PENYAKIT KULIT YANG DISEBABKAN OLEH BAKTERIYunita Hapsari Bagian Ilmu Kesehatan Kulit dan KelaminFakultas Kedokteran Universitas Mataram
PYODERMAInfeksi pada kulit, yang disebabkan oleh bakteri yg membentuk nanah atau piogenik dan mudah menularEtiologi :Staphylococcus S.aureusStreptococcus Streptococcus grup AKedua-duanya
Faktor predisposisi :Daya tahan tubuh rendahHigiene individu kurangHigiene lingkungan kurangTelah ada penyakit kulit sebelumnya
Klasifikasi pyoderma :PRIMERSEKUNDERImpetigo FolikulitisFurunkelKarbunkelParonikia Ektima ImpetigenisataHidradenitis supurativaIntertrigoUlkus
IMPETIGOPyoderma superfisialEtiology : Streptococcus grup A S.aureus2 bentuk :BulosaNon-bulosa / krustosa
Impetigo vesiko-bulosa, cacar monyetEtiology : phage group II S.aureus (strain 77 & 55) exfoliatin toxin type A and BClinical : newborn and older infantsVesicle flaccid bullae (rapid progression)Nikolsky sign (-)Bullae : clear yellow fluid dark yellow & turbidMargin : sharply demarcated w/o erythematous halow/i a day or two bullae rupture & collapseIMPETIGO BULOSA
IMPETIGO BULOSA
Lab : Pengecatan GramKultur histoPADD/ :Dermatitis kontakInsect biteErupsi obat tipe bulosaIMPETIGO BULOSA
Complication :CellulitisLymphangitisBacteremiaOsteomyelitisSeptic arthritisPneumonitisSepticemiaSSSSIMPETIGO BULOSA
Clinical finding:Pruritus; soreness; burning ; painlessCommonly on the skin of the face (around the nares) or extremities after traumaTransient pustule or vesicle honey-colored crusted plaque (2cm) w/i surrounding erythemaNo constitutional symptomsRegional lymphadenopathy (90%)
IMPETIGO NON BULOSA/KRUSTOSA
IMPETIGO NON BULOSA/KRUSTOSA
Lab : Gram stainCulture DD/ :Atopic dermatitisAllergic contact dermatitisVaricella Dermatophyte infectionIMPETIGO NON BULOSA/KRUSTOSA
Complication :Untreated process may persist and new lesions over the course and for several weeksResolve spontaneusly except there is some underlying cutaneous diseaseErysipelasCellulitisBacteremiaMajor serious sequela acute post-streptococcal glomerulonephritisIMPETIGO NON BULOSA/KRUSTOSA
MANAGEMENT OF IMPETIGOWashing (removal of dirt, crusts and debris by soaking with soap & water)Good hygienePharmacology: topical and systemicTopical & Systemic Antibiotic
FOLIKULITISDef : a pyoderma that begins within the hair folliclesClassified : Depth of invasion : superficial and deepMicrobial etiology : bacterial, fungal, viral and parasite infestation
FOLIKULITIS SUPERFISIALImpetigo Bockhart; follicular impetigoEtiology : S.aureusClinical finding:Location : scalp (children); beard area, axillae, extremities, buttocks (adults)A small, fragile, dome-shaped pustule infudibulum of a hair follicleDD/ : - gram-negative folliculitisPityrosporum folliculitisHerpes simplexDeep folliculitis sycosis barbae
FURUNKELBoil / bisulDeep-seated inflammatory nodule that develops around a hair follicle, usually from a preceding, more superficial folliculitis and often evolving into an abcessEtiology: S.aureusClinical finding :Pain surrounding the lesionNodule (hard, tender, red folliculocentric) enlarge fluctuant abcess formation ruptureSingle/multiple
Furunkel / Furunkulosis ?
FURUNKELLab:LeukocytosisGram stain of pusCulture
ComplicationNot commonRecurrent furunculosis
KARBUNKELLarger, more serious inflammatory lesion with a deeper baseExtremely painfulFever, malaiseLocation : nape of the neck, the back or thighsEfl : red, indurated, multiple pustules confluent
MANAGEMENT OF FURUNCLE & CARBUNCLEWashing and hygieneLocalized local application of moist heatSystemic systemic antibiotic Incision and draining the lesions
EKTIMAA cutaneous pyoderma characterized by thick crusted erosions or ulcerationEtiology: S.aureus &/ group A StreptococcusClinical finding:Poor hygiene & neglectLocation : lower extremities (children, DM, elderly)Efl: ulcer punched-out, indurated, raised, violaceous, granulating base slow to heal need several weeks of antibiotic treatments
ERISIPELAS Soft tissue infection erysipelas & cellulitisUsually present at an antecedent lesion or siteLocal pain, variable degrees of erythema, systemic symptoms
SELULITISExtends deeper into the dermis and subcutaneous tissueEtiology : S.aureus and group A Streptococcus (>>)Clinical finding :Erythema, tenderness, painLack of distinctive margins between affected and normal skinDeeper, firmer form of tender indurationCrepitus on palpationBulla and necrosisRegional lymphadenopathy
SELULITIS
PENATALAKSANAANIstirahat tirah baring & elevasi tungkaiSistemik antibioticTopikal : kompres dingin dengan normal saline sterilIntervensi bedah
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