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8/10/2019 K Class 5 TG2 Muscular System Review
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Kinesiology
FES 4
th
Ed Chapter 9 p. 283
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Functions of Muscular System
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Skeletal muscle is connected to bone It is considered voluntary or under conscious
control of the cerebrum
Involuntary movements are also possiblesince sometimes reflexes (protectivemechanisms that occur without thought) willcause skeletal muscle to contract.
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The primary function of skeletal muscle is toexert a pull on the bones which createsmotion.
The contractions of skeletal muscles lift ourfeet, purse our lips for whistling, and expandthe ribcage during breathing.
Skeletal muscle is striated & produces very
strong, rapid contractions Its fibers fatigue more rapidly than those of
smooth or cardiac muscle.
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Skeletal muscles are fragile & are vulnerableto damage.
They have a limited ability to regeneratethemselves after injury.
However, the way muscles cells are bundledtogether & reinforced with connective tissue,protects them during strong musclecontractions.
The connective tissue acts as envelopeswhich then converge to make tendons whichattach the muscles to the bones they move.
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Skeletal muscles can remain upright againstgravity.
They can keep your head up and centered,your trunk straight and erect, and your hipsand knees aligned over your feet.
Skeletal muscle can also adjust and respondto changes in posture, as when you lean over
or stand up from a chair. These postural muscles cannot rest as long
as you are awake and upright.
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Skeletal muscles protect underlyingstructures in areas where bones do not.
The abdomen is not protected by theskeleton so 4 strong abdominal musclesprotect the underlying organs while allowingfree movement of the trunk.
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As skeletal muscles contract to producemovement, they also produce body heat.
This production of heat is called
thermogenesis. Approximately ¾ of the energy created by
muscle tissue is used as body heat.
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Cardiac muscle is responsible for driving thecardiovascular system but skeletal musclesalso play a role.
The contractions of skeletal muscles helppropel the circulation of lymph & venousblood.
The pumping of the heart keeps the pressure
within arteries high, but both lymphaticvessels & veins have relatively low pressure.
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They require help from the contraction ofsurrounding muscles to keep their fluidsmoving forward.
This is especially important where thesefluids must flow upward against gravitysuch as in the legs.
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Muscle Fiber Directions
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Arrangements: these muscles have fibers ofequal length that do not intersect
This arrangement:
◦ Enables the entire muscle to shorten equally& in the same direction
◦ Maximizes range of motion
Most muscles in the arm are parallel.
Types: Strap, Fusiform, Circular, Triangular, &Spiral
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Rectus Abdominus
Long muscle withconsistent width
Examples: Sartorius,quadratus lumborum,
& rectus abdominus Some sources refer to
rectus abdominus as amulti-belly muscle.
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Coracobrachialis
Have a thick centralbelly with tapered ends
The tapered ends applyforce to specific bonylandmarks
Examples: brachialis,biceps brachii &coracobrachialis
Most muscles in body
are fusiform
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Orbicularis oculi
These musclessurround an openingto form a sphincter
They are designed tocontract & close
passageways or relax& open them
Examples: Orbicularisoris, orbicularis oculi& sphincter ani of
anus.
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The fibers of these muscles start with abroad base then converge to a single point.
This fan-shaped arrangement allows themto diversify their actions, creating multiple
movement possibilities. These muscles can pull in different
directions depending upon which fibers arerecruited.
Examples: Deltoid, pectoralis major &trapezius with their multiple & sometimesopposing actions.
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Pectoralis Major Trapezius
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Latissimus dorsi
Some sources referto muscles thathave a twistedarrangement as
spiral muscles
Examples:Latissimus dorsi &
trapezius
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These muscles are feather-shaped withshorter muscle fibers intersecting a centraltendon.
This arrangement maximizes the number offibers in one area.
More muscle fibers mean greater cross-sectional area & greater force production by
these types of muscles. Types: Unipennate, Bipennate & Multipennate
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Extensor Digitorum
Longus
The muscle fibers runobliquely from one sideof a central tendon.
These muscles look likehalf of a feather.
The arrangement allowsstrong force productionfrom one direction.
Examples: lumbricals,tibialis posterior &
extensor digitorum
longus
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Rectus femoris
The muscle fibers runobliquely along bothsides of a central tendon.
These muscles look like afull feather.
Very strong musclecontractions are possiblesince the central tendonis pulled from 2
directions. Example: Rectus femoris
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Deltoid
These muscles havemultiple tendons withoblique fibers on bothsides.
The muscle fibersconnect the tendons &pull from manydirections.
Example: Deltoid
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Skeletal Muscle Properties
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Ability to stretch without sustaining damage whichallows muscles to lengthen when relaxed.
This is important because muscles usually work inopposite directions as they produce force whilemaintaining stability & balance at joints.
If one muscle is shortening, its opposite must relax &lengthen to allow the joint to move in the intendeddirection.
For example, when the anterior muscles of yourupper arm (flexors) shorten, the posterior muscles ofyour upper arm (extensors) must relax & lengthen.
Without extensibility, the lengthening muscle wouldbe damaged.
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Ability to return to its original shape afterlengthening or shortening.
As muscle tissue performs its variousfunctions, its shape changes or deforms.
Once the work is completed, the muscletissue can rest & resume its original form.
This property allows a muscle to maintain a
specific shape when at rest.
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Muscle tissue can respond to a stimulus byproducing electrical signals.
In response to an event such as a touch or adecision to move, nerves at their junction withmuscles release specialized chemicals(neurotransmitters).
The neurotransmitters cause the spread of anelectrical signal (action potential) that in turntriggers a series of events that lead to a muscle
contraction. Without this ability to respond to the nervous
system, muscle would not be able to contract &function.
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The muscle’s ability to propagate transmit or
spread) electrical signals, including action
potentials.
Once muscle tissue is ‘excited’ by the
nervous system, it must carry the electricalsignal to the inner cell structures.
Conductivity allows the action potential to be
transmitted along the muscle cell, activatingthe tissue & initiating a muscle contraction.
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The ability to shorten and thicken in responseto a specific stimulus.
Here the stimulus is the action potentialinitiated by the nervous system.
The ability to shorten is a unique feature ofmuscle tissue
Specialized proteins (myosin and actin) within
muscle tissue interact to shorten and thickenmuscles, generating a force.
Our bodies depend on this force to move.
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Anatomy of Skeletal Muscle
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Macroscopic or Gross Anatomy
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Connective Tissue
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Connective tissue
structures support,
protect separate
portions of muscles
whole muscles.
Endomysium: sheath
of connective tissue
that wraps individual
muscle cells or fibers
or myofibers.
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Perimysium: layer
of connective tissue
that encircles
holds together
bundles of muscle
cells called fascicles
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Epimysium:
◦An envelope
surrounding all the
muscle is a part of
the deep fascia
network◦ Converges to form a
tendon that connects
the muscle to the
bone
◦ Musculotendinous
junction is the point
where muscle belly
ends tendon starts
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Portion of the
muscle between
the tendons
‘Meaty’ portion
of the muscle
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Origin:◦ Usually the heavier & proximal attachment
◦ Attachment that does not move or is most stable
Insertion:
◦ Usually the lighter & distal attachment◦ Attachment that moves or is most mobile
◦Greatest amount of movement takes place at
insertion
Aponeurosis:
◦ Broad sheet-like tendon
◦ Broad sheet of fibrous connective tissue
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Large blood vessels
nerves are
enclosed within the
epimysium
Capillaries nerve
fiber endings are
wrapped within the
endomysium where
they interact with
individual muscle
cells
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Microscopic Anatomy
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Sarcolemma:
◦Surrounds the entire
muscle cell
◦ Serves as a cell
membrane regulates
chemical transport into
and out of the cell.
Sarcoplasm:
◦ Gelatinous substance
that surrounds the
structures within the
muscle cell
◦Functions as the
cytoplasm of the cell
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Mitochondria:
produces ATP, a
compound that
stores the energy
needed for muscle
contraction
Nuclei:
contain the
functional
information for the
cell control its
operations
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Myofibrils
myofilaments:
Myofibrils are
specialized
contractile proteins
that make skeletal
tissue look striated
(or banded)
Stripes or bands
reflect the two types
of filaments or
myofilaments – actin
myosin
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Sarcomeres:
◦ Contain structures from one Z line to the next◦
Functional units of muscle fibers since it is the
shortening of the sarcomere that produces
concentric muscle contractions
Regions of the Sarcomere:◦ Lighter I Band: where the thin filaments or actin
occur alone◦ Darker A band: where the thick or myosin & thin
filaments overlap◦
Z line: The lighter I bands are interrupted by azigzag line called the Z line which marks theborders of the sarcomeres.
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Transverse tubules:
◦Transmit nerve impulses
from the sarcolemma to
the cell interior
Sarcoplasmic
reticulum (SR):
◦ Network of fluid-filled
chambers that cover
each myofibril like a lacy
sleeve
◦These chambers store
calcium ions which help
trigger muscle
contractions.
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Physiology of Muscle Contraction
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Neuromuscular Junction: point of close contactbetween neuron & muscle fibers
Axons of Neurons: long thin structures that reachout from the neuron cell body to transmit anaction potential through its terminal branches to
muscle cells Action potential: nerve impulse that is strong,
invariable, & capable of traveling long distancesin the body – from a neuron in the brain to amuscle cell in a finger.
Synapse or synaptic cleft: gap between the axonbranches & the muscle cell that prevents theaction potential from crossing to the muscle onits own.
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Acetylcholine (Ach):
◦ Neurotransmitter that helps the action potential to jump the gap or synapse
◦ Ach is stored in seminal vesicles at the ends of theaxon branches & is released when an action
potential reaches the NMJ◦ Once across the gap, Ach binds to receptors within
the muscle fiber’s sarcolemma
◦ This binding stimulates a new action potential onthe muscle fiber side of the synapse
◦ The new action potential in turn initiates thechemical processes of muscular contraction
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A neuron sends an electrical signal calledan action potential down its axon
The signal reaches the ends of the axonbranches, where it stimulates synapticvesicles to release acetylcholine (Ach)
Ach molecules cross the synaptic cleft &bind with receptors in the sarcolemma
A muscle action potential travels along thesarcolemma & down the transverse tubules
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Explains how the thick & thin myofilamentsbind & release to produce shortening in thesarcomere in a concentric musclecontraction.
Four contractile proteins are involved:
◦Myosin
◦Actin
◦
Troponin
◦Tropomyosin
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Myosin Actin
Makes up thestrands of thethick myofilament
& the roundedends are calledheads whichattach to binding
sites on actin
Makes up thestrands of thethin filament &
has ‘beads’ onlong strands.
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Tropomyosin: ◦ Its threads covers the actin ‘beads’◦ As long as the muscle is relaxed, it covers the
binding sites on the actin molecules, preventingthem from participating in a muscle contraction.
◦
Myosin heads attach to these binding sites tomove the actin Troponin:
◦ Tropomyosin threads are studded with &controlled by clusters of Troponin
◦
This protein keeps Tropomyosin in place over theactin binding sites in relaxed muscle & moves outof the way to allow a muscle contraction.
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Action potential reachesSarcoplasmic Reticulum &stored calcium ions arereleased into the sarcoplasm.
The calcium ions bind withthe studs of Troponin on the
actin, causing them to ‘moveaside’ the Tropomyosinprotein strands covering thebinding sites on the actin.
Once the binding sites arerevealed, the thin filament is
ready for contraction.
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The myosin heads on the thickfilament are charged withenergy from the breakdown ofATP.
This energy binds the myosinheads to the actin filament
creating connections calledcross-bridges. Once cross-bridges are
formed, a ratcheting actioncalled the power stroke occursas the myosin heads, bound toactin, pull the sarcomere
together.
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Like a line of rowers in a long boatsimultaneously pulling their oars againstthe water, myosin heads along the thickfilament pull & slide the thin filamenttoward the center of the sarcomere,shortening the strand.
As the myosin heads complete theirpower stroke, they bind with more ATP.This provides the energy necessary for
them to release their hold on the actinstrand.
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This process is repeated by alternatingmyosin heads on both sides of the thinfilament along the length of the musclefiber, creating muscle contractions.
Once the thin & thick filaments haveaccomplished muscle contractions, thenerve action potential stops.
Any ACh remaining in the synaptic cleftis broken down and deactivated.
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Calcium ions are released from theTroponin & actively pumped back tothe SR using additional energy from
ATP. The Tropomyosin threads realign withthe actin binding sites, preventingfurther formation of cross-bridges.
The muscle then passively returns toits resting length.
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http://joellezimprich.wikispaces.com/11--
Muscle+Physiology
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Action potential crosses from transversetubules to cell interior.
Calcium ions released from SarcoplasmicReticulum.
Receptor sites on actin exposed as calciumions bind to Troponin.
Charged myosin heads bind to actin
creating cross-bridges. Power strokes pulls ends of sarcomeres
together creating muscle contraction
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Factors Affecting Force Production
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Motor Neurons: neurons responsible for delivering anelectrical impulse to muscle fibers
Motor Unit:
◦ Consists of a motor neuron all the muscle fibers it
controls
◦ Muscles are typically composed of multiple MUs◦ When MU reaches a muscle, it can synapse with 2-3
or up to 2000 muscle cells but average number is100-200 muscle cells.
◦ Some MUs have few fibers (face & hand) which allowsthem to produce fine movements
◦ Smallest MUs are in muscles that move the eye
◦ Other MUs (thigh) have 1000s of muscle fibers & canproduce powerful movements but lack fine control.
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The body can control the amount of forceproduced by a given muscle by varying thenumber & size of MUs recruited
◦ Stimulation of few motor units generates a
small amount of force.◦ Stimulation of all motor units generates
maximal force.
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The larger the
motor units
recruited the
number of motor
units recruited
results in the
greater potential
force production.
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Some motor units stay activated all the time,creating a minimal amount of tension in restingmuscles that keeps them firm & ready tocontraction.
This tension indicates the strength ofconnection between the nervous system &skeletal muscle.
If muscles are utilized frequently, as withexercise, increased tone may result.
Muscle tone helps maintain posture & jointstability & decreases time needed for muscleforce production.
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Excessive tone
that can be
present in
overworked
muscles
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Decreased use orinjury can resultin less muscle
tone
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A major factor influencing muscle forceproduction
The muscle’s ability to generate force relatesmore closely with a muscle’s thickness than
its total volume. Thicker, shorter muscles generate more force
than longer, thinner muscles.
Cross-sectional area is related to the size ofthe myofibrils.
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As myofibrilsbecome largerthrough use(hypertrophy),
muscles increase incross-sectionalarea & can generatemore force.
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A decrease in
size of myofibrils
from lack of use
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Results in
reduced cross-
sectional area
reduced ability to
generate force
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Pennate fiber arrangements generate moretotal force than parallel muscles.
Pennate arrangement allows more musclefibers to reside in a given area.
More muscle fibers increase the muscle’scross-sectional area & ability to generateforce.
Pennate muscles sacrifice ROM for increasedstrength & speed.
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A muscle at its resting length has maximumspace to shorten in the sarcomere & thisallows the greatest force production.
As a muscle stretches beyond resting length,
fewer cross-bridges are formed between theactin & myosin & fewer cross-bridges meanless force production.
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Skeletal Muscle Fiber Types
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Also called slow oxidative fibers; Contract (twitch) slowly but are resistant to
fatigue – they take approximately 1/10 of asecond to reach maximum tension.
They rely on aerobic energy production (usingoxygen).
They are red in color due to their rich blood
supply (highly vascularized).
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Slow twitch fibers are utilized for longduration activities (more than 2 minutes) suchas walking and jogging.
Postural muscles that must remain contracted
for extended periods of time are primarily
composed of slow twitch fibers.
Slow-Twitch (red
–
aerobic) fibers contract
more slowly with less intensity but have
more endurance myoglobin.
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Generate fast, powerful contractions but
quickly fatigue – take approximately 1/20 ofa second to reach maximum tension. Fibers are larger in diameter than slow twitch
fibers due to a greater number of
myofilaments. More myofilaments means the ability to
produce more force. Fast twitch fibers don’t use oxygen for energy
production – are anaerobic Are a lighter color since they don’t have
blood vessels
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Sprinter Climber
Utilize anaerobic energyproduction.
Used for short-durationactivities (less than 2minutes) such assprinting (in the legs of
sprinters) & lifting. Large powerful muscles
are primarily fast twitchfibers.
Contact rapidly
forcefully but fatigue
quickly.
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Have characteristics of both fast & slow
twitch They range from red to pink in color.
We can consider intermediate (mixed) fibersas reservists waiting to be called up when &where the need arises.
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Type 2 2X size of Type 1
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Biceps Brachii
Have a higherpercentage of fast-twitch white fibers
Jump into actionquickly & tire quickly
Usually weaken inresponse to posturalmuscle shortening
Examples: deltoid,
biceps brachii
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Soleus
Higher percentage of
slow twitch red fibers
Relatively slow torespond
Tend to shorten &increase in tensionwhen under strain
Examples are erectorspinae, transverse
abdominus & soleus.
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Distribution is intermingled & geneticallydetermined. Some people have a large amount of slow
twitch fibers & their muscles tend to be long& lean which predisposes them for longduration activities such as marathons ordistance biking.
Others have large amounts of fast twitchfibers & their muscles tend to be larger &thicker making them great sprinters or bodybuilders
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Small motor units are composed of red slowtwitch fibers
Large motor units are composed of white fasttwitch fibers
Motor units are homogenous in that a motorunit either has all red slow twitch or all whitefast twitch fibers
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Types of Muscle Contraction
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Occur when tension is generated in a musclebut the joint angle & muscle length don’t
change
This type of contraction is used to stabilize
joints Pushing or pulling against an immoveable
object or holding an object in a fixed positionrequires effort by the muscles but no motion
in the joints.
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Muscle contractions that change the length
of the muscle and create movement
2 Types:
◦Concentric: in this type of muscle
contraction, the muscle shortens & jointangle decreases.
◦Eccentric: in this type of muscle
contraction, the muscle lengthens & jointangle increases.
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Used to slow & control movements Occurs when the muscle lengthens while under
tension & changes in tension happen to controlthe downward movement of load
The eccentrically contracting muscles can
generate an upward force when we need to resistthe pull of gravity.
Eccentric contractions are the most powerful,
followed by isometric, then concentric.
Eccentric contractions are about 50% moreforceful than concentric but use less oxygen &energy & involve fewer muscle fibers.
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Results in
lengthening of the
active muscle (one
doing the ‘negative
work’) such as the
controlled down-
phase of biceps curl
Sit-ups, deep knee
bends squats are
eccentric
contractions of the
hamstrings
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Injuries often occur with eccentriccontractions when we try to prevent orcontrol movements such as falling ordropping an object.
Eccentric contractions more often causeDelayed Onset Muscle Soreness (DOMS) 8-24hours after activity.
Why DOMS happens is unclear but it may be
that the action of stretching or lengthening ismore likely to cause microtears in the muscle.
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Tonic (Tone):
◦ A continual partial contraction◦ At any one moment a small number of the total
fibers in a muscle contract, producing tautness ofthe muscle rather than a recognizable contraction
& movement
Fibrillation:
◦ An abnormal type of contraction in whichindividual fibers contract asynchronously,producing a flutter of the muscle but no effectivemovement
◦ Fibrillation of the heart occurs fairly often.
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Twitch:
◦ Single quick, jerky muscular contraction from asingle nerve impulse followed by relaxation
Tetanic:
◦ A continuous, forceful muscular contractionarising from a series of at least 30 nerve impulsesper second
◦ Also known as a spasm or cramp
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Concentric contractions create
movement
Eccentric contractions modify
movement
Isometric contractions:
◦ Stop movement
◦ Act to fix or stabilize body parts or
to prevent an undesired joint
movement
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Definitions
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Threshold Stimulus All or None Response
Minimal nervousstimulus neededto initiate amuscularcontraction
If a muscle fibercontracts at all, itwill contractcompletely or allthe way
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Integrating Contraction Types in
Human Movement
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When a jointed area moves intoflexion & the joint angle is decreased:◦ The prime mover synergists areconcentrically contracting.
◦ The antagonist eccentrically contractwhile lengthening
◦ The fixators isometrically contract &
stabilize the attachment at the originof the prime mover
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Integrating Contraction Types in Human
Movement
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The quadriceps muscles are extending theknee, allowing you to rise from the chair.What type of contraction?
The muscles of your back & trunk are keeping
you steady as you rise.What kind of contraction?
If you decide to sit back down, the quadricepsmuscles slow your descent.
What type of contraction?
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Quadriceps are in a concentric contraction.
These are isometric contractions of your back& trunk muscles.
The quadriceps are in an eccentric
contraction. These contractions keep youfrom flopping down into the chair.
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Standing at a sink holding a pot in one hand &filling it with water from the tap. What type of
contraction for elbow flexor muscles?
Once the pot is full, you lift it up & away from the
faucet.What type of contraction for those same elbow
flexors?
You carry the pot to the stove & carefully lower it
to the burner trying not to spill the water or dropthe pot. What kind of contraction? Which
muscles? What are the opposing muscles doing?
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You feel the muscles on the front of yourupper arm (elbow flexors) working harder asthe pot fills. This is an isometric contractionto hold the pot steady.
Lifting the pot is a concentric contraction ofthe elbow flexors.
Eccentric contractions of the elbow flexors
control the lowering of the pot onto the
burner. The elbow extensors are in aconcentric contraction.
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Brachialis
Cross & act on onlyone joint
Example: Brachialis
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Cross act directly
on two joints
Common in the
body
Examples include
Hamstrings, Biceps
Brachii,
Gastrocnemius
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Act on 3 or more
joints due to the line
of pull between their
origin their
insertion crossing
multiple joints
Examples are
muscles that move
the fingers toes
such as flexor
digitorum profundus
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As a muscle shortens, its ability to exert forcediminishes.
Active insufficiency: when a muscle shortensto the point where it can’t generate or
maintain active tension Passive insufficiency: when the opposing
muscle is stretched to the point it can nolonger lengthen & allow movement
These principles are most easily observed inbiarticular & multiarticular muscles.
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A good example is seen in wrist movements.◦ For active insufficiency, making a fist with the
wrist extended is easier than doing it with thewrist flexed.
◦ The shortened flexor muscles are weaker as they
attempt to make a fist.◦ In the multiarticular muscles of the extensors of
the wrist & fingers, passive insufficiency limitsfinger & wrist flexion when they are performedtogether.
◦ A greater range of motion of wrist flexion ispossible with the fingers extended.
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Muscle Relationships or Roles
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Involved in creating a joint movement or desired action.
Also called a prime mover if it is the most powerfulmover.
Prime movers are primarily responsible for moving a joint through a given action such as flexion orabduction.
For example, the deltoid is primarily responsible forshoulder abduction so it is the agonist & prime moverfor this movement.
A group of movers or agonists can exist for a specificaction but it does not mean that every muscle of thatgroup contracts every time the action occurs.
It is possible for one or a few muscles in the group tocontract & produce the movement while the rest of themuscles of the group are relaxed.
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Muscles also have roles by their muscleactions: flexors, extensors, abductors,adductors, rotators, supinator, pronators,dorsiflexors & plantar flexors.
No matter the role they play, muscles stillrespond to the requirements of thresholdstimulus & all-or-none response.
Flexors, internal rotators, & adductors are
about 25%-30% stronger than theirantagonists.
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Usually located on the opposite side of the jointfrom the agonist
Perform opposite actions to the agonist
Example: Latissimus dorsi performs shoulderadduction so it is an antagonist to the deltoid
supraspinatus.
Opposite actions include:
◦ Flexion & extension
◦ Abduction & adduction
◦ Internal & external rotation
Antagonist to biceps in supination - pronator teres
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The agonist-antagonist relationship is criticalfor balanced posture as well as for slowing &controlling movements.
For example, the erector spinae (trunk
extensors) are counterbalanced by theantagonist rectus abdominus (trunk flexor).
Proper development of each is critical formaintaining normal, upright trunk posture.
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In walking, the hip flexors & knee extensorsswing the leg anteriorly thereby helping topropel the body forward.
The hip extensors & knee flexors are required
to slow & stop this movement. Without proper balance between these muscle
groups, the body would not be able to control& finish movements it initiates.
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General Rules:
◦ With upward movements, agonists work &antagonists relax
◦ With horizontal movements, agonists work
& antagonists relax◦ With downward movements, antagonists
work & agonists relax
◦ With faster downward movements,agonists contract & antagonists relax
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Biceps in elbow flexion
Deltoids in shoulder abduction
Quadriceps in knee extension
Sternocleidomastoid in neck flexion
Rectus abdominus in bowing
Pectoralis minor in shoulder protraction
Gastrocnemius in plantar flexion
Gluteus medius in abduction
Iliopsoas in hip flexion
Biceps brachii in supination
Serratus anterior in shoulder protraction
Infraspinatus in laterally rotating humerus
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Supraspinatus
Assist the function of the
agonist. Muscles that have the
same action or actionsare consideredsynergists.
For example,supraspinatus assists the
deltoid in shoulderabduction which makesthis pair of muscles
synergists.
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Latissimus dorsi in shoulder extension Infraspinatus,
Teres minor, major
Triceps in elbow extension Anconeus
Gluteus medius in hip abduction TFL
Gluteus maximus in hip extension Hamstrings
Latissimus dorsi in medial rotation Subscapularis
Teres major
Biceps brachii in supination Supinator
Iliopsoas in hip flexion Rectus femoris
Hamstrings in knee flexion Popliteus
Gastrocnemius
Serratus Anterior in protraction Pectoralis minor
Trapezius in shoulder elevation Levator scapulae
A muscle or a force that can stop an unwanted action at
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A muscle or a force that can stop an unwanted action atthe fixed (‘origin’) attachment of the muscle that is
working When a fixator muscle contracts, it contracts isometrically Example: When the levator scapulae contracts, it can
create a pulling force on the cervical vertebrae & thescapula.
If the right levator scapulae is causing right lateral neck
flexion, the fixed attachment is the scapula & the movingattachment is the cervical vertebra.
Its action on the scapula has been cancelled out by thelower trapezius which is creating a downward force on thescapula.
The lower trapezius is performing an isometriccontraction that ‘fixes’ the scapula in place.
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Trunk stabilizers
◦ Internal obliques & transverse abdominus
◦ Multifidus◦ Quadratus lumborum
Shoulder stabilizers
◦ Trapezius & levator scapulae◦
Rhomboids◦ Serratus anterior & pectoralis minor
Pelvic stabilizers
◦ External rotators◦ Hip abductors: gluteus medius & minimus, TFL◦ Pelvic floor muscles & diaphragm per some
sources
A muscle or a force that can stop (by an isometric
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A muscle or a force that can stop (by an isometriccontraction) an unwanted action at the moving(‘insertion’) attachment of the muscle that is working
Example: When the levator scapulae contracts, it cancreate a pulling force on the cervical vertebrae &scapula.
If the right levator scapulae is contracting & causing theneck to right laterally flex, extend & rotate to the right,
the fixed attachment is the scapula & the movingattachment is the cervical vertebra. If we only want to laterally flex to the right & not extend
or rotate to the right, then the other 2 actions must bestopped.
SCM contracting as a neutralizer stops both undesired
actions. SCM creates a flexion force that cancels out the
extension & a left rotation of the neck that cancels outthe right rotation.
A muscle that can hold another part of the body (by an
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A muscle that can hold another part of the body (by anisometric contraction) in position while the joint action
is occurring Does not work directly at the site of the joint action &
often works far from where the action is occurring
Usually works against gravity
Creates a contraction force that is equal in strength but
opposite in direction to the force gravity exerts on thatbody part
The body part does not fall & the support muscle doesnot contract so hard that the body part is movedupward
Example: When holding a barbell in the right hand &doing bicep curls, the left erector spinae prevents thetorso from laterally flexing toward the weight on theright side of the body
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Myotomes: Group of muscles innervated by
the motor fibers of a single spinal nerve root
Dermatomes: Skin area innervated by thesensory fibers of a single spinal nerve root
While slight variations exist, dermatome &myotome distribution patterns are relativelyconsistent between individuals
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Myotome = orange
Dermatomes = blue
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C3, 4 & 5 supply the diaphragm C5 also supplies the shoulder muscles & biceps C6 is for wrist extensors C7 is for elbow extensors C8 is for finger flexors T1 is for finger abduction
T1–T12 supplies the chest wall & abdominal muscles L2 flexes the hip L3 extends the knee L4 dorsiflexes the foot L5 wiggles the toes
S1 plantar flexes the foot S3, 4 & 5 supply the bladder, bowel & sex organs & the
anal & other pelvic muscles
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In diagrams or maps, the boundaries ofdermatomes are usually sharply defined
However, in life there is considerable overlapof innervation between adjacent dermatomes.
If there is a loss of sensory nerve function byone spinal nerve, sensation from the regionof skin it supplies is not usually completelylost
The skin areas served by spinal nerves have
some overlap but there will be a reducedsensitivity if one spinal nerve is damaged
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Common Injuries
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Salvo p. 148
Results from
overstretching muscletissue, usually themuscle is overloadedwith the severity of theinjury related to theamount & rate of
overloading. Strains can be mild,
moderate & severe Rated as first degree,
second degree & thirddegree.
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Caused by compressive
forces sustained during
impact result in
hematomas or bruises in
the muscle tissue.
Caution is indicated in
massaging over
contusions
Force depth of
pressure need to be
reduced to prevent
further injury.
Lymphatic drainage
applications are usually
appropriate.
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Myositis Ossificans Cramps
A possible complication of
a serious contusion The theory is that fibro-
blasts recruited duringhealing process begin tobecome osteoblasts &
produce a calcified masswithin the muscle tissue.
The mass is usuallyreabsorbed by the bodyover time.
Causes can include
electrolyte imbalance,deficiencies in calciumand magnesium, &dehydration.
Cramps may involvemoderate to severemuscle spasms withproportional levels ofpain.
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Muscle soreness that occurs after unaccustomed
exercise.
Characterized by pain, swelling, stiffness restricted
movement.
Symptoms appear 24 - 48 (or 72) hours after activity.
Causes can include microtears or microtrauma tomuscle tissue which stimulates inflammatory response
Associated more with eccentric & isometriccontractions.
Other causes can include damage to the connectivetissue that holds the muscle together & increasedinterstitial fluid in the area
Can be treated by applications of ice for the first 48-72hrs & by massage with emphasis on lymphatic drainage.
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Bleeding or edema within a musclecompartment from injury or excessivemuscular exertion.
Pressure increases within the compartmentwhich can severely damage nerves & blood
vessels. Symptoms: swelling, discoloration,
diminished distal pulse, loss of sensation,tingling, & loss of motor function.
The individual needs to be referred to aphysician
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Tendonitis
Inflammation of atendon caused byfrequentmicrotrauma with
symptoms ofswelling & pain
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SCM in neck flexion
Biceps in elbow flexion
Quadriceps in knee
extension
Deltoids in shoulder
abduction
Serratus Anterior in
shoulder protraction
Iliopsoas in hip flexion
Serratus Anterior in
shoulder protraction
Trapezius in shoulder
elevation
Gluteus maximus in
hip extension
Iliopsoas in hip flexion
Latissimus in shoulder
extension
Latissimus in medial
rotation