1-22 Osteoporosis & Hip Pain

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    OSTEOPOROSIS & HIP PAIN

    HAZEM ABDELAZEEM

    Egypt April 2008

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    Osteoporosis appears 1st in Hip

    X rays ( Ward triangle)

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    X ray diagnosis means 40 % bone

    loss

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    Bone Densitometry

    More sensitive

    Part of full 4 sites

    diagnosis

    Tests at differentproximal femur sites

    May be done

    bilaterally Indicates Ca ions loss

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    Hip pain with decreased bone

    density ( Porosis or Malacia)

    Osteoporosis may be

    Generalised or local

    forms

    Osteomalacia may bevit D deficiency or

    other diseases

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    Osteomalacia itself is PAINFUL

    Associated with

    pelvic, femoral & other

    long bones

    deformities Vit D deficiency may

    be dietary, or

    associated with

    celiac,hepatic or renaldisease

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    Stress fracture

    History of overuse

    or osteoporosis

    Pain with weight-bearing activity;

    Antalgic gait

    Limited range ofmotion

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    Stress fracture

    May be at neck,

    subtrochanteric or

    less common the

    head May be uni or bilateral

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    Stress fracture

    Pain may be due to

    microscopic fr or

    progressive

    deformation

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    Combined osteoporosis&malacia

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    Impacted fracture neck femur

    Pt may be ambulant &

    bearing weight with

    pain & limping

    Xray AP & LAT arenecessary but may

    not show the fr

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    Ct scan is diagnostic

    in cases not seen in

    X ray

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    Early internal fixation

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    Consequences :Hip fracture

    Fall or trauma

    followed by inability

    to walk

    Limb externally

    rotated, abducted,

    and shortened

    Pain with any

    movement

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    Fracture pubic rami

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    LOCAL FORMS OFOSTEOPOROSIS

    LOCAL OSTEOPOROSIS IS

    ALWAYS PAINFUL

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    Algodystrophy

    Alogodystrophy is a

    Neurodystrophic

    Disorders

    Pain. Swelling.

    Trophic changes.

    Functional incapacity.

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    The term Algodystrophy covers a group of

    painful conditions with association of pain,

    vasomotor and trophic changes, functional

    impairment localized in the distal parts of

    the body

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    Terminology

    * Algodystrophy (AD)

    Sudecks bone atrophy 1900. Reflex sympathetic dystrophy (RSD).

    Decalcifying alogdystrophy.

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    Post traumatic painful osteoporosis.

    Regional migratory osteoporosis.

    Shoulder-hand syndrome. Transient osteoporosis.

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    ESSENTIAL

    ALGODYSTROPHY

    Unrecognized cause

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    Personal Experience

    Post traumatic

    Pregnancy

    Common among

    medical professions

    Bilaterality &

    involvement of two

    joints or more

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    Pathophysiology

    Theories:

    Neurovascular dystrophy

    Bone remodeling

    Hormonal regulation

    Biomechanical

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    Disturbance of Bone

    RemodellingUnbalanced Cellular Coupling

    OsteoblasteXOsteoclast

    Result: Localized

    trabecular bone loss

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    Oedema Pain

    Stiffness

    NeurovascularVasodilation

    ischaemia

    Vasospasm

    Over sympathetic

    tone

    Stiffness

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    Pathology

    Osteoblastic poor activity

    Subchondral cortical and

    cancellous resprotion

    Wide marrow spaces

    Micro fractures

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    The sites most commonly affected

    are the wrist and hand (28%;,

    shoulder (27%), ankle and foot

    (24%), knee (10%), elbow (6%)

    and hip (5%).

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    Clinical basis & staging

    Radiography

    Bone scintography

    MRI

    Densitometry

    Lab. work up

    Histopathology

    Biopsy [core]

    Diagnosis

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    Clinical Picture and stages

    Stage I: 2 to 3 months.

    * Pain : - Dull - Causalgic

    * Vasomotor: - Redness to bluishness.

    - Swelling. - Wormth - Oedema.

    - *Refrain from movement(Painful)(Pseudoinflammatory signs).

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    Clinical Picture and stages

    Stage II:

    Pain decreases

    Trophic changes:

    - Skin atrophy. - Atrophic hairs.

    - Tappering fingers. - Atrophic nails

    - Joints stiffness.

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    Clinical Picture and stages

    SPONTANEOUS REGRESSION OR

    Stage III ( RARE in Hip):

    Joints increase in stiffness to fibrous

    ankylosis.

    Decrease in the pseudoinflammatory signs.

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    Lab:-

    not constant

    Hydroxyprolinuria

    increased erosive remodelling

    (osteoclast) Osteocalcin level increase

    increased osteoblastic activity

    ESR normal

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    RADIOLOGY: Early X-ray is

    negative

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    Radiology Diffuse rarifaction,

    spotty, patchy,

    widened trabeculations

    Cortical erosions

    Total loss of bone

    structure, by moth

    eaten appearance

    Normal joints

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    CT SCAN

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    Bone scan inconclusive

    Scintography

    Hot area

    [remodelling

    activity]

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    Densitometry Weak photon

    densitometry image

    [ decrease bone

    mass]

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    MRI

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    Core biopsy:

    PathologyPeriosteocytic lysis of

    cortical and

    cancellous bone

    Foci of remodelling

    activity

    Osteoclastic boneresorption

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    ALGODYSTROPHY VERSUS AVN

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    Treatment

    The short-term aims of the treatment of

    algodystrophy are the following:

    To relieve the pain.

    To correct or prevent vasomotor disorders.

    To prevent bone demoralization.

    To prevent trophic change and ankylosis.

    To reduce the duration of functional incapacity.

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    Treatment

    Medical treatment.

    Local injections.

    Sympathetic block.

    Nerve block.

    Physical and

    rehabilitation.

    Accupuncture.

    Psychotherapy.

    Surgical treatment.

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    Medical Treatment

    NSAIDA.

    Vasodilators.

    Corticosteroids.

    Betabolckers.

    Calcitonin.

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    Calcitonin *In Moderate Cases:

    - 100 I.U. every day. For 3months

    *In Severe Cases:

    - 100 I.U. every day. For 2 to 4 weeks followed

    by 100 IU every other day for 2 months.

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    Local Injection

    Local anaesthetic + hydrocortisone.

    Sympathetic ganglion block.

    Nerve block.

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    Other conservative modalities

    Physical and rehabilitation

    therapy.

    Acupuncture.

    Psychotherapy.

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    Surgical Treatment In persistent Acute

    Manifestation

    Lumber Sympathectomy

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    Surgical Treatment Persistant cases & when in doubt that it

    may be AVN Core decompression may

    be done taking also core biopsy

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    Painful focal lesions

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    FOCAL LESIONS

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    After excision

    Post op recovery

    1 Year 2 Years

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    CONCLUSION

    Osteoporosis is a silent disease

    Painful hip associated with osteoporosis

    needs special attention

    Generalized osteoporosis associated with

    osteomalacia is painful and leads to

    painful conditions

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    Back up slides

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    Inflammatory arthritis Morning stiffness or

    associated systemicsymptoms

    Previous history ofinflammatory arthritis ormultiple joint affection

    Limited range of motion andpain with passive motion

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    T.B. Hip arthritis

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    Trochanteric bursitis

    Female:male 4:1, fourth to sixth decade Spontaneous, insidious onset

    Point tenderness over greater

    trochanter

    X-rays may show evidence of aprevious fracture, or metal

    implant . There may also be

    calcification or shadows

    suggesting swelling of the soft

    tissues

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    MRI

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    Hip Synovial affections

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    p yAutoimune,metabolic,specific & nonspecific infections

    synovitis

    Synovial tumors and tumor like (PVNS)

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    Pyogenic hip arthritis

    Changes in chronic caseEarly No Change

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