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Communicable Diseases
Caused by a pathogen that enters thebody, multiplies, and causes disease
TransmissibleAfflict the most vulnerable
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Communicable Diseases
Host
AgentEnvironment
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Modes of Transmission
DirectCongenital, Sexual, Direct Contact
IndirectFomiteVector
Mechanical, BiologicalVehicle
Airborne, waterborne
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ASEPSIS AND INFECTION CONTROL
Asepsis- absence of disease producingmicroorganisms
Medical Asepsisclean techniqueReduces number of microorganisms
Surgical Asepsissterile techniqueIncludes all sterile procedure/techniques toeliminate all microorganisms from an area
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Cleansing, Disinfection, SterilizationCleansing- removing visible dirtDisinfection- reduce number of potentialpathogens but spores are notnecessarily destroyed
Sterilization- complete destruction of allmicroorganisms including their spores
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Methods:1. Steam (autoclave)
2. Gas (Ethylene oxide)3. Radiation4. Chemical5. Boiling water
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Infection Control
Handwashing- single most importantinfection control practiceNecessary elements:
FrictionRunning water Cleansing agent
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Removing protective devices:
1. Gloves2. Mask
3. Gown4. Goggles5. Cap
6. Shoe cover
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the tiers of precaution
Standard precautionTransmission-based precaution
Airborne precaution droplet nuclei smaller than 5 mHigh-Efficiency Particulate Air filter Air-filtered roomPrivate roomDoor is shut
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the tiers of precaution
Standard precautionTransmission-based precaution
Droplet precaution droplet nuclei larger than 5 mDoor may be openMask if within 3 feetLimit transportPrivate room
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the tiers of precaution
Standard precautionTransmission-based precaution
Contact precautionGown and glovesDedicated equipmentPrivate room
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Principles of Sterility
A sterile object remains sterile only whentouched by another sterile object.Only sterile objects may be placed on asterile field.A sterile object becomes contaminatedby prolonged exposure to air.
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Principles of Sterility
A sterile object or field out of the range of vision or an object held below a personswaist is contaminated.When sterile surface comes in contactwith a wet, contaminated surface, thesterile object or field becomescontaminated
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Principles of Sterility
The edges of a sterile field are consideredcontaminated.Fluid flows in the direction of gravity.
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Respiratory System
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Respirat ory System
Upper RespiratoryTractLower RespiratoryTract
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Respiratory System
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Respiratory System
Lower Respiratory TractBronchioles
Terminal BronchiolesRespiratoryBronchioles
Alveoli
Type IType IIAlveolar Macrophages (DustCells)
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Respiratory Sys tem
LungsPleural Membrane
Parietal Pleura
Visceral Pleura
Lung Lobes and
Fissures
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Respiratory System
Pulmonary VentilationInspiration andExpiration
Cellular RespirationExternal
Internal
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Respiratory Sy stem
Muscles of Respiration
Quiet RespirationPiston ActionPump Handle Motion
Bucket Handle Motion
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Respiratory Sy stem
MechanicsForced InspirationQuiet Expiration
Forced Expiration
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Respiratory System
Lung VolumesTidal Volume (500 ml)Inspiratory Reserve Volume (IRV = 2100-3200 ml)Expiratory Reserve Volume (ERV = 1200 ml)Residual Volume (RV =1200 ml)
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Respiratory System
Lung CapacitiesInspiratory Capacity (=4000 ml)Vital Capacity (= 4800 ml)Functional Residual Capacity (=2000 ml)Total Lung Capacity (=6000 ml)
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Respiratory System: Control
Respiratory Center In the medulla and pons
Medullary rhythmicity areaPneumotaxic area (>E)Apneustic area (>I)
Cerebral Cortex
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Respiratory System: Control
Hering Breuer ReflexInhibits excessive lung expansion
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Respiratory System: Control
ChemoreceptorsCentralPeripheral
Aortic and carotid bodies
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Respiratory System: Control
OthersTemperatureIrritation of airwaysVolitionPainEmotionAnal Sphincter Stretching
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Assessment
Health Historychief complaint
impact on patient's lifeif chronic, ongoing assessment of abilities& quality of life
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Signs & Symptoms
Dyspneadifficulty breathing
due to decreased lung compliance or increased airway resistance
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Signs & Symptoms
Coughfrom irritation of the membranes
chief protection against accumulation of secretions
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Signs & Symptoms
Sputumreaction of lungs to any constantlyrecurring irritantprofuse & with color usually is bacterialthin & mucoid is viral
bad breath usually is respiratory in origin
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Signs & Symptoms
Wheezingheard with airway narrowing
high-pitched, mainly expiratory
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Signs & Symptoms
Clubbingdistal phalanx of each finger isbulbous & rounded
nail plate is more convexusually due to chronic hypoxiamay be pulmonary or cardiac
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Signs & Symptoms
Hemoptysisexpectoration of blood
underlying disease must be diagnosedregardless of amount of bloodvs. Hematemesis
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Physical Assessment
Nose & Sinuses check external nose for
lesions, asymmetry or
inflammation tilt head backward &
assess the mucosa inspect the septum &
turbinates palpate the sinuses
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Physical Assessment
Pharynx & Mouth open mouth wide & take
a deep breath
check tonsils, uvula & post. pharynx
tongue depressor is put past midpoint of tongue
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Physical Assessment
Thoraxcheck skin color & turgor
check for deformities
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Physical Assessment
ThoraxFunnel Chest (Pectus
excavatum)depression of lower portion of the sternum
may compress theheart
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Physical Assessment
ThoraxPigeon Chest (Pectus Carinatum)
due to displacement of the sternumincrease in AP diameter
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Physical Assessment
ThoraxKyphoscoliosis
elevation of scapulaS-shaped spine
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Physical Assessment
Respiratory Ratesnormal RR: 12-18 bpm
EupneaBradypneaTachypnea
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Physical Assessment
Breathing PatternsHypoventilation
Hyperpnea (depth)Hyperventilation (depth and rate)Apnea
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Physical Assessment
Breathing PatternsKussmaul's
Cheyne-stokesBiot's (Cluster)Apneustic
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Physical Assessment
Thoracic Palpationtenderness, massesrespiratory excursion
costal margin if anterior level of 10th rib if posterior
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Physical Assessment
Thoracic Palpationtactile fremitus
vibration of the chestpatient asked to repeat "99", "eee"air impedes sound, solids conduct sound
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Physical Assessment
Thoracic Percussionto determine content of underlying structuresto estimate size & location of certainstructures within the thoraxdullness at left 3rd - 5th interspace is the heartdullness at right 5th interspace to costalmargin is the liver
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Physical Assessment
Thoracic AuscultationUseful for assessing air flowUsed to evaluate presence of fluid or solidobstructionAllow patient to rest during examinations
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Physical Assessment
Thoracic AuscultationAdventitious Sounds
additional soundsCrackles (Rales)Wheezing
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Diagnostics
Pulmonary Function TestsAssess respiratory function and
dysfunctionMeasures lung volumes and ventilatoryfunction
Studies mechanics of breathing and gasexchange
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Diagnostics
Arterial Blood Gas StudiesMeasures PaO2, PaCO2, pH, HCO3Obtained through an arterial puncture
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Diagnostics
Sputum StudiesFor diagnosis, drug sensitivity testingTo determine whether malignant cells arepresentExpectoration is the usual methodObtained in the morning so specimens
accumulate overnightDo not allow specimen to stand as thismay cause overgrowth
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Diagnostics
Imaging StudiesEndoscopic Procedures
BronchoscopyThoracoscopy
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Respiratory System: Tests
Pulse OximetrySpirometry
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Diagnostics
ProceduresThoracentesisBiopsy
PleuraLungLymph Node
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Client Needs: Oxygenation
Interventions to promote oxygenationDeep breathing and coughing exercises
Abdominal breathingPursed-lip brathing
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Client Needs: Oxygenation
Interventions to promoteoxygenationChest physiotherapy
a. Percussionb. Vibrationc. Postural Drainage
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Client Needs: Oxygenation
Oxygen Therapy
Concentration and liter flowper minuteHumidification
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Obstruction and Trauma
EpistaxisCaused by rupture of tiny vessels in any areaof the nose
Most commonly over the anterior septumwhere the following vessels enter:
Kesselbachs plexus
Sphenopalatine artery (posterosuperior)Internal maxillary (lateral)
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Obstruction and Trauma
Epistaxis (treatment)Direct pressureSilver nitrate, electrocauteryPacking
May remain in place for 48 hours
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Upper Respiratory Tract
Viral Rhinitis (Common Cold)Sx: rhinorrheaHighly contagiousMost common cause of absenteeism fromwork and school
Most common cause is rhinovirus
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Upper Respiratory Tract
Acute SinusitisInfection of the paranasalsinuses
Usually due to drainageobstruction60% are bacterial
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Upper Respiratory Tract
Chronic Sinusitis > 3 wks in adults, > 2 wks in children Same organisms as acute sinusitis
Symptoms most pronounced in themorning
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Upper Respiratory Tract
RhinitisInflammation and irritation of the mucusmembranes
non-allergic or allergicSx: rhinorrheaNursing
Avoid the allergenBlow the nose before any medication in thenasal cavity
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Upper Respiratory Tract
Acute Pharyngitis Mostly viral The most common bacterial cause is
group A beta-hemolytic Streptococci Throat cultures, nasal swabs and blood
cultures may be necessary
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Upper Respiratory Tract
Tonsillitis and Adenoiditis 3 tonsils: palatine, lingual and pharyngeal The pharyngeal tonsils are also called the
adenoids Grp A beta-hemolytic Streptococcus is the
most common causative organism
Post-op: prone with head turned to theside
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Upper Respiratory Tract
Peritonsillar Abscess Collection of purulent exudate between the tonsil and
surrounding structures Believed to be tonsillitis which progressed to local
cellulitis and abscess
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Upper Respiratory Tract
Laryngitis Inflammation of larynx
Almost always viral if infectious With voice changes
and cough
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Obstruction and Trauma
Acute Laryngeal EdemaAllergic, traumatic, inflammatoryHoarseness, shortness of breathInterventions
Epinephrine and corticosteroids
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Obstruction and Trauma
Chronic Laryngeal EdemaObstruction of lymph drainageHoarseness, shortness of breathInterventions
Artificial airway may be necessary
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Obstruction and Trauma
LaryngospasmTrauma or inflammatoryIntervention
OxygenSuccinylcholine
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Obstruction and Trauma
Fractures of the NoseUsually without serious consequencesObstruction or disfigurement may resultRule out a skull fracture if with rhinorrheaReduced 7-10 days after the injury
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Obstruction and Trauma
Obstruction During SleepMost common is sleep apnea syndrome3 Types
Obstructive the most commonCentralMixed
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Obstruction and Trauma
Obstruction During SleepObstructive Sleep Apnea
Frequent and loud snoringBreathing cessation for 10 seconds or moreFive episodes per hour or moreFollowed by awakening abruptly with aloud snort as oxygen levels drop
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Lower Respiratory Tract
AtelectasisClosure or collapse of alveoliDue to reduced alveolar ventilationMay be due to secretions, anyobstruction, pressure
Pneumo-, hemothoraxPleural effusion
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Lower Respiratory Tract
Pulmonary TuberculosisPrimarily an infection of the lung, it mayalso involve other body partsThe agent is Mycobacterium tuberculosisThe leading cause of death frominfectious disease in the world
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Lower Respiratory Tract
Pulmonary Tuberculosis Treatment6-12 monthsDrugs
H, INH Isoniazid - HepatotoxicR, RIF Rifampicin Hepatotoxic, discolorsZ, PZA Pyrazinamide Most hepatotoxic
E, EMB Ethambutol - optic neuritisS, STM Streptomycin - Ototoxic
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Lower Respiratory Tract
Pneumonia Inflammation of
lung parenchymacaused by
infection
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Lower Respiratory Tract
PneumoniaCAP
In community or first 48 hours of hospitalization
S. pneumoniae is the most common causeMycoplasma is common in older children andyoung adultsH. influenzae affects the elderly and those withcomorbidsViruses are the most common cause in infantsand children
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Lower Respiratory Tract
PneumoniaCAP
In adults, the most common viruses are theinfluenza, adenovirus, parainfluenza,coronavirus and varicella-zoster In immunocompromized adults, CMV is the
most common
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Pl
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Pleura
PleuritisInflammation of the pleuraWorse with deep breathing, coughing or sneezing (respiratory movement)Analgesics and find underlying causeTurn to the affected side
Pl
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Pleura
Pleural EffusionAccumulation of fluid in the pleural spaceThe size of the effusion and the underlying
disease determine the severityMost commonly due to infection or malignancyChemical pleurodesis, pleurectomy,
thoracentesis may be done
Pl
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Pleura
EmpyemaLocalized collection of pusMay thicken pleura and restrict the lungUsually complications of lung infection,trauma or surgeryRequires 4-6 weeks of antibioticsThoracentesis, thoracostomy may bedone
L R i t T t
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Lower Respiratory Tract
Bronchitis Acute
Fever, cough,wheezing
Chronic Cough worse in the
evening and morning Lasts 3 months for 2
consecutive years
L R i t T t
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Lower Respiratory Tract
BronchitisTreatment
Bronchodilators, corticosteroidsPostural drainage and chest percussion
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B hi t i
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Bronchiectasis
Chronic wet cough with foul-smellingsputumHemoptysisRecurrent fever and chillsAntimicrobials, bronchodilators may begivenResection, lobectomy may be done
E h
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Emphysema
Abnormal enlargement of the air spacesdistal to the terminal bronchioles withdestruction of alveoli
Increased expiratory effortTreatment: O2, bronchodilators,antimicrobials
Smoking cessationLung transplant
Asthma
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Asthma
Chronic inflammatory disorder of thebronchial airwayWith periods of bronchospasm
Worse at night, with wheezingTreated with bronchodilators and steroidsTreated in a step-wise manner
Status asthmaticus and intubation
COPD
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COPD
Obstruction of air flow due to emphysemaor chronic bronchitisPredisposing Factors:
Cigarette smokingPollutionOccupational exposure to irritants
COPD
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COPD
TreatmentBronchodilatorsOxygen therapy; be careful not to depress
respiratory driveNursing Management
Smoking cessation
Diaphragmatic breathingPursed-lip breathingInspiratory muscle training
Acute Respiratory Failure
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Acute Respiratory Failure
PaO2 < 50mm Hg, PaCO2 > 50 mm Hg,pH
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Acute Respiratory Failure
Restlessness and dyspnea are earlyNeurologic, tachycardia and tachypneaare lateAssist with intubation and mechanicalventilation
Acute Respiratory Distress
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Syndrome
An inflammatory reaction triggers thediseaseDiffuse alveolar capillary damage, severepulmonary edema, respiratory failureBecomes unresponsive to supplementaloxygen and with stiff lungs
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Acute Respiratory Distress
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Syndrome
Medical ManagementPEEPAntibiotics to prevent infection
Treat hypovolemia due to leakage
Under investigation; includes anti-
inflammatories and steroids
Pulmonary Hypertension
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Pulmonary Hypertension
Systolic pulmonary artery pressure > 30mm HgMean Pulmonary Artery Pressure > 25 mm
HgForms
Primary fatal within 5 years of diagnosis,
idiopathicSecondary from existing cardiac or pulmonary disorder (COPD)
Pulmonary Hypertension
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Pulmonary Hypertension
Symptoms of Right-sided heart failureOxygen therapyVasodilatorsHeart transplant
Pulmonary Heart Disease
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Pulmonary Heart Disease
Cor PulmonaleRight ventricular enlargement secondaryto a pulmonary conditionConfusion and somnolence may bepresent due to hypercapniaSymptoms of underlying diseaseSymptoms of heart failure
Pulmonary Heart Disease
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Pulmonary Heart Disease
Cor PulmonaleOxygen therapy and bronchodilatorsIntubation and mechanical ventilationTreatment of CHF
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Pneumoconioses
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Pneumoconioses
Disorders caused by inhalation of irritantsUsually occupationalEffects of substances depend on:
ConcentrationDuration of exposureAbility to initiate an immune responseIndividual susceptibility
Pneumoconioses
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Pneumoconioses
Silicosis Chronic, nodular, dense
pulmonary fibrosis Asbestosis
Diffuse pulmonary fibrosis Black Lung Disease
Coal Workers Pneumonia Cor pulmonale and
respiratory failure
Pneumoconioses
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Pneumoconioses
Management if always removal of irritantfrom work environmentIf unavoidable, institute protectivemeasuresMinimize exposureEnsure ventilation
Bronchogenic Carcinoma
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Bronchogenic Carcinoma
90-95% of all lung tumorsTobacco smoking is the most importantfactor Sx: chronic cough, hoarseness,dysphagiaCXR reveals a solitary peripheral noduleand atelectasis
Mediastinal Tumors
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Mediastinal Tumors
Includes tumors of the thymus, lymphnodesMay cause heart and lung symptoms,
chest pain, dyspneaTreatment with radiation or chemotherapy
Chest Trauma
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Chest Trauma
Pneumothorax Traumatic
Pneumothorax
Tension Pneumothorax Hemothorax
Chest tube placement(2nd or 4 th /5 th )
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Respiratory Care Modalities
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Respiratory Care Modalities
Non-invasiveOxygen TherapyNebulizer Postural DrainageBreathing Retraining
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2-6 lpm5-8 lpm
6-10 lpm
10-15 lpm
4-10 lpm
Respiratory Care Modalities
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Respiratory Care Modalities
Invasive Endotracheal
Intubation Tracheostomy
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Suctioning
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Suct o g
Oropharyngeal10-15 cm along side of mouth
Nasopharyngeal
Along floor 10-15 sec, rotate, 20-30 sec intervals, 5 mintotal
Avoid complicationsHyperinflationHyperoxygenation