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Volume 14 Number 2, Part I February, 1986 in younger age groups following cortisone and adre- nocorticotropic hormone. Am J Pathol 28:315-318, 1952. 152. Bulkey B, Roberts W: The heart in systemic lupus er- ythematosus and the changes induced in it by cortico- steroid therapy. Am J Med 58:243-264, 1975. 153. Kalbak K: Incidence of atherosclerosis in patients with rheumatoid arthritis receiving long-term corticosteroid therapy. Ann Rheum Dis 31:196-200, 1972. 154. Buckley B, Robert W: Steroid therapy during acute myocardial infarction. Am J Med 56:244-249, 1974. 155. Klinenger J, Miller F: Effect of corticosteroids on blood salicylate concentration. JAMA 194:131-134, 1965. 156. Chatterjea JB, Salomon L: Antagonistic effect of A.C.T.H. and cortisone on the anticoagulant activity of ethyl biscoumacetate. Br Med J 2:790-792, 1954. 157. Petereit L, Meikle A: Effectiveness of prednisolone dur- ing phenytoin therapy. Clin Pharmacol Ther 22:913- 916, 1977. 158. Pickup M: Clinical phannacokinetics of prednisone and prednisolone. Clin Pharmacokinet 4:111-128, 1979. 159. Melby J: Systemic corticosteroid therapy, pharmacology and endocrinologic considerations. Ann Intern Med 81:505-512, 1974. 160. Olefsky J, Kimmerling G: Effects of glucocorticoids on carbohydrate metabolism. Am J Med Sci 271:202-210, 1976. 161. Dumler F, Hayashi H, Hunter J, et al: Racial differences Oral glucocorticoids 177 in the incidence of steroid diabetes in renal transplant patients. Henry Ford Hosp Med J 30: 14-16, 1982. 162. Blereau R, Weingarten C: Diabetic acidosis secondary to steroid therapy. N Engl J Med 271:836, 1964. 163. Boyer M: Hyperosmolar anacidotic coma in association with glucocorticoid therapy. lAMA 202:1007-1009, 1967. 164. Pennisi A, Fiedler J, Lipsey A, et al: Hyperlipidemia in pediatric renal allograft recipients. J Pediatr 87: 249-251, 1975. 165. EI-Shaboury A, Hayes T: Hyperlipidemia in asthmatic patients receiving long-term steroid therapy. Br Med J 2:85-86, 1973. 166. Brennan M: Corticosteroids in the treatment of solid tumors. Med Clin North Am 57:1225-1240, 1973. 167. Starzl TE, Penn I, Putnam CW, et al: Iatrogenic altcr- ations of immunologic surveillance in man and their influence on malignancy. Transplant Res 7: 112-145, 1971. 168. Sherlock P: Adrenal cortical steroids in the pattcrns of metastases. lAMA 181:313-315, 1962. 169. Hoshaw R, Schwartz R: Kaposi's sarcoma after im- munosuppressive therapy with prednisone. Arch Der- matoI116:1280-1282, 1980. 170. Leung F, Fam A, Osoba D: Kaposi's sarcoma compli- cating cortical steroid therapy for temporal arteritis. Am J Med 71:320-322, 1982. ABSTRACTS Lipedema (German text) Stenger D, Bahmer FA: Akt Dermatol11:51-54, 1985 Lipedema, manifested by symmetric swelling of the legs, is not well known. The term is misleading, since it is path- ogenetically not an edema but a constitutional disturbance of fat distribution. Young women are mostly affected. Fifty percent of the patients are obese; in another 50% only the lower extremities are obese. Treatment is very unsatisfactory. Phlebedema and lymphedema are to be considered in the differential diagnosis. Alfred Hollander, M.D. Basal cell nevus syndrome and radiation therapy (German text) Roth C, Breuninger H, Rassner G: Akt Dermatol 11:55-57, 1985 In a 32-year-old man with basal cell nevus syndrome, x-ray therapy (10,000 rads) was administered to eight tumors on the trunk that had previously been excised. Subsequently nevoid basal cell carcinomas developed in the irradiated areas. X-ray therapy is contraindicated in basal cell nevus syndrome. Intensive ultraviolet therapy may also be a risk. Alfred Hollander, M.D. Lichen follicularis decalvans, spinulosus et pigmentosus (German text) Stadtler R, Schaumburg-Lever G: Akt Dermatol 11 :58-63, 1985 A 36-year-old patient with Graham-Little syndrome also showed manifestations of erythema dyschromicum perstans on the trunk. Authors are not certain whether this case rep- resents a variant of Graham-Little syndrome or a simultaneous appearance of Graham-Little· syndrome or erythema dys- chromicum perstans. Pathogenetically, toxic influences of medicaments were considered since ash-gray pigmentations appeared and disappeared repeatedly. Alfred Hollander, M.D. Association of HLA antigens with scleroderma (German text) KUhnl P, Schlitz K, Altmeyer P, et al: Z Hautkr 60:866-868, 1985 Progressive systemic and circumscribed scleroderma are associated with different HLA antigens, DRl, DRS, and B27, respectively. HLA typing in suspected cases of progressive systemic scleroderma can be useful in securing the diagnosis. Alfred Hollander, M.D.

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Page 1: Abstracts

Volume 14Number 2, Part IFebruary, 1986

in younger age groups following cortisone and adre­nocorticotropic hormone. Am J Pathol 28:315-318,1952.

152. Bulkey B, Roberts W: The heart in systemic lupus er­ythematosus and the changes induced in it by cortico­steroid therapy. Am J Med 58:243-264, 1975.

153. Kalbak K: Incidence of atherosclerosis in patients withrheumatoid arthritis receiving long-term corticosteroidtherapy. Ann Rheum Dis 31:196-200, 1972.

154. Buckley B, Robert W: Steroid therapy during acutemyocardial infarction. Am J Med 56:244-249, 1974.

155. Klinenger J, Miller F: Effect of corticosteroids on bloodsalicylate concentration. JAMA 194:131-134, 1965.

156. Chatterjea JB, Salomon L: Antagonistic effect ofA.C.T.H. and cortisone on the anticoagulant activity ofethyl biscoumacetate. Br Med J 2:790-792, 1954.

157. Petereit L, Meikle A: Effectiveness of prednisolone dur­ing phenytoin therapy. Clin Pharmacol Ther 22:913­916, 1977.

158. Pickup M: Clinical phannacokinetics of prednisone andprednisolone. Clin Pharmacokinet 4:111-128, 1979.

159. Melby J: Systemic corticosteroid therapy, pharmacologyand endocrinologic considerations. Ann Intern Med81:505-512, 1974.

160. Olefsky J, Kimmerling G: Effects of glucocorticoids oncarbohydrate metabolism. Am J Med Sci 271:202-210,1976.

161. Dumler F, Hayashi H, Hunter J, et al: Racial differences

Oral glucocorticoids 177

in the incidence of steroid diabetes in renal transplantpatients. Henry Ford Hosp Med J 30: 14-16, 1982.

162. Blereau R, Weingarten C: Diabetic acidosis secondaryto steroid therapy. N Engl J Med 271:836, 1964.

163. Boyer M: Hyperosmolar anacidotic coma in associationwith glucocorticoid therapy. lAMA 202:1007-1009,1967.

164. Pennisi A, Fiedler J, Lipsey A, et al: Hyperlipidemiain pediatric renal allograft recipients. J Pediatr 87:249-251, 1975.

165. EI-Shaboury A, Hayes T: Hyperlipidemia in asthmaticpatients receiving long-term steroid therapy. Br Med J2:85-86, 1973.

166. Brennan M: Corticosteroids in the treatment of solidtumors. Med Clin North Am 57:1225-1240, 1973.

167. Starzl TE, Penn I, Putnam CW, et al: Iatrogenic altcr­ations of immunologic surveillance in man and theirinfluence on malignancy. Transplant Res 7: 112-145,1971.

168. Sherlock P: Adrenal cortical steroids in the pattcrns ofmetastases. lAMA 181:313-315, 1962.

169. Hoshaw R, Schwartz R: Kaposi's sarcoma after im­munosuppressive therapy with prednisone. Arch Der­matoI116:1280-1282, 1980.

170. Leung F, Fam A, Osoba D: Kaposi's sarcoma compli­cating cortical steroid therapy for temporal arteritis. AmJ Med 71:320-322, 1982.

ABSTRACTS

Lipedema (German text)

Stenger D, Bahmer FA: Akt Dermatol11:51-54,1985

Lipedema, manifested by symmetric swelling of the legs,is not well known. The term is misleading, since it is path­ogenetically not an edema but a constitutional disturbance offat distribution. Young women are mostly affected. Fiftypercent of the patients are obese; in another 50% only thelower extremities are obese. Treatment is very unsatisfactory.Phlebedema and lymphedema are to be considered in thedifferential diagnosis.

Alfred Hollander, M.D.

Basal cell nevus syndrome and radiation therapy(German text)

Roth C, Breuninger H, Rassner G: Akt Dermatol11:55-57, 1985

In a 32-year-old man with basal cell nevus syndrome,x-ray therapy (10,000 rads) was administered to eight tumorson the trunk that had previously been excised. Subsequentlynevoid basal cell carcinomas developed in the irradiated areas.X-ray therapy is contraindicated in basal cell nevus syndrome.Intensive ultraviolet therapy may also be a risk.

Alfred Hollander, M.D.

Lichen follicularis decalvans, spinulosus etpigmentosus (German text)

Stadtler R, Schaumburg-Lever G: Akt Dermatol11 :58-63, 1985

A 36-year-old patient with Graham-Little syndrome alsoshowed manifestations of erythema dyschromicum perstanson the trunk. Authors are not certain whether this case rep­resents a variant of Graham-Little syndrome or a simultaneousappearance of Graham-Little· syndrome or erythema dys­chromicum perstans. Pathogenetically, toxic influences ofmedicaments were considered since ash-gray pigmentationsappeared and disappeared repeatedly.

Alfred Hollander, M.D.

Association of HLA antigens with scleroderma(German text)

KUhnl P, Schlitz K, Altmeyer P, et al: Z Hautkr60:866-868, 1985

Progressive systemic and circumscribed scleroderma areassociated with different HLA antigens, DRl, DRS, and B27,respectively. HLA typing in suspected cases of progressivesystemic scleroderma can be useful in securing the diagnosis.

Alfred Hollander, M.D.