i

l{!,t i Li \ l,!

9. ARDS phase with d€strucrion ottvr€membmne formatioh._..

46r ) c<, j

I pneumocFes, hyaline

l;rltlCINE 2 POST TEST REMEDIALS 2006

[';EMATOLOGY

I. MCY I2O -MACROCYTTC'

2. Retic,index of 4 - Bone mar.ow re.spon.ling b Anehria ,.

3. Clinical manifesrarions of pancyopcnia exceprA. rauor C. ScDsjsB. gleeding D, Thtombosfs

4. Anemia.. . . . .€t-rhro.. .- Anemia ofChronic .liseose

5. kon sire ofabsorption - rtoderrn

6. Low/absenr iron stores - Iton Dellclenq, Anen h

7. End prodfct of 20 homcostasis - Flbtltl clotlo ntion

8. Factot ll - Prothrcmbin

9. Definkivc rrealmenr for chronic ITp - qrlenecnmy

l0.Ceneml S/SX is due to- AlJinky ofhenogtobin to 02

l l . Vain rredhenr ofcnoice ot Anemia ofchronic rencl05e/rnsuthcr(nc). Ertthtopocitih

PULMONOLOCY

l.,Most common et iology ofcommuniry 6cquired pneumonrl- ltrycobqct lnl

2. AP.J)S - AP|O2/FIO2 < 200nnHg

3 . P\lmonary iufarcr , Hohptot ,s

4. Idiosyncraric type ofastilma- (+) tcsponse to Metacho ne testlhg Oronchoproyocdtpe)

5- Mos! coftmon weaning mode jlmech. venriralon. SIMY

6. Most common support mode in mech. ventilation. AIC

7. Most colnmon presentation ofBronchi€ctasisA. CoughB. Hemoprysis

'C. DyspneaD. Pteural Eftusion

8. Oxidative siress meansA. Alpha I AntiTrypsin DeficiencvB. Elasrase & AnhElastase

11. Pleurodhis. . .

L Characrerisiric ofHONK - pH > 7,3

A. ProliferativeB. Fibrosis

C. EytdattueD. Recovery

12. Characterhtic oIDKA _pfi< ZJ

13. DM symptoins cxcept. tys€rlg/r/|'

!4, Sheehan'$tndrome . P/rlaclt

5. Normal valuc ofFBS - 70-rrr,ng%

i6. Normal value of2ppBS - <tt,

r7. High insulin resisrance . Ttpe II DM

8. Not a hiSh risk facror ofDM - Srnoking

9. Thyrojd CA with a besr prognosis - papilot! t nht

10. Effect ofhyperprolactinemia except - ytrlllz[tioh

I l. Grave's dse. - TBZ J Lttt

12. After surg€ry ofFolicular Adenoma - J?4 lLSf

13. Increases during stress excapl - prctactin

14. Nor a manifeslation ofThyroLoricosis . Crhr?{x.,,

15. RAI primary tr€atrnent....

j,u. ,".*"rr,.u..,..

U 4lnr ) ce, j

POST TEST REMEDIALS 2OO6

Fi'MATOLOGY

I. MCV I2O - MACROCYTIC

2. Retigindex of 4 - Bone mar@v respondlnE ro Anehis -.J. Clinical manifeshrions of pancFopenia exceprA. val lor C. SepsisB. Bleeding D. Tt;rombost

4. Anemia.. . . . .erythro.. .- Anemla o/Chnnic tisease

5. lron site ofabsorption - Darderrh

6. Lowabsenr iron stores - hon Defrclenq) Anemta

7, End ploduct of 20 homeostasis - Flbrtn alotfonatiotl

8. Factor ll - Prothrcmbitl

9, Dcfi!ifivc treatmenr for chrcnic lTp - Spleneuom),

I0.OeneralS/SX is dle to- AIfinlty oJhamogtobht to 02

I l . Main rreamenr ofchoice ofAnemia ofchronic renaldse/insuffrciency

PULMONOLOGY

l,-Most common er iology ofcommunhy acquired pneumonja

2. A,WS - APaO2/FIO2 <200nnHE

3. Pulmonary infarct - ltlr pt nb

4. Idiosyncraiic rype ofasrima- \+) tctponse to Mettchoune testlng orcnchoptovo.ative)

5. Most comnton weaning mode i;rrinech. v€ntilation' SIMY

6. Most comrnolr suppo( mode in mech. ventiladon. AIC

7, Most common presentation ofBronchiectasis

l

l*"Xy-:j*xIi:f:*ction ot tvpe 1 pneumoc)tes'

lA. Protiferative C. Exxdattve

D. rrorosls D, Recovery'

llo. Ot comptication ofmectianical !enrilarion'(. Cholernsb C. Dysphagiarr. Heparo D. l le ir is

il L Ple!rodisis...

lZ Mosl common.. . . . . . . . . . . .Transudarive pleurat e| lLsro.- t'dtapneunon ic Elluston

ll3. Mostcomnon d€fense in upper respiratory tract - 1g,1

14..Most imponant defense mechanism ofrhe r(,plf,trof)- Mucoclllsry clearahcelle noo"*,,ro,.o""

jl, Charact€risitic of HON K - pH > Z.j

12, Characteristic ofDKA -pt< ZJ

13. DM syftptorns excepr - n/eigrrSdln

\]4. sheehan'q syndrome . P;oracrr,

5, Normal valuc ofFBS, Zr-rr, rng%

]6. Normalvalue of2ppBS. <1/,

]7. High insulin reshtance . Trpe II DM

8. Not a high risk factor ofDM - Snoklng

9. Thyroid CA wirh a best prognosis - pipillnry unlotl .^_^^lu. Efiecr ot hyperpro lacrinefiia e\,ce - yhlllzutok

I L Graveb dse. - tTJIJ lf,tl/

12. After su€ery ofFollicutar Ad€noma " ltr' tffl/

ll. Increases during sress exc.pr - pntactitt

14. Not a manifestation ofThyrotoxicosis - Canrrydlio,

l5l I{AI primary trearment....

16. Screeningtest.....i

A. CoughB. Hemoprysis

C. DyspneaD. Pleural Efflsion

8, Oxidative stress meansA- Alpha I AntiTrypsin DeficiencyB. Elastase & AntiElasrase

.JAR-DIOLOGY

L Most common cardiac manifesrarion ofHI.

2. lnfective endocardiris h indicated forA. MRB. Pure MVp

3. Pulsus parr'us et rardus .,r.t

4. Continous murmur - pr.it

5. WidesOread ST elevation with upward concavltyA. Acure MlB, A.ute Pe cafttitis

6. 52 (ciosure ofthe semilunar valve)A. ClosLrre ofAV yalveB. Closure of mitral& tricusDid valvcC. None

7, 4th heart sound is best heard withA, DiaphraSmB. Bel l

8. Enhanced by insprrarion (Carvallo,s sign) - fny, MOst commcn primary tumor of$c heartA' MtxomaB, Sarcoma

10. Systol ic murmu.A, MSB, ARC,'NONE

CIf. Dr. Lopcz

l, Denotes pfognosis except - Mass on andoscop!

2. Chronic secrctory dianhea- l/IpOMA

3. Fealure of Ulcerative Colitis - Backwash lteltts

4. Most common sile ofbleeding in diverticulith - C.can

5. Absenr in Ileus - Cofakl abdortt rat palh

6. Choleslasis -R irtbln

7. Diagnosis ofgallbladder dse, - ILltrusoun.l

8. Charcoats triad except - Coisttpdllon

9. Not aprognostic factor ofAcute pancreatitis

10, Most common cause ofAcute pancreatitis- Gallstone

NEPHRO - Dr. Lu

rl. Congo red srain - Amytoidosis

12. AUT "lB in XP.ay - Cateallcarionl^,

J: l)[];yy, r,v o,,,,,ive nepruosc rerosis...onion . . :n .e. on

I]4. Normal ptasma osmolalirv| 275 - 290 nos;oikg

5. Mosr common o€anism inuTl - E, Coti

O.. . . . . . . . "xc"ptA. Oliglria C. RBC

18. Pyuria D. Hemaruria

l/. rrue or Matrgnanr Nephrosclerosis

B. Hyaline arreriosclcaosis

iC, Afferent ancriosclerosis

NDPHRO - Dr. Cort€z

Case., , , , .

,

l. Diagnosh- Acule on Chtunlc Rena! Folture

2. F€aturc ofthe Abovc diagnosis excrp!A. votume ocplet ionB. InfectionC.Obstructioo

JD. Ctro n tc c toneru,,...

]3.Ir indicatcs ctlroniciry inpE- pattar

4. Mos! common finding inR9CN " Crcscentlonrdh,l

15. Post Strep. Glomerutoncpfuilis

6. ....,in serum creatinine-,larg. changct In GFR conetpon.ttng to ont! n stfulf ch.mgcr

7, What is the immediate thing to do in the above caseA. Stop medicationB, Hydratec.D. Dialysis

(I{DUMATOLOGY

.. A 50 years old female with progessive, bilaleral,syinmetr icat. . . . . . . . . . . . . .3 hours momin; st i f fness

1. wlat is the diagnosis - X..l(pIP)

2.ilF-inrhk pt. h t-,, whut i" ri" rrue "t

t.ment- ,gr n toA, Ute$ can he prcsent b, horrnol tn.lirldual

J. The ff. manifeslation are tluc ofRA exceorl- tweUing & ercslon ofDIp

4. The FF. are rrue ofRA exceD!- ESR & RF shoutd ar,eoys b; rcpealed

t^The,pr. didnor.respond ro NSAID & low dosc sterolds for 3mos. what is the bcst tbing to do- KeJcr pL to a thaumatologlsl

--. .-. J0 years old femalc (+) fcv.r, dimrsc inflammedallnfltls. weigh! Ioss, malar nsh

6. what is the diaqnosis- Luptti (SLE) -

7. Most sensitive tcst for Lupus -,{y'{,{

8. Mos! specific tesr for Lupus- rrrly,{

9.ln.case ofsLrspected Lupus flare or cxaccrbanons, which of:Tj|,$Ui,:f ::.j?led

to rorrow the dse. acrrviry orsLE

l0- rhe fL are nanifcsrarion ofSLE cxccpr- Jow etuston on XRat

I L What is the best laborarory test lo diagnosod drug - induced

- Antl-hbtoae ontlbod!

12. Best maintenance treatment for tbe constitutional synptoms

- Eldtuqtchloroqutfie

1];;jffi ur"*' <*l'""'et ""9l4 Esophageal dysmoritiry telangiecrasia...,

ioNcoLocyil.

Heparoccllular marker ",4Fp

l?. fi|; ;;^"" ^"o"tty for disseminated cancers

i . . . . .l';*I;n

Is chemorhe.aey wiI be rhe main rrea,menr

l

ll,tlit:!|il:'f:'--t which indicates thar the dse is no

j- P,oercsstve

^e.5. Risk factors for breast C A excepr _ Earl! pregnanct

16. Most inpoftant prognostic factot - Axiuary LN

i7. Poor progDosis for Lun g Ca. except - Fenute

]l.Jf;:l,l*r a"rn*t assrcssivc bur responr.!e ro ,redrmenr- smau ceu

i9. True ofcotorectat Ca e xcept - Lelr Si.tetlI

l)'ii;;' :i|: :i:,ffi"' "ctor or cororectar ca

I L True ofnanagement ofColorecralCa exceD.l- AuJuvant ch.motheruplJor stuge It & I

I I 2. Glcason score . Crad.

13. Tr€atrnofi for ;ctastrti a prost^ri. Ca - Hothon l

]14. Stage ctinically - C.rvtca, Ca

ll5. Risk factor for ovariane.- Fe ttt!)) drug

116. Mosr comnon site ofptostatic Ca - pe ph.ttl

l.' #;P;Y; ;t * "r

Nasopharynsear ca

I 18. Not a risk facror of .....,....,.,.......Ca - pAgnanc!

I ]fl;;;;.non*, .i.t ructor fox Head & Neck ca

IItIEUMATOLOCY - Drd. R. Sy

I . The pain in OA arises from &e following srucruresexceptA. Artic lar ca itase eroslonB. Stsetching ofjoint capsuleC SEetch-ing ofperiosteum covering lIe osreoph)lesu, syDovttrs

OA -chronic. non jnflammarory. pajn is nor scvere(Jn Acurc, lnf laf tmarory,monGanicular

2. 1he fottowin8 a,e LhJracrer isr ic ofa coury Anhnl isexceptA. Acute in onsetD. Plasma UA tevet ma) Dor bc f in aU !6cs

- :::",:;i!",::*"r **t t' n.c.ssary da hs theD, tjsually rhonoaniculaf, acute, iflanmsrory (CA)

Civing. 4ltopurinot unty protongs the dumrion ofCouhAnhri t is

3. lh€ (4^str l founJ in rhc syno! ial f lu id in tseudogourA. Ca Oxalarc8, Calclum Dt ,tlhosphate delrydnteL. lll0testcrotD. Mouosodiun uratc

Pscudogour - CppD_ rhomboid, triangular

GA * MSU- needle shape

e, Acu:e gouly.a(hriris pJthog.ncsis/\, ueIydfttronB. D-cpdsirior) of|rlonosodium uralc crysrats injoin!s\. E nauot hgeslionD. lncnclton of utule crlstots &pMN

5. The hosr c-ommon route by \.v/c bacrcda efltel thejoinl/\. Lonog0Us site ofinfectionB. Direct InoculationC, HematogenousD. Trauma li

STAI'H AUREUS - mosr.common organism in s€pticA(hdtis

TB Artlriris - cluonic (2 yrs, to slrcw clanges)

SEPTIC ARTHRITIS _ don,r show moloallicularhvolvemedt

6 A rubifacirnr dmg !rhose mechanism ofacrion is looeprere subeance l , in rhe tocatnerve ending4. LaNaAn crcamB. Menthotared cream

NSAID creamTranadol

NSAID-COX2- prod. OlpG inducibte

. COX2 Specific/ Setective. COX2 Non Spccific- inhibits COXi & COX2

_ good housekeeping COX. - promotes renal blood ilow_ prevenb Eastric ulce|arior

7. Not a render point sideA. clunealB. I"ow cervicalC. Scapato,D. Trapezius

T€nder point side- Fibromyalgia- ach€s & pajn

- gencmlized & lyidesDred\l- tcnder poiot side r 80% sufe

8. Moir comjnon sirc ofOA in eld,:rly in thf dcrc.ol-ocounriesA. Arlle (ttt_ reperitive, coustant uscl

C, HsndD. Hips (2M - wt. bearingj$.)

OsreoArthririsBefore a8c 50 - 4\ malaAfterage 50 - 6 fqnale

Hand OA - with g€netic predisposition

Join;Entargemert. Bony hyp€rtrophy' S),rlovialhlperpl.sia. Fluid in thcjoinr

Bony DIP- HEBERDBN'S NOD6SPIP- BOURCHARD'S NODES

. BOUTONIERE'S NODES .RA

. PIANO KEY - RA

9. Th€ force applied lo a reoder poinr ofribrom)i.r-r.rA. 40 gmsB.400gnsC. 4000 gtnsD. 40,000 grns

I0. One ofrhe fol lowing nanifcsr with chrunirmonoanic u lar arth riris

A. CrystalinducedB. FractueC, Mlcoblcte um TBD. Staph. Aureus -4-6 wks IV antibidjc rx

C.D.

pff*#g#li'#ffiu*ln){i,irIL fhe most appropriate thing jou

are going ro Oo at ttrisA, Do hothlngD. slan Altopurinol lo ! rhe UA;: ffi lii,T?lilf"i8td:Ii,^ * *

3i*:ff:;|i"t:11*rhia when there's cA, Renar

J' ,DOCofOA PARACETAMOUACEI_r grn per day (maxim_uit};no""u*

:'' o"q "p6a - Nljir2oo/o

irelderlv

_ drawback; expcnsivo

.1-,li "*,';*::^l'::1,:, rresen* wirh vcry painrul

:ec. coC ii rj.iii![i" ankre No hx ortrauma. remp. is

/2. Whar ar€ you gojng ro do at rhi3 ri-"

; :;::::l1l,rlflI,J"J.,ff rxtoranrrritis; );;;;.;;:;:;i;i;i,;):ilt|!

"", *,,,,*,N cell court ar the joint 2.2000

r l*1*ll*?,rrffi:*f, jffi ,'n"'"*- "o. uram staijt & cukurcr: yrgar & prorcthu. Lrysrdlanalysk

14. Most common orgarism jn lnfectious arthritis

RA - FemalecA - Male .t

-' ),fi;' ;'.xD. "".i"*' 0"",",'an

16,. Management ofOA include(, meramol, NSAlD..ubifocienr dru8q dnn & cold compressr r. srerord injectjon

T.Changes in OAI " d€srrucdod ofhyaline canilage

18. Acule CA cha.ractrk,i^.- monoanicular- tnflainrnabry- acute tn ons€t

l9. .Acr ion of Coichicine- 'nnrbrts

pl\4N acrjvation by inhjbiting prorern20. comrion bacteria for OA _ STA\H AIlnEU.\^

i'r+1fif3ff,f"fi!" vc bacteria eme,s !rre.joj,),22.- ljgaments, tcndons attaches to the bone

- zero (_) spondyto

14;$;"0"tt.", "n-r". t oe cadilage.ofoA will res!lr irA V^ir nurrl ber of chondroc) rcs - Tru,.

^ --"iffifi:ff

'ndrecirrirasc ..rj'ur

:. . .lo,s: of proreogty;n & coltdlen _ r,u. vnn tnnune ciipt* ir;;;=;;;;i,: , , ,.OA - no immune cornplex in th! cartilage

" ';*iii:'ffiffi:fiT*1ith elcepr {arJ neubrJ ( ns. nypoparathyroid;srn . tflrcr. xnemta _ false

'u,oot*",oto t *o rx of arrhliris is drc ro Inc-cox2

- FibroDyalSia

Bursa, terdon, ljgament, epiphy,(is _soll thsue rheuoratisrlr

i"f"lil, j:"Tj jxT;lf ,lxli,ft:il;*[5,i"i,fr ]illlRisk Factor:

.' f;ffi - most imnonunt 'r

#ffi1p1::rx:iff &Til"*,""

:9,A,lbroceDrcsis in the R knee. . ..Ca pfophospnalc

- Rhottbold

30. False OA tr€a8nentA. no drug to prevent OAB. Slstehk stercids arc benellcttt

ll. ioinl replocemenr in OA wilt improve quatiry oflife.Sugery is inctuded if

A. ft can mainhin desired body weightB. (f) oneophlr,es do!\ corelate w clin. manC. elevated DSR not a! ijdication3 for surge.yD. ,eiactabk pol & los, o unctlan

J/- une or the Joflowing is nor a diagnostix xRay finding ofvA uornr canrtage darhage)

A, subchondBlsclerosisB. loss ofjoi;lt spaceC. ostec.phltosisl). osaeophorosts (t^6e.ularjt. Bone lo6s)

33. Mechanism ofaction ofNSAID- hlhlbib COX2

34. pr. wirh OA raki0gNSA.ID devetoped CI side cffecl.Risk Ficror for NSATD(se: grsEopathy) EXCEpT

A. Using anricoaSulants - TB. Past Hx ofot bleedinr -TC. Pt. with esophagitis --TD. 20 "30 !.o patient _ FALSE

35. JS.40 yo obcse. post driD.king sproo... wnkc up...6rrhntts. , , diagnosjs.GA

36. hyperuriccnia results in retal dse cxceDtA. u'ate ncpbropathy - TF. nrorithiasis - TC. UTI _ FALSE

37. ll)?eruricemia secondary !o purine overproduction is

A. V excretion -FB. dehydration - FC. low dose salicylare -F \lD. Henotrttc d/d -.fr.tld

38. Cbronic, polyarthdtis, BilateEl involvemont-EA

RA. PIP. DIP

39. RhF in this pt is (-), rvhat is thc true statemenrA. Dx is unlikely RA ifR.F is (-)B. RhF is spccific for RAC- NtF l lote tlters can beprese t tn N tndh,tdaal

40. Tbe following arc nsifesrations otRA ex.. t,.A, bwatr treck deformiryB. Boutomier'sC. Joinr.erosion & peri-afticutar osreopnyre.u. twethg & ercston ofDtp

4 L th; fotlowing are truc of RA exceptA. More common in f€oaleA. Assoclated pirh.lacc atkn oJ ESR and Rht

should ateats be.

42. acdvc pl. didn'tresponded lo NSAJD & los oo,csrcrords lbrJ mos. What is lbe best rhinA lo do

A. Add Methotlr'exateB. Chatrge to another NSAID or ,1, lhe dose ot

conicosteroids

30 y/o female,-febrile, diffuse inflammation arthrfrs, wrloss. malarrash, discoidrash, alopecia. moLrh L.!e,,pr€ufl trc chestlain. Laboratory fi ndings:anemra. thromboc' ' topenia, Glomerulo.. , , r , /D\D\ 4,(r)anti,smhh.

^ ESR Urinatysis: 8rbc. I0 kb(. . -l A\^vc3&c4

43. Diagnosis -SLE44. wbich oflhc followillg is tI( most sensiri\e r0.l forLrlp'-ls - ANl45. Most sp€cific for Lrpus - rrrN,4

46. in casc_of Lupus flarc or€xacirbarjon. whjcr ofrherorowng taboratory cxam could bi repsated !o fo,low rirdsc activity ofSLE- ESR, D'DNA, C3 & C1

47. Iianif€starion ofSLE cxcepa-Jolnl etuslon on xra!

48. Most importa lab test to dri drus-induce SLL. ANTI HISTONE Ab

49, bestmaint€nance trcamenr for connirutional {\ & -.r, ,ofSLE

A, HldtoxychlonqubteB. NSAIDC, Pr€dnisorcD. Cyclgphosphamide

50 l-0 y/o wirl' headache, claudicrrion ofuE. Rr hp .60/10- Rehcet,s Dsc