel ischemia

– Bowel obstructive syndromes

Page 16: ™ Revised 2009 by Dorothy W. Bird, MD Suresh Agarwal, MD, FACS Department of Surgery Boston University Medical Center Based on the original presentation

™ Slide 16

Pancreatitis

• Epigastric/upper quadrant pain, radiates to back

• + Nausea, vomiting, fever

• ICU Etiology:

– Medications: furosemide, thiazide diuretics, metronidazole, bactrim, ACE-inhibitors, many others

– EtOH, gallstones, ERCP, trauma

– Hyperlipidemia (triglycerides >1,000mg/dl), hypercalcemia

Page 17: ™ Revised 2009 by Dorothy W. Bird, MD Suresh Agarwal, MD, FACS Department of Surgery Boston University Medical Center Based on the original presentation

™ Slide 17

Pancreatitis

• Mortality predicted by Ranson Criteria:

– Score 0 to 2 : 2% mortality

– Score 3 to 4 : 15% mortality

– Score 5 to 6 : 40% mortality

– Score 7 to 8 : 100% mortality

• Management

– NPO, IVF, antibiotics if infection or gall stones

– Treat underlying cause

– Surgery only for infected necrosis

On admission Within 48 hours

Age >55 years Hct decreases by >10

WBC >16,000 BUN increases by >5

Glucose >200mg/dl Calcium <8mg/dl

LDH >350 PaO2 <60mmHg

AST >250 Fluid Requirement >6L

Base deficit >4mEq/L

Page 18: ™ Revised 2009 by Dorothy W. Bird, MD Suresh Agarwal, MD, FACS Department of Surgery Boston University Medical Center Based on the original presentation

™ Slide 18

Acalculous Cholecystitis

• 5% -10% of all cases of acute cholecystitis

– Observed in the setting of very ill patients, especially trauma and burn victims, also long-term TPN (>3 months)

• Signs/Symptoms: RUQ pain, fever, leukocytosis

• Diagnosis: CT or US: pericholecystic fluid, NO STONES

• Etiology: unclear; stasis vs ischemia

– Higher incidence of gangrene and perforation compared to calculous disease, greater mortality (40%)

• Management: IV fluid, IV antibiotics, emergent cholecystectomy (or cholecystotomy if surgical risk is high and risk of perforation is low)

Page 19: ™ Revised 2009 by Dorothy W. Bird, MD Suresh Agarwal, MD, FACS Department of Surgery Boston University Medical Center Based on the original presentation

™ Slide 19

Bowel Ischemia

• Etiology:– ICU patients: Nonocclusive mesenteric ischemia (NOMI) -

splanchnic low flow and/or vasoconstriction• Seen in hemodynamically unstable patients• Decreased CO, hypovolemia, vasoconstrictor medications

– General population: mesenteric arterial embolus, mesenteric arterial thrombus, mesenteric venous thrombus

• NOMI Signs: Abdominal pain, leukocytosis, GI mucosal sloughing, bleeding

• NOMI Diagnosis: Angiography

• NOMI Treatment: optimize volume status, relieve splanchnic vasocontriction; selective intraarterial vasodilators (papaverine, glucagon)

Page 20: ™ Revised 2009 by Dorothy W. Bird, MD Suresh Agarwal, MD, FACS Department of Surgery Boston University Medical Center Based on the original presentation

™ Slide 20

Bowel Obstruction

• Mechanical– Gut lumen is blocked due to foreign body, tumor,

intussusception, adhesions; partial vs complete– Open loop obstruction: amenable to proximal decompression;

use NG tube– Closed loop obstruction: inflow and outflow blocked: hernia

incarceration, torsion around adhesive band, volvulus; surgical emergency!

• Functional (neurogenic)– Ileus (small bowel): +/-NG tube, judicious narcotic use– Olgvie’s pseudoobstruction (large bowel): neostigmine +/-

colonoscopic decompression if cecum>10-12cm or if symptomatic >48h; correct electrolytes, reduce narcotics, NG tube

Page 21: ™ Revised 2009 by Dorothy W. Bird, MD Suresh Agarwal, MD, FACS Department of Surgery Boston University Medical Center Based on the original presentation

™ Slide 21

Abdominal Compartment Syndrome

• Acute increase in intra-abdominal pressure with resultant critical organ dysfunction

• Seen in trauma patients after laparoptomy, non-operative hepatic or renal trauma victims, burn victims, any patient who receives large-volume resuscitation

Page 22: ™ Revised 2009 by Dorothy W. Bird, MD Suresh Agarwal, MD, FACS Department of Surgery Boston University Medical Center Based on the original presentation

™ Slide 22

Abdominal Compartment Syndrome

• Consequences of elevated intraabominal pressure:

– decreases ventilation→ hypoxia, acidosis

– reduces venous return →decreased cardiac output

– venous congestion → reduced capillary perfusion, ischemia, inflammation

– decreased blood flow to kidney →oliguia, renal failure

– decreased blood flow to liver, gut →impaired function

• Early recognition and diagnosis are vital to prevent complications!

– Identify those at risk, measure baseline IAP!

Page 23: ™ Revised 2009 by Dorothy W. Bird, MD Suresh Agarwal, MD, FACS Department of Surgery Boston University Medical Center Based on the original presentation

™ Slide 23

Abdominal Compartment Syndrome

• Clinical triad:

– Tense, distended abdomen

– Increased airway pressures

– Oliguira (despite ample resuscitation)

• Diagnosis: Bladder pressure

– Surrogate for intraabdominal pressure

– Bladder filled with 50 cc of sterile saline via Foley and pressure monitor connected to side port with 18-gauge needle

Page 24: ™ Revised 2009 by Dorothy W. Bird, MD Suresh Agarwal, MD, FACS Department of Surgery Boston University Medical Center Based on the original presentation

™ Slide 24

Page 25: ™ Revised 2009 by Dorothy W. Bird, MD Suresh Agarwal, MD, FACS Department of Surgery Boston University Medical Center Based on the original presentation

™ Slide 25

Abdominal Compartment Syndrome

• Intraabdominal pressure (IAP)

– Normal: <10mm Hg

– Intraabdominal hypertension (IAH): ≥12mmHg

– Abdominal compartment syndrome (ACS): ≥20mmHg with new organ dysfunction

– WSACS IAP Grading:• I 12-15mmHg

• II 16-20mmHg

• III 21-25mmHg

• IV >25mmHg

Page 26: ™ Revised 2009 by Dorothy W. Bird, MD Suresh Agarwal, MD, FACS Department of Surgery Boston University Medical Center Based on the original presentation

™ Slide 26

Abdominal Compartment Syndrome

• Management:

– Prevention! Judicious resuscitation!

– Neuromuscular blockade

– Diuresis (only with hemodynamic monitoring)

– Catheter drainage: bedside ultrasound to guide catheter drainage of intraabdominal fluid

– Decompressive laparotomy- definitive• Abdominal fascia left open, often with VAC or Bogota bag

covering wound

• Delayed primary closure

Page 27: ™ Revised 2009 by Dorothy W. Bird, MD Suresh Agarwal, MD, FACS Department of Surgery Boston University Medical Center Based on the original presentation

™ Slide 27

Bogota Bag

Page 28: ™ Revised 2009 by Dorothy W. Bird, MD Suresh Agarwal, MD, FACS Department of Surgery Boston University Medical Center Based on the original presentation

™ Slide 28

Upper GI Bleeding

• Gastric (ulcer vs. gastritis)

• Duodenal

• Esophageal varices

• Mallory-Weiss

Page 29: ™ Revised 2009 by Dorothy W. Bird, MD Suresh Agarwal, MD, FACS Department of Surgery Boston University Medical Center Based on the original presentation

™ Slide 29

Upper GI Bleeding

• Immediately:– 2 large-bore peripheral IVs

– 2 L crystalloid

– STAT labs: CBC, PT/PTT, Type & screen

– NGT, gastric lavage

– Foley catheter

– Consider central line (CVP) or Swan catheter

Page 30: ™ Revised 2009 by Dorothy W. Bird, MD Suresh Agarwal, MD, FACS Department of Surgery Boston University Medical Center Based on the original presentation

™ Slide 30

Upper GI Bleeding

• Management

– PPI, H2-blocker

– EGD

– Arteriography

• Treat Varices: vasopressin, octreotide, sclerotherapy, Sengstaken-Blakemore tube, TIPS

• Operative intervention if bleeding remains uncontrolled

Page 31: ™ Revised 2009 by Dorothy W. Bird, MD Suresh Agarwal, MD, FACS Department of Surgery Boston University Medical Center Based on the original presentation

™ Slide 31

Mallory-Weiss tear

• UGI bleeding after violent emesis

– Gastric mucosal tear at cardia

– Typically (not always) in alcoholic patients

• Usually stops spontaneously

• May attempt Blakemore tube using gastric balloon for direct pressure.

• Nonoperative treatment: endoscopic electrocoagulation, banding, injection

• Operative intervention rarely needed: oversew laceration

Page 32: ™ Revised 2009 by Dorothy W. Bird, MD Suresh Agarwal, MD, FACS Department of Surgery Boston University Medical Center Based on the original presentation

™ Slide 32

Lower GI Bleeding

• Most arise from the colon and rectum

• Large bowel etiologies: diverticula, angiodysplastic lesions, neoplasms, IBD, hemorrhoids, and anal fissures

• Small bowel etiologies: neoplasm, IBD, Meckel’s diverticulum

Page 33: ™ Revised 2009 by Dorothy W. Bird, MD Suresh Agarwal, MD, FACS Department of Surgery Boston University Medical Center Based on the original presentation

™ Slide 33

Lower GI Bleeding

• Initial management: as for upper GI bleeding

• Diagnosis:

– Rectal exam

– Colonoscopy

– Radionuclide scan• Bleeding scan

– Arteriography

Page 34: ™ Revised 2009 by Dorothy W. Bird, MD Suresh Agarwal, MD, FACS Department of Surgery Boston University Medical Center Based on the original presentation

™ Slide 34

Lower GI Bleeding

Bleeding scan

Source of LGIB

Angiography

Source of LGIB

From: http://brighamrad.harvard.edu/Cases/bwh/hcache/126/full.html

Page 35: ™ Revised 2009 by Dorothy W. Bird, MD Suresh Agarwal, MD, FACS Department of Surgery Boston University Medical Center Based on the original presentation

™ Slide 35

Lower GI Bleeding

• Management:

• Arteriographic intervention: vasopressin, coils, gel foam

• 80% success, 50% rebleed risk

• Operative: hemodynamic unstable with >8 units PRBC

• Localization is key, unlocalized LGI bleeding will lead to a blind subtotal colectomy, which is a higher mortality procedure for your patient!

Page 36: ™ Revised 2009 by Dorothy W. Bird, MD Suresh Agarwal, MD, FACS Department of Surgery Boston University Medical Center Based on the original presentation

™ Slide 36

Cold Legs

• Acute arterial embolus

– Signs: 6 Ps: pain, pallor, pulselessness, paresthesia, poikilothermia, paralysis

– Contralateral leg is normal

– No chronic ischemic changes

– Etiology: atrial fibrillation (most common)• Embolus usually obstructs common femoral artery

• Treatment: Embolectomy +/- fasciotomy

• Rare: aortoiliac emboli- loss of pulses to both feet, requires bilateral embolectomies

Page 37: ™ Revised 2009 by Dorothy W. Bird, MD Suresh Agarwal, MD, FACS Department of Surgery Boston University Medical Center Based on the original presentation

™ Slide 37

Cold Legs

• Acute arterial thrombosis

– Signs: 6 Ps: pain, pallor, pulselessness, paresthesia, poikilothermia, paralysis

– History of claudication, signs of chronic ischemia

– Poor pulses in contralateral leg

– Not associated with atrial fibrillation

• Treatment: heparin anticoagulation, OR for thrombectomy or angiography for catheter-directed thrombolysis

Page 38: ™ Revised 2009 by Dorothy W. Bird, MD Suresh Agarwal, MD, FACS Department of Surgery Boston University Medical Center Based on the original presentation

™ Slide 38

Swollen Legs

• Most common “surgical” etiology is DVT

• Does your patient need an IVC filter?

• Indications:

– DVT and

– Contraindication to anticoagulation and

– High risk of PE

• Percutaneous placement of IVC filter (femoral or jugular)

Page 39: ™ Revised 2009 by Dorothy W. Bird, MD Suresh Agarwal, MD, FACS Department of Surgery Boston University Medical Center Based on the original presentation

™ Slide 39

Phlegmasia Cerulea Dolens

• Simultaneous thrombosis of iliac, femoral, common femoral, and superficial femoral veins

• Associated with other critical illnesses, cachexia, dehydration

• Appearance: massively swollen, blue, mottled

• Treatment:

– Limb elevation

– Heparin anticoagulation

– +/- catheter-directed thrombolysis

– +/- thrombectomy

Page 40: ™ Revised 2009 by Dorothy W. Bird, MD Suresh Agarwal, MD, FACS Department of Surgery Boston University Medical Center Based on the original presentation

™ Slide 40

Extremity Compartment Syndrome

• Acute increase in pressure within myofascial compartment of an extremity

• Can occur in any compartment, most often lower extremity, anterior compartment

• Complications related to compression of contents of compartment

• Causes rhabdomyolysis, ischemic neuritis, arterial insufficiency, venous gangrene, and limb loss

Page 41: ™ Revised 2009 by Dorothy W. Bird, MD Suresh Agarwal, MD, FACS Department of Surgery Boston University Medical Center Based on the original presentation

™ Slide 41

Compartment Syndrome

• Etiology: increase in muscle swelling, hematoma, or interstitial fluid; often secondary to reperfusion injury, burns, fractures, crush injury, tight cast

• Signs/Symptoms:– Extreme pain on flexion is often first sign– Swollen, tense extremity– Loss of sensation first neurologic sign followed by weakness– Last sign is decrease in pulses

• Diagnose: Direct pressure measurement using 18-gauge needle and arterial monitor or Stryker monitor– Pressure >20mmHg OR clinical suspicion– Delta P method: diastolic blood pressure – compartment

pressure ≤30mmHg is indicative of compartment syndrome

Page 42: ™ Revised 2009 by Dorothy W. Bird, MD Suresh Agarwal, MD, FACS Department of Surgery Boston University Medical Center Based on the original presentation

™ Slide 42

Compartment Syndrome

• Treatment: Release pressure immediately!

• Evacuate hematoma

• Perform fasciotomy

– +/- VAC wound therapy

– delayed closure

– split-thickness skin graft

Page 43: ™ Revised 2009 by Dorothy W. Bird, MD Suresh Agarwal, MD, FACS Department of Surgery Boston University Medical Center Based on the original presentation

™ Slide 43

References

• Koster W, Strohm PC, Sudkamp NP. Acute compartment syndrome of the limb. Injury, Int. J. Care Injured (2005) 36, 992-998.

• Ridley RW, Zwischenberger JB. Tracheoinnominate fistula: surgical managemnt of an iatrogenic disaster. The Journal of Laryngology and Otology (2006) 120, 676-680.

• An G, West MA, Abdominal compartment syndrome: A concise clinical review. Crit Care Med (2008) 36, 1304-1310.

• Maerz L, Kaplan LJ. Abdominal compartment syndrome. Crit Care Med (2008) 36 Suppl, S212-215.

Page 44: ™ Revised 2009 by Dorothy W. Bird, MD Suresh Agarwal, MD, FACS Department of Surgery Boston University Medical Center Based on the original presentation

™ Slide 44

References

• Greenfield’s Surgery: Scientific Principles and Practice. Fourth Edition. Mulholland MW, Lillemoe KD, Doherty GM, Maier RV, Upchurch Jr. GR. New York, NY, Lippincott Williams and Wilkins.

• ACS Surgery: Principles and Practice. Online Edition. Ashley SW et al. http://www.acssurgey.com

• Bowers Rebecca C, Weaver Jeffrey D, "Chapter 8. Compromised Airway" (Chapter). Stone CK, Humphries RL: CURRENT Diagnosis & Treatment: Emergency Medicine, 6th Edition: http://www.accessmedicine.com/content.aspx?aID=3118968.

• Gomella LG, Haist SA, "Chapter 13. Bedside Procedures" (Chapter). Gomella LG, Haist SA: Clinician's Pocket Reference: The Scut Monkey, 11th Edition: http://www.accessmedicine.com/content.aspx?aID=2694363.